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Egr2 and 3 Inhibit T-bet-Mediated IFN-γ Production in T Cells

The Journal of immunology (1950), 2017-06, Vol.198 (11), p.4394-4402

2017

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Inhibition of Bromodomain Proteins Enhances Oncolytic HAdVC5 Replication and Efficacy in Pancreatic Ductal Adenocarcinoma (PDAC) Models

International journal of molecular sciences, 2024-01, Vol.25 (2), p.1265

2024

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Early growth response gene-2 controls IL-17 expression and Th17 differentiation by negatively regulating Batf

The Journal of immunology (1950), 2013-01, Vol.190 (1), p.58-65

2013

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Suppressor of Cytokine Signaling-3 Suppresses the Ability of Activated Signal Transducer and Activator of Transcription-3 to Stimulate Neurite Growth in Rat Primary Sensory Neurons

The Journal of neuroscience, 2006-09, Vol.26 (37), p.9512-9519

2006

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Early growth response gene-2 (Egr-2) regulates the development of B and T cells

PloS one, 2011-04, Vol.6 (4), p.e18498-e18498

2011

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RESPONSES OF THE NERVE CELL BODY TO AXOTOMY

Neurosurgery, 2009-10, Vol.65 (4), p.A74-A79

2009

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SAT-383 Rapid analysis of HBV-specific T cell secretomes reveals distinct antiviral immune profiles among chronic HBV patients

Journal of hepatology, 2024-06, Vol.80, p.S742-S743

2024

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The Transcription Factors Egr2 and Egr3 Are Essential for the Control of Inflammation and Antigen-Induced Proliferation of B and T Cells

Immunity (Cambridge, Mass.), 2012-10, Vol.37 (4), p.685-696

2012

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Egr2 and 3 maintain anti-tumour responses of exhausted tumour infiltrating CD8 + T cells

Cancer Immunology, Immunotherapy, 2023-05, Vol.72 (5), p.1139-1151

2023

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Egr-2 is a tumour repressive gene (165.1)

The Journal of immunology (1950), 2011-04, Vol.186 (1_Supplement), p.165-165.1

2011

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Egr2 regulation in T cells is mediated through IFNγ/STAT1 and IL-6/STAT3 signalling pathway

Pathology, research and practice, 2020-12, Vol.216 (12), p.153259-153259, Article 153259

2020

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Early Growth Response Genes 2 and 3 Regulate the Expression of Bcl6 and Differentiation of T Follicular Helper Cells

The Journal of biological chemistry, 2015-08, Vol.290 (33), p.20455-20465

2015

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Early growth response genes -2 and -3 are required for naïve T cell activation and primary response against virus infection (P1150)

The Journal of immunology (1950), 2013-05, Vol.190 (1_Supplement), p.64-64.22

2013

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Egr2 and 3 control inflammation, but maintain homeostasis, of PD-1high memory phenotype CD4 T cells

Life science alliance, 2020-09, Vol.3 (9)

2020

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Egr2 and 3 control inflammation, but maintain homeostasis, of PD-1 high memory phenotype CD4 T cells

Life science alliance, 2020-09, Vol.3 (9), p.e202000766

2020

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Nano-antigen presenting cells for the development of therapeutic vaccines (53.2)

The Journal of immunology (1950), 2011-04, Vol.186 (1_Supplement), p.53-53.2

2011

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Transcription factors early growth response gene (Egr) 2 and 3 control inflammatory responses of tolerant T cells

Immunity, Inflammation and Disease, 2018-06, Vol.6 (2), p.221-233

2018

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Modulation of antigen-specific T-cells as immune therapy for chronic infectious diseases and cancer

Frontiers in immunology, 2014-06, Vol.5, p.293-293

2014

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Early Growth Response Gene-2 regulates the development of B and T cells (153.15)

The Journal of immunology (1950), 2011-04, Vol.186 (1_Supplement), p.153-153.15

2011

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Early growth response gene 2 and 3 are essential for the regulation of tumour suppressor genes, Ikaros, Aiolos and FOXO3

Frontiers in immunology, 2013, Vol.4

2013

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IL-2-engineered nano-APC effectively activates viral antigen-mediated T cell responses from chronic hepatitis B virus-infected patients

The Journal of immunology (1950), 2012-02, Vol.188 (3), p.1534-1543

2012

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SOCS3 suppresses AP-1 transcriptional activity in neuroblastoma cells through inhibition of c-Jun N-terminal kinase

Molecular and cellular neuroscience, 2008-02, Vol.37 (2), p.367-375

2008

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Egr2 and 3 Inhibit T-bet-Mediated IFN-γ Production in T Cells

Inhibition of Bromodomain Proteins Enhances Oncolytic HAdVC5 Replication and Efficacy in Pancreatic Ductal Adenocarcinoma (PDAC) Models

Early growth response gene-2 controls IL-17 expression and Th17 differentiation by negatively regulating Batf

Suppressor of Cytokine Signaling-3 Suppresses the Ability of Activated Signal Transducer and Activator of Transcription-3 to Stimulate Neurite Growth in Rat Primary Sensory Neurons

Early growth response gene-2 (Egr-2) regulates the development of B and T cells

RESPONSES OF THE NERVE CELL BODY TO AXOTOMY

SAT-383 Rapid analysis of HBV-specific T cell secretomes reveals distinct antiviral immune profiles among chronic HBV patients

The Transcription Factors Egr2 and Egr3 Are Essential for the Control of Inflammation and Antigen-Induced Proliferation of B and T Cells

Egr2 and 3 maintain anti-tumour responses of exhausted tumour infiltrating CD8 + T cells

Egr-2 is a tumour repressive gene (165.1)

Egr2 regulation in T cells is mediated through IFNγ/STAT1 and IL-6/STAT3 signalling pathway

Early Growth Response Genes 2 and 3 Regulate the Expression of Bcl6 and Differentiation of T Follicular Helper Cells

Early growth response genes -2 and -3 are required for naïve T cell activation and primary response against virus infection (P1150)

Egr2 and 3 control inflammation, but maintain homeostasis, of PD-1high memory phenotype CD4 T cells

Egr2 and 3 control inflammation, but maintain homeostasis, of PD-1 high memory phenotype CD4 T cells

Nano-antigen presenting cells for the development of therapeutic vaccines (53.2)

Transcription factors early growth response gene (Egr) 2 and 3 control inflammatory responses of tolerant T cells

Modulation of antigen-specific T-cells as immune therapy for chronic infectious diseases and cancer

Early Growth Response Gene-2 regulates the development of B and T cells (153.15)

Early growth response gene 2 and 3 are essential for the regulation of tumour suppressor genes, Ikaros, Aiolos and FOXO3

IL-2-engineered nano-APC effectively activates viral antigen-mediated T cell responses from chronic hepatitis B virus-infected patients

SOCS3 suppresses AP-1 transcriptional activity in neuroblastoma cells through inhibition of c-Jun N-terminal kinase

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