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Entry inhibitors in the treatment of HIV-1 infection

Antiviral research, 2010, Vol.85 (1), p.91-100

2010

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Analysis of Individual Viral Particles by Flow Virometry

Viruses, 2024-05, Vol.16 (5), p.802

2024

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Premature Activation of the HIV-1 Protease Is Influenced by Polymorphisms in the Hinge Region

Viruses, 2024-05, Vol.16 (6), p.849

2024

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Extracellular vesicles carry SARS‐CoV‐2 spike protein and serve as decoys for neutralizing antibodies

Journal of extracellular vesicles, 2021-06, Vol.10 (8), p.e12112-n/a

2021

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Engineering HIV-resistant human CD4+ T cells with CXCR4-specific zinc-finger nucleases

PLoS pathogens, 2011-04, Vol.7 (4), p.e1002020-e1002020

2011

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Phenotypic and Immunologic Comparison of Clade B Transmitted/Founder and Chronic HIV-1 Envelope Glycoproteins

Journal of Virology, 2011-09, Vol.85 (17), p.8514-8527

2011

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A Maraviroc-Resistant HIV-1 with Narrow Cross-Resistance to Other CCR5 Antagonists Depends on both N-Terminal and Extracellular Loop Domains of Drug-Bound CCR5

Journal of Virology, 2010-10, Vol.84 (20), p.10863-10876

2010

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Molecular basis for positive and negative signaling by the natural killer cell receptor 2B4 (CD244)

Blood, 2005-06, Vol.105 (12), p.4722-4729

2005

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COVID-19 and Cardiovascular Disease: From Bench to Bedside

Circulation research, 2021-04, Vol.128 (8), p.1214-1236

2021

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HIV: cell binding and entry

Cold Spring Harbor perspectives in medicine, 2012-08, Vol.2 (8), p.a006866-a006866

2012

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Global phosphoproteomics of CCR5-tropic HIV-1 signaling reveals reprogramming of cellular protein production pathways and identifies p70-S6K1 and MK2 as HIV-responsive kinases required for optimal infection of CD4+ T cells

Retrovirology, 2018-07, Vol.15 (1), p.44-44, Article 44

2018

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HIV-1 Resistance to CCR5 Antagonists Associated with Highly Efficient Use of CCR5 and Altered Tropism on Primary CD4+ T Cells

Journal of Virology, 2010-07, Vol.84 (13), p.6505-6514

2010

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Entry inhibitors and their use in the treatment of HIV-1 infection

Antiviral research, 2013-05, Vol.98 (2), p.158-170

2013

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The HIV-1 Viral Protease Is Activated during Assembly and Budding Prior to Particle Release

Journal of virology, 2022-05, Vol.96 (9), p.e0219821-e0219821

2022

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High sensitivity detection and sorting of infectious human immunodeficiency virus (HIV-1) particles by flow virometry

Virology (New York, N.Y.), 2017-05, Vol.505, p.80-90

2017

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Primary Infection by a Human Immunodeficiency Virus with Atypical Coreceptor Tropism

Journal of Virology, 2011-10, Vol.85 (20), p.10669-10681

2011

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Diminished Proliferation of Human Immunodeficiency Virus-Specific CD4+ T Cells Is Associated with Diminished Interleukin-2 (IL-2) Production and Is Recovered by Exogenous IL-2

Journal of Virology, 2003-10, Vol.77 (20), p.10900-10909

2003

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The NIH Somatic Cell Genome Editing program

Nature (London), 2021-04, Vol.592 (7853), p.195-204

2021

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Multifaceted Mechanisms of HIV Inhibition and Resistance to CCR5 Inhibitors PSC-RANTES and Maraviroc

Antimicrobial Agents and Chemotherapy, 2013-06, Vol.57 (6), p.2640-2650

2013

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The histone deacetylase inhibitor vorinostat (SAHA) increases the susceptibility of uninfected CD4+ T cells to HIV by increasing the kinetics and efficiency of postentry viral events

Journal of virology, 2014-09, Vol.88 (18), p.10803-10812

2014

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Human Immunodeficiency Virus Viremia Induces Plasmacytoid Dendritic Cell Activation In Vivo and Diminished Alpha Interferon Production In Vitro

Journal of Virology, 2008-04, Vol.82 (8), p.3997-4006

2008

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Changes in Paracrine Interleukin-2 Requirement, CCR7 Expression, Frequency, and Cytokine Secretion of Human Immunodeficiency Virus-Specific CD4+ T Cells Are a Consequence of Antigen Load

Journal of Virology, 2007-03, Vol.81 (6), p.2713-2725

2007

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Comparisons of CD8+ T Cells Specific for Human Immunodeficiency Virus, Hepatitis C Virus, and Cytomegalovirus Reveal Differences in Frequency, Immunodominance, Phenotype, and Interleukin-2 Responsiveness

Journal of Virology, 2009-03, Vol.83 (6), p.2728-2742

2009

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Extracellular vesicles as carriers of viruses

ExRNA, 2021-12, Vol.3, p.13-13

2021

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Entry inhibitors in the treatment of HIV-1 infection

Analysis of Individual Viral Particles by Flow Virometry

Premature Activation of the HIV-1 Protease Is Influenced by Polymorphisms in the Hinge Region

Extracellular vesicles carry SARS‐CoV‐2 spike protein and serve as decoys for neutralizing antibodies

Transmitted/founder and chronic subtype C HIV-1 use CD4 and CCR5 receptors with equal efficiency and are not inhibited by blocking the integrin α4β7

Engineering HIV-resistant human CD4+ T cells with CXCR4-specific zinc-finger nucleases

Phenotypic and Immunologic Comparison of Clade B Transmitted/Founder and Chronic HIV-1 Envelope Glycoproteins

A Maraviroc-Resistant HIV-1 with Narrow Cross-Resistance to Other CCR5 Antagonists Depends on both N-Terminal and Extracellular Loop Domains of Drug-Bound CCR5

Molecular basis for positive and negative signaling by the natural killer cell receptor 2B4 (CD244)

COVID-19 and Cardiovascular Disease: From Bench to Bedside

HIV: cell binding and entry

Global phosphoproteomics of CCR5-tropic HIV-1 signaling reveals reprogramming of cellular protein production pathways and identifies p70-S6K1 and MK2 as HIV-responsive kinases required for optimal infection of CD4+ T cells

HIV-1 Resistance to CCR5 Antagonists Associated with Highly Efficient Use of CCR5 and Altered Tropism on Primary CD4+ T Cells

Entry inhibitors and their use in the treatment of HIV-1 infection

The HIV-1 Viral Protease Is Activated during Assembly and Budding Prior to Particle Release

High sensitivity detection and sorting of infectious human immunodeficiency virus (HIV-1) particles by flow virometry

Primary Infection by a Human Immunodeficiency Virus with Atypical Coreceptor Tropism

Diminished Proliferation of Human Immunodeficiency Virus-Specific CD4+ T Cells Is Associated with Diminished Interleukin-2 (IL-2) Production and Is Recovered by Exogenous IL-2

The NIH Somatic Cell Genome Editing program

Multifaceted Mechanisms of HIV Inhibition and Resistance to CCR5 Inhibitors PSC-RANTES and Maraviroc

The histone deacetylase inhibitor vorinostat (SAHA) increases the susceptibility of uninfected CD4+ T cells to HIV by increasing the kinetics and efficiency of postentry viral events

Human Immunodeficiency Virus Viremia Induces Plasmacytoid Dendritic Cell Activation In Vivo and Diminished Alpha Interferon Production In Vitro

Changes in Paracrine Interleukin-2 Requirement, CCR7 Expression, Frequency, and Cytokine Secretion of Human Immunodeficiency Virus-Specific CD4+ T Cells Are a Consequence of Antigen Load

Comparisons of CD8+ T Cells Specific for Human Immunodeficiency Virus, Hepatitis C Virus, and Cytomegalovirus Reveal Differences in Frequency, Immunodominance, Phenotype, and Interleukin-2 Responsiveness

Extracellular vesicles as carriers of viruses

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