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Tetanus toxin is a zinc protein and its inhibition of neurotransmitter release and protease activity depend on zinc

The EMBO journal, 1992-10, Vol.11 (10), p.3577-3583

1992

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Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia

The Journal of physiology, 1998-03, Vol.507 (2), p.405-414

1998

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Nitric oxide transforms serotonin into an inactive form and this affects neuromodulation

Neuroscience, 1999-01, Vol.93 (2), p.597-603

1999

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Non vesicular release of neurotransmitter

Physiological reviews, 1982-07, Vol.62 (3), p.857-893

Tauc, L

1982

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Opposite Actions of Nitric Oxide on Cholinergic Synapses: Which Pathways?

Proceedings of the National Academy of Sciences - PNAS, 1996-08, Vol.93 (16), p.8721-8726

1996

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Diversity of the modes of activity of the nerve cells of the disconnected ganglion of Aplysia

Comptes rendus des seances de la Societe de biologie et de ses filiales, 1960, Vol.154, p.17-21

TAUC, L

1960

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Quantal neurotransmitter release: Vesicular or not vesicular?

Neurophysiology (New York), 1997-07, Vol.29 (4-5), p.219-226

Tauc, L.

1997

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A role for the interchain disulfide or its participating thiols in the internalization of botulinum neurotoxin A revealed by a toxin derivative that binds to ecto-acceptors and inhibits transmitter release intracellularly

The Journal of biological chemistry, 1993-10, Vol.268 (28), p.20838-20844

1993

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Control of the calcium concentration involved in acetylcholine release and its facilitation: an additional role for synaptic vesicles?

Neuroscience, 1998-07, Vol.85 (1), p.85-91

1998

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N- and P-type Ca(2 plus) channels are involved in acetylcholine release at a neuroneuronal synapse: Only the N-type channel is the target of neuromodulators

Proceedings of the National Academy of Sciences - PNAS, 1994-05, Vol.91 (11), p.4771

1994

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Presynaptic mechanisms regulating Ca2+ concentration triggering acetylcholine release at an identified neuro-neuronal synapse of Aplysia

Neuroscience, 1994-11, Vol.63 (2), p.405-414

1994

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NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G

The Journal of physiology, 1996-06, Vol.493 (Pt 3), p.769-784

1996

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A nitric oxide synthase activity is involved in the modulation of acetylcholine release in Aplysia ganglion neurons: A histological, voltammetric and electrophysiological study

Neuroscience, 1995, Vol.69 (3), p.985-995

1995

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Minimal essential domains specifying toxicity of the light chains of tetanus toxin and botulinum neurotoxin type A

The Journal of biological chemistry, 1992-07, Vol.267 (21), p.14721-14729

1992

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Histamine and FLRFamide regulate acetylcholine release at an identified synapse in Aplysia in opposite ways

The Journal of physiology, 1990-10, Vol.429 (1), p.147-168

1990

Volltextzugriff (PDF)

Neurotransmitter Release is Blocked Intracellularly by Botulinum Neurotoxin, and this Requires Uptake of Both Toxin Polypeptides by a Process Mediated by the Larger Chain

Proceedings of the National Academy of Sciences - PNAS, 1988-06, Vol.85 (11), p.4090-4094

1988

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N- and P-Type Ca2+Channels are Involved in Acetylcholine Release at a Neuroneuronal Synapse: Only the N-Type Channel is the Target of Neuromodulators

Proceedings of the National Academy of Sciences - PNAS, 1994-05, Vol.91 (11), p.4771-4775

1994

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Presynaptic actions of curare and atropine on quantal acetylcholine release at a central synapse of Aplysia

The Journal of physiology, 1987-07, Vol.388 (1), p.665-680

1987

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Activation of protein kinase C by presynaptic FLRFamide receptors facilitates transmitter release at an aplysia cholinergic synapse

Neuron (Cambridge, Mass.), 1990-10, Vol.5 (4), p.479-486

1990

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Desensitization of cholinergic receptors by acetylcholine in the central neurons of gastropods

Journal de physiologie, 1963, Vol.55, p.177

1963

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The central neuron of Mollusca studied with the "voltage clamp" method

Journal de physiologie, 1962-03, Vol.54, p.415

1962

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Acetylcholine as a probable transmitter of synaptic excitation in the central nervous system of Mollusca

Journal de physiologie, 1962-03, Vol.54, p.416

1962

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Acetylcholine as a possible transmittor of synaptic inhibition in Aplysia

Comptes rendus hebdomadaires des séances de l'Académie des sciences, 1960-12, Vol.251, p.3076-3078

1960

Volltextzugriff (PDF)

On the partial distribution of efferent influxes in the branchings of a non-myelinized axon

Journal de physiologie, 1961-03, Vol.53, p.483

1961

Volltextzugriff (PDF)

Acetylcholine as a probable transmittor of synaptic inhibition in the central nervous system of Mollusca

Journal de physiologie, 1961-03, Vol.53, p.482

1961

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Tetanus toxin is a zinc protein and its inhibition of neurotransmitter release and protease activity depend on zinc

Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia

Nitric oxide transforms serotonin into an inactive form and this affects neuromodulation

Non vesicular release of neurotransmitter

Opposite Actions of Nitric Oxide on Cholinergic Synapses: Which Pathways?

Diversity of the modes of activity of the nerve cells of the disconnected ganglion of Aplysia

Quantal neurotransmitter release: Vesicular or not vesicular?

A role for the interchain disulfide or its participating thiols in the internalization of botulinum neurotoxin A revealed by a toxin derivative that binds to ecto-acceptors and inhibits transmitter release intracellularly

Control of the calcium concentration involved in acetylcholine release and its facilitation: an additional role for synaptic vesicles?

N- and P-type Ca(2 plus) channels are involved in acetylcholine release at a neuroneuronal synapse: Only the N-type channel is the target of neuromodulators

Presynaptic mechanisms regulating Ca2+ concentration triggering acetylcholine release at an identified neuro-neuronal synapse of Aplysia

NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G

A nitric oxide synthase activity is involved in the modulation of acetylcholine release in Aplysia ganglion neurons: A histological, voltammetric and electrophysiological study

Minimal essential domains specifying toxicity of the light chains of tetanus toxin and botulinum neurotoxin type A

Histamine and FLRFamide regulate acetylcholine release at an identified synapse in Aplysia in opposite ways

Neurotransmitter Release is Blocked Intracellularly by Botulinum Neurotoxin, and this Requires Uptake of Both Toxin Polypeptides by a Process Mediated by the Larger Chain

N- and P-Type Ca2+Channels are Involved in Acetylcholine Release at a Neuroneuronal Synapse: Only the N-Type Channel is the Target of Neuromodulators

Presynaptic actions of curare and atropine on quantal acetylcholine release at a central synapse of Aplysia

Activation of protein kinase C by presynaptic FLRFamide receptors facilitates transmitter release at an aplysia cholinergic synapse

Desensitization of cholinergic receptors by acetylcholine in the central neurons of gastropods

The central neuron of Mollusca studied with the "voltage clamp" method

Acetylcholine as a probable transmitter of synaptic excitation in the central nervous system of Mollusca

Acetylcholine as a possible transmittor of synaptic inhibition in Aplysia

On the partial distribution of efferent influxes in the branchings of a non-myelinized axon

Acetylcholine as a probable transmittor of synaptic inhibition in the central nervous system of Mollusca

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