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Early autophagic response in a novel knock-in model of Huntington disease

Human molecular genetics, 2010-10, Vol.19 (19), p.3702-3720

2010

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Differential recruitment of UBQLN2 to nuclear inclusions in the polyglutamine diseases HD and SCA3

Neurobiology of disease, 2015-10, Vol.82, p.281-288

2015

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Longitudinal Evaluation of the Hdh(CAG)150 Knock-In Murine Model of Huntington's Disease

The Journal of neuroscience, 2007-08, Vol.27 (34), p.8989-8998

2007

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Localization of mGluR1a-like immunoreactivity and mGluR5-like immunoreactivity in identified populations of striatal neurons

Brain research, 1998-01, Vol.780 (2), p.210-217

1998

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Differential effects of delayed aging on phenotype and striatal pathology in a murine model of Huntington disease

The Journal of neuroscience, 2014-11, Vol.34 (47), p.15658-15668

2014

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Evaluation of tetrathiomolybdate in the R6/2 model of Huntington disease

Neuroscience letters, 2009-03, Vol.452 (1), p.60-62

2009

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Lack of efficacy of NMDA receptor-NR2B selective antagonists in the R6/2 model of Huntington disease

Experimental neurology, 2010-10, Vol.225 (2), p.402-407

2010

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Treating Mouse Models of Huntington Disease

Neuropsychopharmacology (New York, N.Y.), 2011-11, Vol.36 (12), p.2373-2374

2011

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Hprt(CAG)146 mice: Age of onset of behavioral abnormalities, time course of neuronal intranuclear inclusion accumulation, neurotransmitter marker alterations, mitochondrial function markers, and susceptibility to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

Journal of comparative neurology (1911), 2003-10, Vol.465 (2), p.205-219

2003

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The de-ubiquitinating enzyme ataxin-3 does not modulate disease progression in a knock-in mouse model of Huntington disease

Journal of Huntington's disease, 2013-01, Vol.2 (2), p.201-215

2013

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Ectopically Expressed CAG Repeats Cause Intranuclear Inclusions and a Progressive Late Onset Neurological Phenotype in the Mouse

Cell, 1997-12, Vol.91 (6), p.753-763

1997

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Nucleocytoplasmic transport signals affect the age at onset of abnormalities in knock-in mice expressing polyglutamine within an ectopic protein context

Human molecular genetics, 2003-07, Vol.12 (13), p.1621-1629

2003

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Neurological abnormalities in a knock-in mouse model of Huntington's disease

Human molecular genetics, 2001-01, Vol.10 (2), p.137-144

2001

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Allelic series of Huntington's disease knock-in mice reveals expression discorrelates

Human molecular genetics, 2016-04, Vol.25 (8), p.1619-1636

2016

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Mitochondrial calcium uptake capacity as a therapeutic target in the R6/2 mouse model of Huntington's disease

Human molecular genetics, 2010-09, Vol.19 (17), p.3354-3371

2010

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Dystonia Associated With Mutation of the Neuronal Sodium Channel Scn8a and Identification of the Modifier Locus Scnm1 on Mouse Chromosome 3

Human molecular genetics, 1999-03, Vol.8 (3), p.471-479

1999

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Excitatory amino acid binding sites in the trigeminal principal sensory and spinal trigeminal nuclei of the rat

Neuroscience letters, 1992-07, Vol.141 (1), p.79-83

1992

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Longitudinal Evaluation of the Hdh super((CAG)150) Knock-In Murine Model of Huntington's Disease

The Journal of neuroscience, 2007-08, Vol.27 (34), p.8989-8998

2007

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Localization of striatal excitatory amino acid binding site subtypes to striatonigral projection neurons

Brain research, 1992-10, Vol.594 (1), p.165-170

1992

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AMPA and NMDA binding sites in the hypothalamic lateral tuberal nucleus: implications for Huntington's disease

Neurology, 1993-08, Vol.43 (8), p.1593-1593

1993

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Studies on the coexistence of serotonin and substance P in the brainstem of the rat

Greene, Sara Tallaksen

1991

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Early autophagic response in a novel knock-in model of Huntington disease

Differential recruitment of UBQLN2 to nuclear inclusions in the polyglutamine diseases HD and SCA3

Longitudinal Evaluation of the Hdh(CAG)150 Knock-In Murine Model of Huntington's Disease

Localization of mGluR1a-like immunoreactivity and mGluR5-like immunoreactivity in identified populations of striatal neurons

Differential effects of delayed aging on phenotype and striatal pathology in a murine model of Huntington disease

Evaluation of tetrathiomolybdate in the R6/2 model of Huntington disease

Lack of efficacy of NMDA receptor-NR2B selective antagonists in the R6/2 model of Huntington disease

Treating Mouse Models of Huntington Disease

Hprt(CAG)146 mice: Age of onset of behavioral abnormalities, time course of neuronal intranuclear inclusion accumulation, neurotransmitter marker alterations, mitochondrial function markers, and susceptibility to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

The de-ubiquitinating enzyme ataxin-3 does not modulate disease progression in a knock-in mouse model of Huntington disease

Ectopically Expressed CAG Repeats Cause Intranuclear Inclusions and a Progressive Late Onset Neurological Phenotype in the Mouse

Nucleocytoplasmic transport signals affect the age at onset of abnormalities in knock-in mice expressing polyglutamine within an ectopic protein context

Neurological abnormalities in a knock-in mouse model of Huntington's disease

Allelic series of Huntington's disease knock-in mice reveals expression discorrelates

Mitochondrial calcium uptake capacity as a therapeutic target in the R6/2 mouse model of Huntington's disease

Dystonia Associated With Mutation of the Neuronal Sodium Channel Scn8a and Identification of the Modifier Locus Scnm1 on Mouse Chromosome 3

Excitatory amino acid binding sites in the trigeminal principal sensory and spinal trigeminal nuclei of the rat

Longitudinal Evaluation of the Hdh super((CAG)150) Knock-In Murine Model of Huntington's Disease

Localization of striatal excitatory amino acid binding site subtypes to striatonigral projection neurons

AMPA and NMDA binding sites in the hypothalamic lateral tuberal nucleus: implications for Huntington's disease

Studies on the coexistence of serotonin and substance P in the brainstem of the rat

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