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Critical pathways in cellular senescence and immortalization revealed by gene expression profiling

Oncogene, 2008-10, Vol.27 (46), p.5975-5987

2008

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Breast cancer and other neoplasms in women with neurofibromatosis type 1: A retrospective review of cases in the Detroit metropolitan area

American journal of medical genetics. Part A, 2012-12, Vol.158A (12), p.3061-3064

2012

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Control of Angiogenesis in Fibroblasts by p53 Regulation of Thrombospondin-1

Science (American Association for the Advancement of Science), 1994-09, Vol.265 (5178), p.1582-1584

1994

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PolyADP-ribose polymerase is a coactivator for AP-2-mediated transcriptional activation

Nucleic acids research, 1999-02, Vol.27 (3), p.866

1999

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Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles

Cell, 1992-09, Vol.70 (6), p.937-948

1992

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In vitro growth characteristics of embryo fibroblasts isolated from p53-deficient mice

Oncogene, 1993-09, Vol.8 (9), p.2457-2467

1993

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Loss of AP-2 results in up-regulation of MCAM/MUC18 and an increase in tumor growth and metastasis of human melanoma cells

The Journal of biological chemistry, 1998-06, Vol.273 (26), p.16501-16508

1998

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Diverse system stresses: common mechanisms of chromosome fragmentation

Cell death & disease, 2011-06, Vol.2 (6), p.e178-e178

2011

Volltextzugriff (PDF)

An alternatively spliced mRNA from the AP-2 gene encodes a negative regulator of transcriptional activation by AP-2

Molecular and Cellular Biology, 1993-07, Vol.13 (7), p.4174-4185

1993

Volltextzugriff (PDF)

The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation

Molecular and Cellular Biology, 1987-06, Vol.7 (6), p.2104-2111

1987

Volltextzugriff (PDF)

Loss of AP-2 results in downregulation of c-KIT and enhancement of melanoma tumorigenicity and metastasis

The EMBO journal, 1998-08, Vol.17 (15), p.4358-4369

1998

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Spontaneous abnormalities in normal fibroblasts from patients with Li-Fraumeni cancer syndrome : aneuploidy and immortalization

Cancer research (Chicago, Ill.), 1990-12, Vol.50 (24), p.7979-7984

1990

Volltextzugriff (PDF)

Telomerase activity during spontaneous immortalization of Li-Fraumeni syndrome skin fibroblasts

Oncogene, 1998-08, Vol.17 (6), p.709-717

1998

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N-ras oncogene causes AP-2 transcriptional self-interference, which leads to transformation

Genes & development, 1994-06, Vol.8 (11), p.1258-1269

1994

Volltextzugriff (PDF)

Ras gene mutation and amplification in human nonmelanoma skin cancers

Molecular carcinogenesis, 1991, Vol.4 (3), p.196-202

1991

Volltextzugriff (PDF)

p53 and retinoid chemoprevention of oral carcinogenesis

Cancer research (Chicago, Ill.), 1995-01, Vol.55 (1), p.16-19

1995

Volltextzugriff (PDF)

Analysis of genomic instability in Li-Fraumeni fibroblasts with germline p53 mutations

Oncogene, 1996-06, Vol.12 (11), p.2267-2278

1996

Volltextzugriff (PDF)

Cloning and characterization of the Drosophila homologue of the AP-2 transcription factor

Oncogene, 1998-10, Vol.17 (15), p.1911-1922

1998

Volltextzugriff (PDF)

Visualization of the timing of gene amplification during multistep head and neck tumorigenesis

Cancer research (Chicago, Ill.), 2000-11, Vol.60 (22), p.6496-6502

2000

Volltextzugriff (PDF)

The p53 tumor suppressor gene inhibits angiogenesis by stimulating the production of thrombospondin

Cold Spring Harbor Symposia on Quantitative Biology, 1994, Vol.59, p.483

1994

Volltextzugriff (PDF)

The genomic structure of the human AP-2 transcription factor

Nucleic acids research, 1994-04, Vol.22 (8), p.1413-1420

1994

Volltextzugriff (PDF)

Diverse system stresses: common mechanisms of chromosome fragmentation

Cell death & disease, 2011-06, Vol.2 (6), p.e178-e178

2011

Volltextzugriff (PDF)

Functional interaction between a RARE and an AP-2 binding site in the regulation of the human HOX A4 gene promoter

Nucleic acids research, 1996-07, Vol.24 (14), p.2849

1996

Volltextzugriff (PDF)

Germ Line p53 Mutations in a Familial Syndrome of Breast Cancer, Sarcomas, and Other Neoplasms

Science (American Association for the Advancement of Science), 1990-11, Vol.250 (4985), p.1233-1238

1990

Volltextzugriff (PDF)

The Li–Fraumeni Syndrome: From Clinical Epidemiology to Molecular Genetics

American journal of epidemiology, 1992-01, Vol.135 (2), p.190-199

1992

Volltextzugriff (PDF)

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Critical pathways in cellular senescence and immortalization revealed by gene expression profiling

Breast cancer and other neoplasms in women with neurofibromatosis type 1: A retrospective review of cases in the Detroit metropolitan area

Control of Angiogenesis in Fibroblasts by p53 Regulation of Thrombospondin-1

PolyADP-ribose polymerase is a coactivator for AP-2-mediated transcriptional activation

Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles

In vitro growth characteristics of embryo fibroblasts isolated from p53-deficient mice

Loss of AP-2 results in up-regulation of MCAM/MUC18 and an increase in tumor growth and metastasis of human melanoma cells

Diverse system stresses: common mechanisms of chromosome fragmentation

An alternatively spliced mRNA from the AP-2 gene encodes a negative regulator of transcriptional activation by AP-2

The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation

Loss of AP-2 results in downregulation of c-KIT and enhancement of melanoma tumorigenicity and metastasis

Spontaneous abnormalities in normal fibroblasts from patients with Li-Fraumeni cancer syndrome : aneuploidy and immortalization

Telomerase activity during spontaneous immortalization of Li-Fraumeni syndrome skin fibroblasts

N-ras oncogene causes AP-2 transcriptional self-interference, which leads to transformation

Ras gene mutation and amplification in human nonmelanoma skin cancers

p53 and retinoid chemoprevention of oral carcinogenesis

Analysis of genomic instability in Li-Fraumeni fibroblasts with germline p53 mutations

Cloning and characterization of the Drosophila homologue of the AP-2 transcription factor

Visualization of the timing of gene amplification during multistep head and neck tumorigenesis

The p53 tumor suppressor gene inhibits angiogenesis by stimulating the production of thrombospondin

The genomic structure of the human AP-2 transcription factor

Diverse system stresses: common mechanisms of chromosome fragmentation

Functional interaction between a RARE and an AP-2 binding site in the regulation of the human HOX A4 gene promoter

Germ Line p53 Mutations in a Familial Syndrome of Breast Cancer, Sarcomas, and Other Neoplasms

The Li–Fraumeni Syndrome: From Clinical Epidemiology to Molecular Genetics

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