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Behçet’s syndrome as a tool to dissect the mechanisms of thrombo‐inflammation: clinical and pathogenetic aspects

Clinical and experimental immunology, 2019-03, Vol.195 (3), p.322-333

2019

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Management of Psychosis in Parkinson’s Disease: Emphasizing Clinical Subtypes and Pathophysiological Mechanisms of the Condition

Parkinson's disease, 2017-01, Vol.2017 (2017), p.1-18

2017

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Erythrocyte oxidative stress is associated with cell deformability in patients with retinal vein occlusion

Journal of thrombosis and haemostasis, 2016-11, Vol.14 (11), p.2287-2297

2016

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Changes in glial cell phenotypes precede overt neurofibrillary tangle formation, correlate with markers of cortical cell damage, and predict cognitive status of individuals at Braak III-IV stages

Acta neuropathologica communications, 2022-05, Vol.10 (1), p.72-20, Article 72

2022

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Altered redox status in the blood of psoriatic patients: involvement of NADPH oxidase and role of anti-TNF-α therapy

Redox report : communications in free radical research, 2013-05, Vol.18 (3), p.100-106

2013

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Tau Oligomer-Containing Synapse Elimination by Microglia and Astrocytes in Alzheimer Disease

JAMA neurology, 2023-11, Vol.80 (11), p.1209

2023

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In situ seeding assay: A novel technique for direct tissue localization of bioactive tau

Journal of neuropathology and experimental neurology, 2024-10, Vol.83 (10), p.870-881

2024

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Diagnostic delay in narcolepsy type 1: combining the patients' and the doctors' perspectives

Journal of sleep research, 2016-12, Vol.25 (6), p.709-715

2016

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New Symptomatic Treatments for the Management of Motor and Nonmotor Symptoms of Parkinson's Disease

International review of neurobiology, 2017, Vol.132, p.407-452

2017

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Depression and Anxiety in Parkinson's Disease

International review of neurobiology, 2017, Vol.133, p.623-655

2017

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Optimizing MSLT Specificity in Narcolepsy With Cataplexy

Sleep (New York, N.Y.), 2017-12, Vol.40 (12)

2017

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Rapid communication: a vegetable oil extract restores redox status in fibroblasts from psoriatic patients

Journal of biological regulators and homeostatic agents, 2016-04, Vol.30 (2 Suppl 3), p.129-131

2016

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Serum Uric Acid Levels and Non-Motor Symptoms in Parkinson’s Disease

Journal of Parkinson's disease, 2020-01, Vol.10 (3), p.1003-1010

2020

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Treatment of Nonmotor Symptoms in Parkinson's Disease

International review of neurobiology, 2017, Vol.132, p.361-379

2017

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Rationalization of the effects of mutations on peptide and protein aggregation rates

Nature (London), 2003-08, Vol.424 (6950), p.805-808

2003

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Investigation of the effects of copper ions on protein aggregation using a model system

Cellular and molecular life sciences : CMLS, 2004-04, Vol.61 (7-8), p.982-991

2004

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Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

Nature (London), 2002-04, Vol.416 (6880), p.507-511

2002

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Rotigotine transdermal patch and sleep in Parkinson’s disease: where are we now?

NPJ Parkinson's Disease, 2017-09, Vol.3 (1), p.28-9, Article 28

2017

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Insights into acylphosphatase structure and catalytic mechanism

Cellular and molecular life sciences : CMLS, 1997-02, Vol.53 (2), p.141-151

1997

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Designing Conditions for in vitro Formation of Amyloid Protofilaments and Fibrils

Proceedings of the National Academy of Sciences - PNAS, 1999-03, Vol.96 (7), p.3590-3594

1999

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Studies of the Aggregation of Mutant Proteins in vitro Provide Insights into the Genetics of Amyloid Diseases

Proceedings of the National Academy of Sciences - PNAS, 2002-12, Vol.99 (suppl_4), p.16419-16426

2002

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Assessing the role of aromatic residues in the amyloid aggregation of human muscle acylphosphatase

Protein science, 2006-04, Vol.15 (4), p.862-870

2006

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Stabilisation of α-helices by site-directed mutagenesis reveals the importance of secondary structure in the transition state for acylphosphatase folding

Journal of molecular biology, 2000-07, Vol.300 (3), p.633-647

2000

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Kinetic partitioning of protein folding and aggregation

Nature structural & molecular biology, 2002-02, Vol.9 (2), p.137-143

2002

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Aggregation of the Acylphosphatase from Sulfolobus solfataricus: the folded and partially unfolded states can both be precursors for amyloid formation

The Journal of biological chemistry, 2004-04, Vol.279 (14), p.14111-14119

2004

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Behçet’s syndrome as a tool to dissect the mechanisms of thrombo‐inflammation: clinical and pathogenetic aspects

Management of Psychosis in Parkinson’s Disease: Emphasizing Clinical Subtypes and Pathophysiological Mechanisms of the Condition

Erythrocyte oxidative stress is associated with cell deformability in patients with retinal vein occlusion

Changes in glial cell phenotypes precede overt neurofibrillary tangle formation, correlate with markers of cortical cell damage, and predict cognitive status of individuals at Braak III-IV stages

Altered redox status in the blood of psoriatic patients: involvement of NADPH oxidase and role of anti-TNF-α therapy

Tau Oligomer-Containing Synapse Elimination by Microglia and Astrocytes in Alzheimer Disease

In situ seeding assay: A novel technique for direct tissue localization of bioactive tau

Diagnostic delay in narcolepsy type 1: combining the patients' and the doctors' perspectives

New Symptomatic Treatments for the Management of Motor and Nonmotor Symptoms of Parkinson's Disease

Depression and Anxiety in Parkinson's Disease

Optimizing MSLT Specificity in Narcolepsy With Cataplexy

Rapid communication: a vegetable oil extract restores redox status in fibroblasts from psoriatic patients

Serum Uric Acid Levels and Non-Motor Symptoms in Parkinson’s Disease

Treatment of Nonmotor Symptoms in Parkinson's Disease

Rationalization of the effects of mutations on peptide and protein aggregation rates

Investigation of the effects of copper ions on protein aggregation using a model system

Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

Rotigotine transdermal patch and sleep in Parkinson’s disease: where are we now?

Insights into acylphosphatase structure and catalytic mechanism

Designing Conditions for in vitro Formation of Amyloid Protofilaments and Fibrils

Studies of the Aggregation of Mutant Proteins in vitro Provide Insights into the Genetics of Amyloid Diseases

Assessing the role of aromatic residues in the amyloid aggregation of human muscle acylphosphatase

Stabilisation of α-helices by site-directed mutagenesis reveals the importance of secondary structure in the transition state for acylphosphatase folding

Kinetic partitioning of protein folding and aggregation

Aggregation of the Acylphosphatase from Sulfolobus solfataricus: the folded and partially unfolded states can both be precursors for amyloid formation

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