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Effects of hypercapnia on the lung

The Journal of physiology, 2017-04, Vol.595 (8), p.2431-2437

2017

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Recovering from a pandemic: pulmonary fibrosis after SARS-CoV-2 infection

European respiratory review, 2021-12, Vol.30 (162), p.210194

2021

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Orphan Nuclear Receptor Family 4A (NR4A) Members NR4A2 and NR4A3 Selectively Modulate Elements of the Monocyte Response to Buffered Hypercapnia

International journal of molecular sciences, 2024-03, Vol.25 (5), p.2852

2024

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Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection

The Journal of clinical investigation, 2016-04, Vol.126 (4), p.1566-1580

2016

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High CO2 Levels Cause Skeletal Muscle Atrophy via AMP-activated Kinase (AMPK), FoxO3a Protein, and Muscle-specific Ring Finger Protein 1 (MuRF1)

The Journal of biological chemistry, 2015-04, Vol.290 (14), p.9183-9194

2015

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Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1

American journal of physiology. Lung cellular and molecular physiology, 2009-12, Vol.297 (6), p.L1120-L1130

2009

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Linear ubiquitin assembly complex regulates lung epithelial-driven responses during influenza infection

The Journal of clinical investigation, 2020-03, Vol.130 (3), p.1301-1314

2020

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Editorial: Elevated Carbon Dioxide Sensing and Physiologic Effects

Frontiers in physiology, 2022-04, Vol.13, p.894222-894222

2022

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Hypercapnia increases airway smooth muscle contractility via caspase-7-mediated miR-133a-RhoA signaling

Science translational medicine, 2018-09, Vol.10 (457)

2018

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Hypercapnia Regulates Gene Expression and Tissue Function

Frontiers in physiology, 2020-11, Vol.11, p.598122-598122

2020

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Hypercapnia Impairs Na,K-ATPase Function by Inducing Endoplasmic Reticulum Retention of the β-Subunit of the Enzyme in Alveolar Epithelial Cells

International journal of molecular sciences, 2020-02, Vol.21 (4), p.1467

2020

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Hypercapnia impairs lung neutrophil function and increases mortality in murine pseudomonas pneumonia

American journal of respiratory cell and molecular biology, 2013-11, Vol.49 (5), p.821-828

2013

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The Emerging Role of the Ubiquitin Proteasome in Pulmonary Biology and Disease

American journal of respiratory and critical care medicine, 2013-09, Vol.188 (5), p.530-537

2013

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Activation of p21 limits acute lung injury and induces early senescence after acid aspiration and mechanical ventilation

Translational research : the journal of laboratory and clinical medicine, 2021-07, Vol.233, p.104-116

2021

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Transcriptional Profiling of Monocytes Deficient in Nuclear Orphan Receptors NR4A2 and NR4A3 Reveals Distinct Signalling Roles Related to Antigen Presentation and Viral Response

Frontiers in immunology, 2021-06, Vol.12, p.676644-676644

2021

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Hypercapnia: An Aggravating Factor in Asthma

Journal of clinical medicine, 2020-10, Vol.9 (10), p.3207

2020

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Elevated CO2 Levels Delay Skeletal Muscle Repair by Increasing Fatty Acid Oxidation

Frontiers in physiology, 2021-01, Vol.11

2021

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High CO2 levels impair alveolar epithelial function independently of pH

PloS one, 2007-11, Vol.2 (11), p.e1238

2007

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Elevated CO2 regulates the Wnt signaling pathway in mammals, Drosophila melanogaster and Caenorhabditis elegans

Scientific reports, 2019-12, Vol.9 (1), p.1-8, Article 18251

2019

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Evolutionary conserved role of c-Jun-N-terminal kinase in CO2-induced epithelial dysfunction

PloS one, 2012-10, Vol.7 (10), p.e46696

2012

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Pleural Hypercarbia After Lung Surgery Is Associated With Persistent Alveolopleural Fistulae

Chest, 2016, Vol.149 (1), p.220-227

2016

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The response to high CO2 levels requires the neuropeptide secretion component HID-1 to promote pumping inhibition

PLoS genetics, 2014-08, Vol.10 (8), p.e1004529

2014

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High CO2 Leads to Na,K-ATPase Endocytosis via c-Jun Amino-Terminal Kinase-Induced LMO7b Phosphorylation

Molecular and cellular biology, 2015-12, Vol.35 (23), p.3962-3973

2015

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AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis

The Journal of clinical investigation, 2008-02, Vol.118 (2), p.752-762

2008

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Ubiquitination and Proteolysis in Acute Lung Injury

Chest, 2012-03, Vol.141 (3), p.763-771

2012

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Effects of hypercapnia on the lung

Recovering from a pandemic: pulmonary fibrosis after SARS-CoV-2 infection

Orphan Nuclear Receptor Family 4A (NR4A) Members NR4A2 and NR4A3 Selectively Modulate Elements of the Monocyte Response to Buffered Hypercapnia

Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection

High CO2 Levels Cause Skeletal Muscle Atrophy via AMP-activated Kinase (AMPK), FoxO3a Protein, and Muscle-specific Ring Finger Protein 1 (MuRF1)

Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1

Linear ubiquitin assembly complex regulates lung epithelial-driven responses during influenza infection

Editorial: Elevated Carbon Dioxide Sensing and Physiologic Effects

Hypercapnia increases airway smooth muscle contractility via caspase-7-mediated miR-133a-RhoA signaling

Hypercapnia Regulates Gene Expression and Tissue Function

Hypercapnia Impairs Na,K-ATPase Function by Inducing Endoplasmic Reticulum Retention of the β-Subunit of the Enzyme in Alveolar Epithelial Cells

Hypercapnia impairs lung neutrophil function and increases mortality in murine pseudomonas pneumonia

The Emerging Role of the Ubiquitin Proteasome in Pulmonary Biology and Disease

Activation of p21 limits acute lung injury and induces early senescence after acid aspiration and mechanical ventilation

Transcriptional Profiling of Monocytes Deficient in Nuclear Orphan Receptors NR4A2 and NR4A3 Reveals Distinct Signalling Roles Related to Antigen Presentation and Viral Response

Hypercapnia: An Aggravating Factor in Asthma

Elevated CO2 Levels Delay Skeletal Muscle Repair by Increasing Fatty Acid Oxidation

High CO2 levels impair alveolar epithelial function independently of pH

Elevated CO2 regulates the Wnt signaling pathway in mammals, Drosophila melanogaster and Caenorhabditis elegans

Evolutionary conserved role of c-Jun-N-terminal kinase in CO2-induced epithelial dysfunction

Pleural Hypercarbia After Lung Surgery Is Associated With Persistent Alveolopleural Fistulae

The response to high CO2 levels requires the neuropeptide secretion component HID-1 to promote pumping inhibition

High CO2 Leads to Na,K-ATPase Endocytosis via c-Jun Amino-Terminal Kinase-Induced LMO7b Phosphorylation

AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis

Ubiquitination and Proteolysis in Acute Lung Injury

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