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Bioenergetic regulation of microglia

Glia, 2018-06, Vol.66 (6), p.1200-1212

2018

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Bioenergetic and excitotoxic determinants of cofilactin rod formation

Journal of neurochemistry, 2024-05, Vol.168 (5), p.899-909

2024

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A tandem activity-based sensing and labeling strategy enables imaging of transcellular hydrogen peroxide signaling

Proceedings of the National Academy of Sciences - PNAS, 2021-03, Vol.118 (9)

2021

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Glia in brain energy metabolism: A perspective

Glia, 2018-06, Vol.66 (6), p.1134-1137

2018

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Microglial Activation in Stroke: Therapeutic Targets

Neurotherapeutics, 2010-10, Vol.7 (4), p.378-391

2010

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Superoxide and Non-ionotropic Signaling in Neuronal Excitotoxicity

Frontiers in neuroscience, 2020-09, Vol.4, p.861-861

2020

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Glucose availability limits microglial nitric oxide production

Journal of neurochemistry, 2021-12, Vol.159 (6), p.1008-1015

2021

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Bioenergetic state regulates innate inflammatory responses through the transcriptional co-repressor CtBP

Nature communications, 2017-09, Vol.8 (1), p.624-13, Article 624

2017

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Opportunities for the repurposing of PARP inhibitors for the therapy of non‐oncological diseases

British journal of pharmacology, 2018-01, Vol.175 (2), p.192-222

2018

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Targeted Lipid Profiling Discovers Plasma Biomarkers of Acute Brain Injury

PloS one, 2015-06, Vol.10 (6), p.e0129735-e0129735

2015

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NADPH oxidase is the primary source of superoxide induced by NMDA receptor activation

Nature neuroscience, 2009-07, Vol.12 (7), p.857-863

2009

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Cofilin-actin rod formation in neuronal processes after brain ischemia

PloS one, 2018-10, Vol.13 (10), p.e0198709-e0198709

2018

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Activation of neuronal NMDA receptors induces superoxide-mediated oxidative stress in neighboring neurons and astrocytes

The Journal of neuroscience, 2012-09, Vol.32 (37), p.12973-12978

2012

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Hyperglycemia promotes tissue plasminogen activator-induced hemorrhage by Increasing superoxide production

Annals of neurology, 2011-10, Vol.70 (4), p.583-590

2011

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Cofilactin rod formation mediates inflammation-induced neurite degeneration

Cell reports (Cambridge), 2024-03, Vol.43 (3), p.113914-113914, Article 113914

2024

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Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase

The Journal of clinical investigation, 2007-04, Vol.117 (4), p.910-918

2007

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Glucose and NADPH oxidase drive neuronal superoxide formation in stroke

Annals of neurology, 2008-12, Vol.64 (6), p.654-663

2008

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Hypoglycemia, brain energetics, and hypoglycemic neuronal death

Glia, 2007-09, Vol.55 (12), p.1280-1286

2007

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Minocycline Inhibits Poly(ADP-Ribose) Polymerase-1 at Nanomolar Concentrations

Proceedings of the National Academy of Sciences - PNAS, 2006-06, Vol.103 (25), p.9685-9690

2006

Volltextzugriff (PDF)

Poly(ADP-ribose) Polymerase-1-mediated Cell Death in Astrocytes Requires NAD+ Depletion and Mitochondrial Permeability Transition

The Journal of biological chemistry, 2004-04, Vol.279 (18), p.18895-18902

2004

Volltextzugriff (PDF)

Intracellular pH reduction prevents excitotoxic and ischemic neuronal death by inhibiting NADPH oxidase

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (46), p.E4362-E4368

2013

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Assessment at the single-cell level identifies neuronal glutathione depletion as both a cause and effect of ischemia-reperfusion oxidative stress

The Journal of neuroscience, 2015-05, Vol.35 (18), p.7143-7152

2015

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Microglial activation induced by brain trauma is suppressed by post-injury treatment with a PARP inhibitor

Journal of neuroinflammation, 2012-02, Vol.9 (1), p.31-31, Article 31

2012

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Blast-induced axonal degeneration in the rat cerebellum in the absence of head movement

Scientific reports, 2022-01, Vol.12 (1), p.143-143, Article 143

2022

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N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1−/− mouse

Annals of neurology, 2011-03, Vol.69 (3), p.509-520

2011

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Bioenergetic regulation of microglia

Bioenergetic and excitotoxic determinants of cofilactin rod formation

A tandem activity-based sensing and labeling strategy enables imaging of transcellular hydrogen peroxide signaling

Glia in brain energy metabolism: A perspective

Microglial Activation in Stroke: Therapeutic Targets

Superoxide and Non-ionotropic Signaling in Neuronal Excitotoxicity

Glucose availability limits microglial nitric oxide production

Bioenergetic state regulates innate inflammatory responses through the transcriptional co-repressor CtBP

Opportunities for the repurposing of PARP inhibitors for the therapy of non‐oncological diseases

Targeted Lipid Profiling Discovers Plasma Biomarkers of Acute Brain Injury

NADPH oxidase is the primary source of superoxide induced by NMDA receptor activation

Cofilin-actin rod formation in neuronal processes after brain ischemia

Activation of neuronal NMDA receptors induces superoxide-mediated oxidative stress in neighboring neurons and astrocytes

Hyperglycemia promotes tissue plasminogen activator-induced hemorrhage by Increasing superoxide production

Cofilactin rod formation mediates inflammation-induced neurite degeneration

Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase

Glucose and NADPH oxidase drive neuronal superoxide formation in stroke

Hypoglycemia, brain energetics, and hypoglycemic neuronal death

Minocycline Inhibits Poly(ADP-Ribose) Polymerase-1 at Nanomolar Concentrations

Poly(ADP-ribose) Polymerase-1-mediated Cell Death in Astrocytes Requires NAD+ Depletion and Mitochondrial Permeability Transition

Intracellular pH reduction prevents excitotoxic and ischemic neuronal death by inhibiting NADPH oxidase

Assessment at the single-cell level identifies neuronal glutathione depletion as both a cause and effect of ischemia-reperfusion oxidative stress

Microglial activation induced by brain trauma is suppressed by post-injury treatment with a PARP inhibitor

Blast-induced axonal degeneration in the rat cerebellum in the absence of head movement

N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1−/− mouse

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