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The importance of E-cadherin binding partners to evaluate the pathogenicity of E-cadherin missense mutations associated to HDGC

European journal of human genetics : EJHG, 2013-03, Vol.21 (3), p.301-309

2013

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The prevalence of PIK3CA mutations in gastric and colon cancer

European journal of cancer (1990), 2005-07, Vol.41 (11), p.1649-1654

2005

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Gastric cancer: New genetic developments

Journal of surgical oncology, 2005-06, Vol.90 (3), p.114-133

2005

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De novo expression of CD44 variants in sporadic and hereditary gastric cancer

Laboratory investigation, 2010-11, Vol.90 (11), p.1604-1614

2010

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ADP-ribosylation factor 6 mediates E-cadherin recovery by chemical chaperones

PloS one, 2011-08, Vol.6 (8), p.e23188-e23188

2011

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Epidermal growth factor receptor structural alterations in gastric cancer

BMC cancer, 2008-01, Vol.8 (1), p.10-10, Article 10

2008

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α-Catenin mediates initial E-cadherin-dependent cell-cell recognition and subsequent bond strengthening

Proceedings of the National Academy of Sciences - PNAS, 2008-11, Vol.105 (47), p.18331-18336

2008

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E-cadherin mutations and cell motility: A genotype–phenotype correlation

Experimental cell research, 2009-05, Vol.315 (8), p.1393-1402

2009

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A model to infer the pathogenic significance of CDH1 germline missense variants

Journal of molecular medicine (Berlin, Germany), 2006-12, Vol.84 (12), p.1023-1031

2006

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Helicobacter pylori Induces Gastric Epithelial Cell Invasion in a c-Met and Type IV Secretion System-dependent Manner

The Journal of biological chemistry, 2006-11, Vol.281 (46), p.34888-34896

2006

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Association between Functional EGF+61 Polymorphism and Glioma Risk

Clinical cancer research, 2007-05, Vol.13 (9), p.2621-2626

2007

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The intracellular E-cadherin germline mutation V832 M lacks the ability to mediate cell-cell adhesion and to suppress invasion

Oncogene, 2003-08, Vol.22 (36), p.5716-5719

2003

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Screening E-cadherin in gastric cancer families reveals germline mutations only in hereditary diffuse gastric cancer kindred

Human mutation, 2002-05, Vol.19 (5), p.510-517

2002

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E-Cadherin ( CDH1) and p53 rather than SMAD4 and Caspase-10 germline mutations contribute to genetic predisposition in Portuguese gastric cancer patients

European journal of cancer (1990), 2004-08, Vol.40 (12), p.1897-1903

2004

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Loss of functional E-cadherin renders cells more resistant to the apoptotic agent taxol in vitro

Experimental cell research, 2005-10, Vol.310 (1), p.99-104

2005

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Founder and recurrent CDH1 mutations in families with hereditary diffuse gastric cancer

JAMA : the journal of the American Medical Association, 2007-06, Vol.297 (21), p.2360

2007

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Genetic screening for familial gastric cancer

Hereditary cancer in clinical practice, 2004-05, Vol.2 (2), p.51-64, Article 51

2004

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E-cadherin impairment increases cell survival through Notch-dependent upregulation of Bcl-2

Human molecular genetics, 2012-01, Vol.21 (2), p.334-343

2012

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EGFR regulates RhoA-GTP dependent cell motility in E-cadherin mutant cells

Human molecular genetics, 2007-07, Vol.16 (13), p.1639-1647

2007

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Endoplasmic reticulum quality control: a new mechanism of E-cadherin regulation and its implication in cancer

Human molecular genetics, 2008-11, Vol.17 (22), p.3566-3576

2008

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Identification of CDH1 germline missense mutations associated with functional inactivation of the E-cadherin protein in young gastric cancer probands

Human molecular genetics, 2003-03, Vol.12 (5), p.575-582

2003

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E-cadherin germline missense mutations and cell phenotype: evidence for the independence of cell invasion on the motile capabilities of the cells

Human molecular genetics, 2003-11, Vol.12 (22), p.3007-3016

2003

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Mixed lineage kinase 3 gene mutations in mismatch repair deficient gastrointestinal tumours

Human molecular genetics, 2010-02, Vol.19 (4), p.697-706

2010

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Epistatic interactions: how strong in disease and evolution?

Trends in genetics, 2006-11, Vol.22 (11), p.581-585

2006

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The importance of E-cadherin binding partners to evaluate the pathogenicity of E-cadherin missense mutations associated to HDGC

The prevalence of PIK3CA mutations in gastric and colon cancer

Gastric cancer: New genetic developments

De novo expression of CD44 variants in sporadic and hereditary gastric cancer

ADP-ribosylation factor 6 mediates E-cadherin recovery by chemical chaperones

Epidermal growth factor receptor structural alterations in gastric cancer

α-Catenin mediates initial E-cadherin-dependent cell-cell recognition and subsequent bond strengthening

E-cadherin mutations and cell motility: A genotype–phenotype correlation

A model to infer the pathogenic significance of CDH1 germline missense variants

Helicobacter pylori Induces Gastric Epithelial Cell Invasion in a c-Met and Type IV Secretion System-dependent Manner

Association between Functional EGF+61 Polymorphism and Glioma Risk

The intracellular E-cadherin germline mutation V832 M lacks the ability to mediate cell-cell adhesion and to suppress invasion

Screening E-cadherin in gastric cancer families reveals germline mutations only in hereditary diffuse gastric cancer kindred

E-Cadherin ( CDH1) and p53 rather than SMAD4 and Caspase-10 germline mutations contribute to genetic predisposition in Portuguese gastric cancer patients

Loss of functional E-cadherin renders cells more resistant to the apoptotic agent taxol in vitro

Founder and recurrent CDH1 mutations in families with hereditary diffuse gastric cancer

Genetic screening for familial gastric cancer

E-cadherin impairment increases cell survival through Notch-dependent upregulation of Bcl-2

EGFR regulates RhoA-GTP dependent cell motility in E-cadherin mutant cells

Endoplasmic reticulum quality control: a new mechanism of E-cadherin regulation and its implication in cancer

Characterization of a Recurrent Germ Line Mutation of the E-Cadherin Gene: Implications for Genetic Testing and Clinical Management

Identification of CDH1 germline missense mutations associated with functional inactivation of the E-cadherin protein in young gastric cancer probands

E-cadherin germline missense mutations and cell phenotype: evidence for the independence of cell invasion on the motile capabilities of the cells

Mixed lineage kinase 3 gene mutations in mismatch repair deficient gastrointestinal tumours

Epistatic interactions: how strong in disease and evolution?

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