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Ablation of Doublecortin-Like Kinase 1 in the Colonic Epithelium Exacerbates Dextran Sulfate Sodium-Induced Colitis

PloS one, 2015-08, Vol.10 (8), p.e0134212

2015

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Editorial: Novel immunotherapies to treat gastrointestinal solid tumor cancers

Frontiers in oncology, 2022-10, Vol.12, p.1043615-1043615

2022

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DCLK1 regulates pluripotency and angiogenic factors via microRNA-dependent mechanisms in pancreatic cancer

PloS one, 2013-09, Vol.8 (9), p.e73940-e73940

2013

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XMD8-92 inhibits pancreatic tumor xenograft growth via a DCLK1-dependent mechanism

Cancer letters, 2014-08, Vol.351 (1), p.151-161

2014

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Identification of a Novel Putative Gastrointestinal Stem Cell and Adenoma Stem Cell Marker, Doublecortin and CaM Kinase‐Like‐1, Following Radiation Injury and in Adenomatous Polyposis Coli/Multiple Intestinal Neoplasia Mice

Stem cells (Dayton, Ohio), 2008-03, Vol.26 (3), p.630-637

2008

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Small molecule kinase inhibitor LRRK2-IN-1 demonstrates potent activity against colorectal and pancreatic cancer through inhibition of doublecortin-like kinase 1

Molecular cancer, 2014-05, Vol.13 (1), p.103-103, Article 103

2014

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DCAMKL-1 Regulates Epithelial―Mesenchymal Transition in Human Pancreatic Cells through a miR-200a―Dependent Mechanism

Cancer research (Chicago, Ill.), 2011-03, Vol.71 (6), p.2328-2338

2011

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Doublecortin-like kinase 1 is elevated serologically in pancreatic ductal adenocarcinoma and widely expressed on circulating tumor cells

PloS one, 2015-02, Vol.10 (2), p.e0118933

2015

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Could Nutraceutical Approaches Possibly Attenuate the Cytokine Storm in COVID-19 Patients?

Frontiers in cellular and infection microbiology, 2021-04, Vol.11, p.667733

2021

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Nanoparticle-based delivery of siDCAMKL-1 increases microRNA-144 and inhibits colorectal cancer tumor growth via a Notch-1 dependent mechanism

Journal of nanobiotechnology, 2011-09, Vol.9 (1), p.40-40, Article 40

2011

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DCLK1 Monoclonal Antibody-Based CAR-T Cells as a Novel Treatment Strategy against Human Colorectal Cancers

Cancers, 2019-12, Vol.12 (1), p.54

2019

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Brief Report: Dclk1 Deletion in Tuft Cells Results in Impaired Epithelial Repair After Radiation Injury

Stem cells (Dayton, Ohio), 2014-03, Vol.32 (3), p.822-827

2014

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Doublecortin and CaM Kinase‐like‐1 and Leucine‐Rich‐Repeat‐Containing G‐Protein‐Coupled Receptor Mark Quiescent and Cycling Intestinal Stem Cells, Respectively

Stem cells (Dayton, Ohio), 2009-10, Vol.27 (10), p.2571-2579

2009

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Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury

Scientific reports, 2016-11, Vol.6 (1), p.37667, Article 37667

2016

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DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinoma

Oncotarget, 2015-02, Vol.6 (4), p.2193-2205

2015

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DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition

Oncotarget, 2014-10, Vol.5 (19), p.9269-9280

2014

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Hepatitis C Virus-Induced Cancer Stem Cell-Like Signatures in Cell Culture and Murine Tumor Xenografts

Journal of Virology, 2011-12, Vol.85 (23), p.12292-12303

2011

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Dietary Pectin Increases Intestinal Crypt Stem Cell Survival following Radiation Injury

PloS one, 2015-08, Vol.10 (8), p.e0135561

2015

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Fluvastatin interferes with hepatitis C virus replication via microtubule bundling and a doublecortin-like kinase-mediated mechanism

PloS one, 2013-11, Vol.8 (11), p.e80304

2013

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Dclk1+ small intestinal epithelial tuft cells display the hallmarks of quiescence and self-renewal

Oncotarget, 2015-10, Vol.6 (31), p.30876-30886

2015

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Identification of the putative intestinal stem cell marker doublecortin and CaM kinase-like-1 in Barrett's esophagus and esophageal adenocarcinoma

Journal of gastroenterology and hepatology, 2012-04, Vol.27 (4), p.773-780

2012

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Identification of a novel putative pancreatic stem/progenitor cell marker DCAMKL-1 in normal mouse pancreas

American journal of physiology: Gastrointestinal and liver physiology, 2010-08, Vol.299 (2), p.G303-G310

2010

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Reg IV Regulates Normal Intestinal and Colorectal Cancer Cell Susceptibility to Radiation-Induced Apoptosis

Gastroenterology (New York, N.Y. 1943), 2010-02, Vol.138 (2), p.616-626.e2

2010

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Inflammatory and oncogenic roles of a tumor stem cell marker doublecortin-like kinase (DCLK1) in virus-induced chronic liver diseases

Oncotarget, 2015-08, Vol.6 (24), p.20327-20344

2015

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Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells

Molecular cancer, 2017-02, Vol.16 (1), p.30-30, Article 30

2017

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Ablation of Doublecortin-Like Kinase 1 in the Colonic Epithelium Exacerbates Dextran Sulfate Sodium-Induced Colitis

Editorial: Novel immunotherapies to treat gastrointestinal solid tumor cancers

DCLK1 regulates pluripotency and angiogenic factors via microRNA-dependent mechanisms in pancreatic cancer

XMD8-92 inhibits pancreatic tumor xenograft growth via a DCLK1-dependent mechanism

Identification of a Novel Putative Gastrointestinal Stem Cell and Adenoma Stem Cell Marker, Doublecortin and CaM Kinase‐Like‐1, Following Radiation Injury and in Adenomatous Polyposis Coli/Multiple Intestinal Neoplasia Mice

Small molecule kinase inhibitor LRRK2-IN-1 demonstrates potent activity against colorectal and pancreatic cancer through inhibition of doublecortin-like kinase 1

DCAMKL-1 Regulates Epithelial―Mesenchymal Transition in Human Pancreatic Cells through a miR-200a―Dependent Mechanism

Doublecortin-like kinase 1 is elevated serologically in pancreatic ductal adenocarcinoma and widely expressed on circulating tumor cells

Could Nutraceutical Approaches Possibly Attenuate the Cytokine Storm in COVID-19 Patients?

Nanoparticle-based delivery of siDCAMKL-1 increases microRNA-144 and inhibits colorectal cancer tumor growth via a Notch-1 dependent mechanism

DCLK1 Monoclonal Antibody-Based CAR-T Cells as a Novel Treatment Strategy against Human Colorectal Cancers

Brief Report: Dclk1 Deletion in Tuft Cells Results in Impaired Epithelial Repair After Radiation Injury

Doublecortin and CaM Kinase‐like‐1 and Leucine‐Rich‐Repeat‐Containing G‐Protein‐Coupled Receptor Mark Quiescent and Cycling Intestinal Stem Cells, Respectively

Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury

DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinoma

DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition

Hepatitis C Virus-Induced Cancer Stem Cell-Like Signatures in Cell Culture and Murine Tumor Xenografts

Dietary Pectin Increases Intestinal Crypt Stem Cell Survival following Radiation Injury

Fluvastatin interferes with hepatitis C virus replication via microtubule bundling and a doublecortin-like kinase-mediated mechanism

Dclk1+ small intestinal epithelial tuft cells display the hallmarks of quiescence and self-renewal

Identification of the putative intestinal stem cell marker doublecortin and CaM kinase-like-1 in Barrett's esophagus and esophageal adenocarcinoma

Identification of a novel putative pancreatic stem/progenitor cell marker DCAMKL-1 in normal mouse pancreas

Reg IV Regulates Normal Intestinal and Colorectal Cancer Cell Susceptibility to Radiation-Induced Apoptosis

Inflammatory and oncogenic roles of a tumor stem cell marker doublecortin-like kinase (DCLK1) in virus-induced chronic liver diseases

Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells

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