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DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice

Nature communications, 2015-11, Vol.6 (1), p.8897-8897, Article 8897

2015

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Robust Control of a Brain-Persisting Parasite through MHC I Presentation by Infected Neurons

Cell reports (Cambridge), 2019-06, Vol.27 (11), p.3254-3268.e8

2019

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Histone acetylation in neuronal (dys)function

Biomolecular concepts, 2016-05, Vol.7 (2), p.103-116

2016

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Neurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infection

PLoS pathogens, 2011-11, Vol.7 (11), p.e1002393-e1002393

2011

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Borna Disease Virus 1 Phosphoprotein Forms a Tetramer and Interacts with Host Factors Involved in DNA Double-Strand Break Repair and mRNA Processing

Viruses, 2022-10, Vol.14 (11), p.2358

2022

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IMPACT OF CHRONIC TOXOPLASMA GONDII INFECTION ON THE ONSET AND PROGRESSION OF ALZHEIMER’S DISEASE

IBRO neuroscience reports, 2023-10, Vol.15, p.S373-S373

2023

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Borna disease virus docks on neuronal DNA double-strand breaks to replicate and dampens neuronal activity

iScience, 2022-01, Vol.25 (1), p.103621-103621, Article 103621

2022

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Human Cytomegalovirus Infection Changes the Pattern of Surface Markers of Small Extracellular Vesicles Isolated From First Trimester Placental Long-Term Histocultures

Frontiers in cell and developmental biology, 2021-09, Vol.9 (689122), p.689122-689122

2021

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Proteomic Analysis Reveals Selective Impediment of Neuronal Remodeling upon Borna Disease Virus Infection

Journal of Virology, 2008-12, Vol.82 (24), p.12265-12279

2008

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High reproducibility of two-dimensional liquid chromatography using pH-driven fractionation with a pressure-resistant electrode

Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 2008-08, Vol.871 (1), p.125-129

2008

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Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β

Nature neuroscience, 2013-05, Vol.16 (5), p.613-621

2013

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Zika virus in semen and spermatozoa

The Lancet infectious diseases, 2016-10, Vol.16 (10), p.1106-1107

2016

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Real‐Time and High‐Resolution Monitoring of Neuronal Electrical Activity and pH Variations Based on the Co‐Integration of Nanoelectrodes and Chem‐FinFETs (Small 27/2024)

Small (Weinheim an der Bergstrasse, Germany), 2024-07, Vol.20 (27), p.n/a

2024

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Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β

The Journal of cell biology, 2015-05, Vol.209 (3), p.419-433

2015

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Real‐Time and High‐Resolution Monitoring of Neuronal Electrical Activity and pH Variations Based on the Co‐Integration of Nanoelectrodes and Chem‐FinFETs

Small (Weinheim an der Bergstrasse, Germany), 2024-07, Vol.20 (27), p.e2309055-n/a

2024

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Targeting APC-Host Microbe interactions 2Robust control of a neurotropic parasite through MHC I presentation by infected neurons

Molecular immunology, 2022-10, Vol.150, p.19

2022

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Hippocampal expression of a virus-derived protein impairs memory in mice

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (7), p.1611-1616

2018

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DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice

Nature communications, 2015-12, Vol.6 (1)

2015

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Tau reduction prevents A[Beta]-induced axonal transport deficits by blocking activation of GSK3[Beta]

The Journal of cell biology, 2015-05, Vol.209 (3), p.419

2015

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Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-[beta]

Nature neuroscience, 2013-05, Vol.16 (5), p.613

2013

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Physiological Brain Activity Causes DNA Double Strand Breaks in Neurons — Exacerbation by Amyloid-β

Nature neuroscience, 2013-03, Vol.16 (5), p.613-621

2013

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Tracking antigen-specific CD8 + T cells in the rat using MHC class I multimers

Journal of immunological methods, 2007-03, Vol.320 (1), p.30-39

2007

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Histone acetylation in neuronal (dys)function

Biomolecular concepts, 2016-05, Vol.7 (2), p.103

2016

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Robust Control of a Neurotropic Parasite Through MHC I Presentation by Infected Neurons

SSRN Electronic Journal

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DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice

Robust Control of a Brain-Persisting Parasite through MHC I Presentation by Infected Neurons

Histone acetylation in neuronal (dys)function

Neurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infection

Borna Disease Virus 1 Phosphoprotein Forms a Tetramer and Interacts with Host Factors Involved in DNA Double-Strand Break Repair and mRNA Processing

IMPACT OF CHRONIC TOXOPLASMA GONDII INFECTION ON THE ONSET AND PROGRESSION OF ALZHEIMER’S DISEASE

Borna disease virus docks on neuronal DNA double-strand breaks to replicate and dampens neuronal activity

Human Cytomegalovirus Infection Changes the Pattern of Surface Markers of Small Extracellular Vesicles Isolated From First Trimester Placental Long-Term Histocultures

Proteomic Analysis Reveals Selective Impediment of Neuronal Remodeling upon Borna Disease Virus Infection

High reproducibility of two-dimensional liquid chromatography using pH-driven fractionation with a pressure-resistant electrode

Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β

Zika virus in semen and spermatozoa

Real‐Time and High‐Resolution Monitoring of Neuronal Electrical Activity and pH Variations Based on the Co‐Integration of Nanoelectrodes and Chem‐FinFETs (Small 27/2024)

Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β

Real‐Time and High‐Resolution Monitoring of Neuronal Electrical Activity and pH Variations Based on the Co‐Integration of Nanoelectrodes and Chem‐FinFETs

Targeting APC-Host Microbe interactions 2Robust control of a neurotropic parasite through MHC I presentation by infected neurons

Hippocampal expression of a virus-derived protein impairs memory in mice

DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice

Tau reduction prevents A[Beta]-induced axonal transport deficits by blocking activation of GSK3[Beta]

Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-[beta]

Physiological Brain Activity Causes DNA Double Strand Breaks in Neurons — Exacerbation by Amyloid-β

Tracking antigen-specific CD8 + T cells in the rat using MHC class I multimers

Histone acetylation in neuronal (dys)function

Robust Control of a Neurotropic Parasite Through MHC I Presentation by Infected Neurons

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