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Lack of involvement of nitric oxide in NMDA-induced neuronal cell death in cortical culture

Neuroreport, 1993-11, Vol.5 (2), p.148-150

1993

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HA-966 acts at a modulatory glycine site to inhibit N-methyl-D-aspartate-evoked neurotransmitter release

European journal of pharmacology, 1989-08, Vol.166 (3), p.393-400

1989

Volltextzugriff (PDF)

The inhibition of [ 125I]ω-conotoxin GVIA binding to neuronal membranes by neomycin may be mediated by a GTP-binding protein

European journal of pharmacology, 1991-02, Vol.206 (2), p.155-158

1991

Volltextzugriff (PDF)

Stereoselective enhancement by (R)-HA-966 of the binding of [ 3H]CPP to the NMDA receptor complex

European journal of pharmacology, 1990-09, Vol.189 (2), p.237-239

1990

Volltextzugriff (PDF)

Glutamine mimics glycine to enhance [ 3H]TCP binding at the NMDA receptor complex

European journal of pharmacology, 1989-10, Vol.170 (1), p.121-122

1989

Volltextzugriff (PDF)

Lethal eosinophilic crystalline pneumonia in mice expressing a stabilized Csf2 mRNA

The FASEB journal, 2023-08, Vol.37 (8), p.e23100-n/a

2023

Volltextzugriff (PDF)

Chorioallantoic Fusion Defects and Embryonic Lethality Resulting from Disruption of Zfp36L1, a Gene Encoding a CCCH Tandem Zinc Finger Protein of the Tristetraprolin Family

Molecular and Cellular Biology, 2004-07, Vol.24 (14), p.6445-6455

2004

Volltextzugriff (PDF)

RECQL, a Member of the RecQ Family of DNA Helicases, Suppresses Chromosomal Instability

Molecular and Cellular Biology, 2007-03, Vol.27 (5), p.1784-1794

2007

Volltextzugriff (PDF)

The Drosophila Tis11 Protein and Its Effects on mRNA Expression in Flies

The Journal of biological chemistry, 2014-12, Vol.289 (51), p.35042-35060

2014

Volltextzugriff (PDF)

Tristetraprolin Prevents Gastric Metaplasia in Mice by Suppressing Pathogenic Inflammation

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.12 (5), p.1831-1845

2021

Volltextzugriff (PDF)

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice: The Role of Tumor Necrosis Factor α

The American journal of pathology, 2010-03, Vol.176 (3), p.1484-1493

2010

Volltextzugriff (PDF)

Tristetraprolin Prevents Gastric Metaplasia in Mice by Suppressing Pathogenic InflammationSummary

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.12 (5), p.1831-1845

2021

Volltextzugriff (PDF)

Myristoylated alanine rich C kinase substrate (MARCKS) heterozygous mutant mice exhibit deficits in hippocampal mossy fiber-CA3 long-term potentiation

Hippocampus, 2006, Vol.16 (5), p.495-503

2006

Volltextzugriff (PDF)

Effect of myristoylated alanine-rich C kinase substrate (MARCKS) overexpression on hippocampus-dependent learning and hippocampal synaptic plasticity in MARCKS transgenic mice

Hippocampus, 2005, Vol.15 (5), p.675-683

2005

Volltextzugriff (PDF)

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice

The American journal of pathology, 2010, Vol.176 (3), p.1484-1493

2010

Volltextzugriff (PDF)

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

American Journal of Physiology: Cell Physiology, 1999-07, Vol.277 (1), p.C163-C173

1999

Volltextzugriff (PDF)

Effect of myristoylated alanine-rich C kinase substrate (MARCKS) overexpression on hippocampus-dependent learning and hippocampal synaptic plasticity inMARCKS transgenic mice

Hippocampus, 2005, Vol.15 (5), p.675-683

2005

Volltextzugriff (PDF)

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

American Journal of Physiology: Cell Physiology, 1999-07, Vol.46 (1), p.C163-C173

1999

Volltextzugriff (PDF)

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice: The Role of Tumor Necrosis Factor {alpha}

The American journal of pathology, 2010-03, Vol.176 (3), p.1484

2010

Volltextzugriff (PDF)

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

American Journal of Physiology: Cell Physiology, 1999-07, Vol.277 (1), p.C163

1999

Volltextzugriff (PDF)

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Lack of involvement of nitric oxide in NMDA-induced neuronal cell death in cortical culture

HA-966 acts at a modulatory glycine site to inhibit N-methyl-D-aspartate-evoked neurotransmitter release

The inhibition of [ 125I]ω-conotoxin GVIA binding to neuronal membranes by neomycin may be mediated by a GTP-binding protein

Stereoselective enhancement by (R)-HA-966 of the binding of [ 3H]CPP to the NMDA receptor complex

Glutamine mimics glycine to enhance [ 3H]TCP binding at the NMDA receptor complex

Lethal eosinophilic crystalline pneumonia in mice expressing a stabilized Csf2 mRNA

Chorioallantoic Fusion Defects and Embryonic Lethality Resulting from Disruption of Zfp36L1, a Gene Encoding a CCCH Tandem Zinc Finger Protein of the Tristetraprolin Family

RECQL, a Member of the RecQ Family of DNA Helicases, Suppresses Chromosomal Instability

The Drosophila Tis11 Protein and Its Effects on mRNA Expression in Flies

Tristetraprolin Prevents Gastric Metaplasia in Mice by Suppressing Pathogenic Inflammation

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice: The Role of Tumor Necrosis Factor α

Tristetraprolin Prevents Gastric Metaplasia in Mice by Suppressing Pathogenic InflammationSummary

Myristoylated alanine rich C kinase substrate (MARCKS) heterozygous mutant mice exhibit deficits in hippocampal mossy fiber-CA3 long-term potentiation

Effect of myristoylated alanine-rich C kinase substrate (MARCKS) overexpression on hippocampus-dependent learning and hippocampal synaptic plasticity in MARCKS transgenic mice

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

Effect of myristoylated alanine-rich C kinase substrate (MARCKS) overexpression on hippocampus-dependent learning and hippocampal synaptic plasticity inMARCKS transgenic mice

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

Left-Sided Cardiac Valvulitis in Tristetraprolin-Deficient Mice: The Role of Tumor Necrosis Factor {alpha}

Phagocytic and macropinocytic activity in MARCKS-deficient macrophages and fibroblasts

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