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A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore

The Journal of general physiology, 2007-07, Vol.130 (1), p.11-20

2007

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A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis

The Journal of clinical investigation, 2011-10, Vol.121 (10), p.4082-4094

2011

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A calcium channel mutant mouse model of hypokalemic periodic paralysis

The Journal of clinical investigation, 2012-12, Vol.122 (12), p.4580-4591

2012

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Gating pore currents in DIIS4 mutations of NaV1.4 associated with periodic paralysis: saturation of ion flux and implications for disease pathogenesis

The Journal of general physiology, 2008-10, Vol.132 (4), p.447-464

2008

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Neurotrimin Mediates Bifunctional Effects on Neurite Outgrowth via Homophilic and Heterophilic Interactions

The Journal of neuroscience, 1998-11, Vol.18 (22), p.9312-9325

1998

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An Exploratory Study Using Electronic Medical Records to Assess the Feasibility of Establishing Cohorts of Patients with Genetic Causes of Parkinson's Disease

Journal of Parkinson's disease, 2022-01, Vol.12 (5), p.1645-1653

2022

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The Human Skeletal Muscle Na Channel Mutation R669H Associated with Hypokalemic Periodic Paralysis Enhances Slow Inactivation

The Journal of neuroscience, 2000-12, Vol.20 (23), p.8610-8617

2000

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Paradoxical depolarization of BA2+- treated muscle exposed to low extracellular K+: Insights into resting potential abnormalities in hypokalemic paralysis

Muscle & nerve, 2008-03, Vol.37 (3), p.326-337

2008

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Slow inactivation does not block the aqueous accessibility to the outer pore of voltage-gated Na channels

The Journal of general physiology, 2002-10, Vol.120 (4), p.509-516

2002

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Paradoxical depolarization of BA 2+ ‐ treated muscle exposed to low extracellular K + : Insights into resting potential abnormalities in hypokalemic paralysis

Muscle & nerve, 2008-03, Vol.37 (3), p.326-337

2008

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A Na super(+) Channel Mutation Linked to Hypokalemic Periodic Paralysis Exposes a Proton-selective Gating Pore

The Journal of general physiology, 2007-07, Vol.130 (1), p.11-20

2007

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Cloning of neurotrimin defines a new subfamily of differentially expressed neural cell adhesion molecules

The Journal of neuroscience, 1995-03, Vol.15 (3), p.2141-2156

1995

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Quantification of tau in cerebrospinal fluid by immunoaffinity enrichment and tandem mass spectrometry

Clinical chemistry (Baltimore, Md.), 2014-04, Vol.60 (4), p.683-689

2014

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Longitudinal investigation for enhancing Down Syndrome Research (LIFE‐DSR) Study: Tau PET and CSF sub‐studies

Alzheimer's & dementia, 2021-12, Vol.17 (S4), p.n/a

2021

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A multinational study distinguishing Alzheimer's and healthy patients using cerebrospinal fluid tau/Aβ42 cutoff with concordance to amyloid positron emission tomography imaging

Alzheimer's & dementia : diagnosis, assessment & disease monitoring, 2017, Vol.6 (1), p.201-209

2017

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Localization of Human Papillomavirus Type 16 DNA Using the Polymerase Chain Reaction in the Cervix Uteri of Women with Cervical Intraepithelial Neoplasia

Journal of general virology, 1989-10, Vol.70 (10), p.2555-2562

1989

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Human papillomavirus type 16 transcripts expressed from viral-cellular junctions and full-length viral copies in CaSki cells and in a cervical carcinoma

Virology (New York, N.Y.), 1991-06, Vol.182 (2), p.870-873

1991

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Detection of human papillomavirus types by the polymerase chain reaction and the differentiation between high-risk and low-risk cervical lesions

Virchows Archiv. Abteilung B. Zellpathologie, 1992-12, Vol.62 (3), p.167-171

1992

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A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore

A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis

A calcium channel mutant mouse model of hypokalemic periodic paralysis

Gating pore currents in DIIS4 mutations of NaV1.4 associated with periodic paralysis: saturation of ion flux and implications for disease pathogenesis

Neurotrimin Mediates Bifunctional Effects on Neurite Outgrowth via Homophilic and Heterophilic Interactions

An Exploratory Study Using Electronic Medical Records to Assess the Feasibility of Establishing Cohorts of Patients with Genetic Causes of Parkinson's Disease

The Human Skeletal Muscle Na Channel Mutation R669H Associated with Hypokalemic Periodic Paralysis Enhances Slow Inactivation

Paradoxical depolarization of BA2+- treated muscle exposed to low extracellular K+: Insights into resting potential abnormalities in hypokalemic paralysis

Slow inactivation does not block the aqueous accessibility to the outer pore of voltage-gated Na channels

Paradoxical depolarization of BA 2+ ‐ treated muscle exposed to low extracellular K + : Insights into resting potential abnormalities in hypokalemic paralysis

A Na super(+) Channel Mutation Linked to Hypokalemic Periodic Paralysis Exposes a Proton-selective Gating Pore

Cloning of neurotrimin defines a new subfamily of differentially expressed neural cell adhesion molecules

Quantification of tau in cerebrospinal fluid by immunoaffinity enrichment and tandem mass spectrometry

Longitudinal investigation for enhancing Down Syndrome Research (LIFE‐DSR) Study: Tau PET and CSF sub‐studies

A multinational study distinguishing Alzheimer's and healthy patients using cerebrospinal fluid tau/Aβ42 cutoff with concordance to amyloid positron emission tomography imaging

Localization of Human Papillomavirus Type 16 DNA Using the Polymerase Chain Reaction in the Cervix Uteri of Women with Cervical Intraepithelial Neoplasia

Human papillomavirus type 16 transcripts expressed from viral-cellular junctions and full-length viral copies in CaSki cells and in a cervical carcinoma

Detection of human papillomavirus types by the polymerase chain reaction and the differentiation between high-risk and low-risk cervical lesions

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