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Nitroprusside induces melanoma ferroptosis with serum supplementation and prolongs survival under serum depletion or hypoxia

Biochemical and biophysical research communications, 2020-05, Vol.525 (3), p.626-632

2020

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Hypoxic resistance of KRAS mutant tumor cells to 3-Bromopyruvate is counteracted by Prima-1 and reversed by N-acetylcysteine

BMC cancer, 2016-11, Vol.16 (1), p.902, Article 902

2016

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Disulfiram anti-cancer efficacy without copper overload is enhanced by extracellular H2O2 generation: antagonism by tetrathiomolybdate

Oncotarget, 2015-10, Vol.6 (30), p.29771-29781

2015

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Abstract 3049: BRAF status and low glutamine/glucose influence glutamine addiction modulated by oxidative stress in human melanoma cells

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.3049-3049

2015

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Abstract 2907: AMPK induction, lysosomal acidification and melanoma survival increased by p53 dysfunction are counteracted by inhibiting autophagy

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.2907-2907

2015

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Abstract 4331: Lowering glucose increases nutlin-3 toxicity against melanomas irrespective of BRAF,NRAS or p53 mutations

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.4331-4331

2014

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p53 inactivation decreases dependence on estrogen/ERK signalling for proliferation but promotes EMT and susceptility to 3-bromopyruvate in ERα+ breast cancer MCF-7 cells

Biochemical pharmacology, 2014-03, Vol.88 (2), p.169-177

2014

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Hypoxia and hypoxia mimetic cooperate to counteract tumor cell resistance to glucose starvation preferentially in tumor cells with mutant p53

Biochemical and biophysical research communications, 2014-01, Vol.443 (1), p.120-125

2014

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Abstract 5435: Increased tumor susceptibility to low glucose under hypoxia is increased by BRAF(V600E) mutation: correlation with persistent phosphoP90RSK activation

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.5435-5435

2013

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Abstract 1890: Superoxide modulates tumor cell survival in response to hypoxia and metabolic stress: role of p53 status

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.1890-1890

2013

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Hypoxia, Mn-SOD and H(2)O(2) regulate p53 reactivation and PRIMA-1 toxicity irrespective of p53 status in human breast cancer cells

Biochemical pharmacology, 2012-12, Vol.84 (12), p.1563

2012

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Hypoxia, Mn-SOD and H2O2 regulate p53 reactivation and PRIMA-1 toxicity irrespective of p53 status in human breast cancer cells

Biochemical pharmacology, 2012-12, Vol.84 (12), p.1563-1570

2012

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Abstract 3212: N-acetylcysteine protects melanoma cells from glucose depletion through unequal modulation of SOD1 and Copper chaperone for SOD1 depending on p53 mutational status

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.3212-3212

2012

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Abstract C36: Hypoxia or glutathione depletion increase apoptotic response to PRIMA-1 in p53-mutated and nonmutated human breast cancer cells

Molecular cancer therapeutics, 2011-11, Vol.10 (11_Supplement), p.C36-C36

2011

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Metabolic utilization of exogenous pyruvate by mutant p53 (R175H) human melanoma cells promotes survival under glucose depletion

Cancer biology & therapy, 2011-10, Vol.12 (7), p.647-656

2011

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Abstract 1265: Survival under glucose and serum deprivation is increased by DN-mutant p53 R175H in human melanoma: A possible gain of oncogenic function

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.1265-1265

2011

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H sub(2)O sub(2) preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

Chemico-biological interactions, 2010-10, Vol.188 (1), p.134-143

2010

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H(2)O(2) preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

Chemico-biological interactions, 2010-10, Vol.188 (1), p.134

2010

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H2O2 preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

Chemico-biological interactions, 2010-10, Vol.188 (1), p.134-143

2010

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Abstract 576: Hydrogen peroxide and iron released from nitroprusside cooperate to induce melanoma cell death: role of the p53 genotype

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.576-576

2010

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Abstract 1107: Mutant p53 increases epithelial mesenchymal transition, estrogen receptor alpha and erbB2/Her2/neu

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.1107-1107

2010

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Nitric Oxide Donors or Nitrite Counteract Copper-[dithiocarbamate]2-Mediated Tumor Cell Death and Inducible Nitric Oxide Synthase Down-Regulation: Possible Role of a Nitrosyl-Copper [Dithiocarbamate]2 Complex

Journal of medicinal chemistry, 2010-02, Vol.53 (4), p.1627-1635

2010

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Nitric oxide donors or nitrite counteract copper-[dithiocarbamate](2)-mediated tumor cell death and inducible nitric oxide synthase down-regulation: possible role of a nitrosyl-copper [dithiocarbamate](2) complex

Journal of medicinal chemistry, 2010-02, Vol.53 (4), p.1627-1635

2010

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H 2O 2 preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

Chemico-biological interactions, 2010, Vol.188 (1), p.134-143

2010

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DN‐R175H p53 mutation is more effective than p53 interference in inducing epithelial disorganization and activation of proliferation signals in human carcinoma cells: Role of E‐cadherin

International journal of cancer, 2009-10, Vol.125 (7), p.1604-1612

2009

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Nitroprusside induces melanoma ferroptosis with serum supplementation and prolongs survival under serum depletion or hypoxia

Hypoxic resistance of KRAS mutant tumor cells to 3-Bromopyruvate is counteracted by Prima-1 and reversed by N-acetylcysteine

Disulfiram anti-cancer efficacy without copper overload is enhanced by extracellular H2O2 generation: antagonism by tetrathiomolybdate

Abstract 3049: BRAF status and low glutamine/glucose influence glutamine addiction modulated by oxidative stress in human melanoma cells

Abstract 2907: AMPK induction, lysosomal acidification and melanoma survival increased by p53 dysfunction are counteracted by inhibiting autophagy

Abstract 4331: Lowering glucose increases nutlin-3 toxicity against melanomas irrespective of BRAF,NRAS or p53 mutations

p53 inactivation decreases dependence on estrogen/ERK signalling for proliferation but promotes EMT and susceptility to 3-bromopyruvate in ERα+ breast cancer MCF-7 cells

Hypoxia and hypoxia mimetic cooperate to counteract tumor cell resistance to glucose starvation preferentially in tumor cells with mutant p53

Abstract 5435: Increased tumor susceptibility to low glucose under hypoxia is increased by BRAF(V600E) mutation: correlation with persistent phosphoP90RSK activation

Abstract 1890: Superoxide modulates tumor cell survival in response to hypoxia and metabolic stress: role of p53 status

Hypoxia, Mn-SOD and H(2)O(2) regulate p53 reactivation and PRIMA-1 toxicity irrespective of p53 status in human breast cancer cells

Hypoxia, Mn-SOD and H2O2 regulate p53 reactivation and PRIMA-1 toxicity irrespective of p53 status in human breast cancer cells

Abstract 3212: N-acetylcysteine protects melanoma cells from glucose depletion through unequal modulation of SOD1 and Copper chaperone for SOD1 depending on p53 mutational status

Abstract C36: Hypoxia or glutathione depletion increase apoptotic response to PRIMA-1 in p53-mutated and nonmutated human breast cancer cells

Metabolic utilization of exogenous pyruvate by mutant p53 (R175H) human melanoma cells promotes survival under glucose depletion

Abstract 1265: Survival under glucose and serum deprivation is increased by DN-mutant p53 R175H in human melanoma: A possible gain of oncogenic function

H sub(2)O sub(2) preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

H(2)O(2) preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

H2O2 preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

Abstract 576: Hydrogen peroxide and iron released from nitroprusside cooperate to induce melanoma cell death: role of the p53 genotype

Abstract 1107: Mutant p53 increases epithelial mesenchymal transition, estrogen receptor alpha and erbB2/Her2/neu

Nitric Oxide Donors or Nitrite Counteract Copper-[dithiocarbamate]2-Mediated Tumor Cell Death and Inducible Nitric Oxide Synthase Down-Regulation: Possible Role of a Nitrosyl-Copper [Dithiocarbamate]2 Complex

Nitric oxide donors or nitrite counteract copper-[dithiocarbamate](2)-mediated tumor cell death and inducible nitric oxide synthase down-regulation: possible role of a nitrosyl-copper [dithiocarbamate](2) complex

H 2O 2 preferentially synergizes with nitroprusside to induce apoptosis associated with superoxide dismutase dysregulation in human melanoma irrespective of p53 status: Antagonism by o-phenanthroline

DN‐R175H p53 mutation is more effective than p53 interference in inducing epithelial disorganization and activation of proliferation signals in human carcinoma cells: Role of E‐cadherin

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