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VEGF-C sustains VEGFR2 activation under bevacizumab therapy and promotes glioblastoma maintenance

Neuro-oncology (Charlottesville, Va.), 2018-10, Vol.20 (11), p.1462-1474

2018

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Mechanisms for oncogenic activation of the epidermal growth factor receptor

Cellular signalling, 2007-10, Vol.19 (10), p.2013-2023

2007

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Clinical variables serve as prognostic factors in a model for survival from glioblastoma multiforme: an observational study of a cohort of consecutive non-selected patients from a single institution

BMC cancer, 2013-09, Vol.13 (1), p.402-402, Article 402

2013

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Cetuximab, bevacizumab, and irinotecan for patients with primary glioblastoma and progression after radiation therapy and temozolomide: a phase II trial

Neuro-oncology (Charlottesville, Va.), 2010-05, Vol.12 (5), p.508-516

2010

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Activation of the EGFR Gene Target EphA2 Inhibits Epidermal Growth Factor–Induced Cancer Cell Motility

Molecular cancer research, 2007-03, Vol.5 (3), p.283-293

2007

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Differentiation of glioblastoma multiforme stem-like cells leads to downregulation of EGFR and EGFRvIII and decreased tumorigenic and stem-like cell potential

Cancer biology & therapy, 2014-02, Vol.15 (2), p.216-224

2014

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Level of Notch activation determines the effect on growth and stem cell-like features in glioblastoma multiforme neurosphere cultures

Cancer biology & therapy, 2013-07, Vol.14 (7), p.625-637

2013

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Cell adhesion and EGFR activation regulate EphA2 expression in cancer

Cellular signalling, 2010-04, Vol.22 (4), p.636-644

2010

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The Notch pathway in cancer: Differentiation gone awry

European journal of cancer (1990), 2005-11, Vol.41 (17), p.2620-2629

2005

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Maintenance of EGFR and EGFRvIII expressions in an in vivo and in vitro model of human glioblastoma multiforme

Experimental cell research, 2011-07, Vol.317 (11), p.1513-1526

2011

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EGFR induces expression of IRF‐1 via STAT1 and STAT3 activation leading to growth arrest of human cancer cells

International journal of cancer, 2008-01, Vol.122 (2), p.342-349

2008

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Prospective evaluation of angiogenic, hypoxic and EGFR-related biomarkers in recurrent glioblastoma multiforme treated with cetuximab, bevacizumab and irinotecan

APMIS : acta pathologica, microbiologica et immunologica Scandinavica, 2010-08, Vol.118 (8), p.585-594

2010

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Irinotecan and bevacizumab in recurrent glioblastoma multiforme

Expert opinion on pharmacotherapy, 2011-04, Vol.12 (5), p.825-833

2011

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Inhibition of Notch signaling alters the phenotype of orthotopic tumors formed from glioblastoma multiforme neurosphere cells but does not hamper intracranial tumor growth regardless of endogene Notch pathway signature

Cancer biology & therapy, 2014-07, Vol.15 (7), p.862-877

2014

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Combined EGFR- and notch inhibition display additive inhibitory effect on glioblastoma cell viability and glioblastoma-induced endothelial cell sprouting in vitro

Cancer cell international, 2016-04, Vol.16 (1), p.34-34, Article 34

2016

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Cetuximab Insufficiently Inhibits Glioma Cell Growth Due to Persistent EGFR Downstream Signaling

Cancer investigation, 2010-09, Vol.28 (8), p.775-787

2010

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Notch signaling and brain tumors

Advances in experimental medicine and biology, 2012, Vol.727, p.289

2012

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Regulation of the Notch target gene Hes-1 by TGFα induced Ras/MAPK signaling in human neuroblastoma cells

Experimental cell research, 2005-10, Vol.310 (1), p.218-228

2005

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Comparison of (18)F-FET and (18)F-FLT small animal PET for the assessment of anti-VEGF treatment response in an orthotopic model of glioblastoma

Nuclear medicine and biology, 2016-03, Vol.43 (3), p.198-205

2016

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sup.18F-FET MicroPET and MicroMRI for Anti-VEGF and Anti-PlGF Response Assessment in an Orthotopic Murine Model of Human Glioblastoma

PloS one, 2015-02, Vol.10 (2)

2015

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The Use of Longitudinal .sup.18F-FET MicroPET Imaging to Evaluate Response to Irinotecan in Orthotopic Human Glioblastoma Multiforme Xenografts

PloS one, 2014-06, Vol.9 (6)

2014

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18F-FET microPET and microMRI for anti-VEGF and anti-PlGF response assessment in an orthotopic murine model of human glioblastoma

PloS one, 2015, Vol.10 (2), p.e0115315-e0115315

2015

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Notch signaling in neuroblastoma

Seminars in cancer biology, 2004-10, Vol.14 (5), p.365-373

2004

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The use of longitudinal 18F-FET MicroPET imaging to evaluate response to irinotecan in orthotopic human glioblastoma multiforme xenografts

PloS one, 2014, Vol.9 (2), p.e100009-e100009

2014

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Improved Response by Co-targeting EGFR/EGFRvIII and Src Family Kinases in Human Cancer Cells

Cancer investigation, 2009-01, Vol.27 (2), p.178-183

2009

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VEGF-C sustains VEGFR2 activation under bevacizumab therapy and promotes glioblastoma maintenance

Mechanisms for oncogenic activation of the epidermal growth factor receptor

Clinical variables serve as prognostic factors in a model for survival from glioblastoma multiforme: an observational study of a cohort of consecutive non-selected patients from a single institution

Cetuximab, bevacizumab, and irinotecan for patients with primary glioblastoma and progression after radiation therapy and temozolomide: a phase II trial

Activation of the EGFR Gene Target EphA2 Inhibits Epidermal Growth Factor–Induced Cancer Cell Motility

Differentiation of glioblastoma multiforme stem-like cells leads to downregulation of EGFR and EGFRvIII and decreased tumorigenic and stem-like cell potential

Level of Notch activation determines the effect on growth and stem cell-like features in glioblastoma multiforme neurosphere cultures

Cell adhesion and EGFR activation regulate EphA2 expression in cancer

The Notch pathway in cancer: Differentiation gone awry

Maintenance of EGFR and EGFRvIII expressions in an in vivo and in vitro model of human glioblastoma multiforme

EGFR induces expression of IRF‐1 via STAT1 and STAT3 activation leading to growth arrest of human cancer cells

Prospective evaluation of angiogenic, hypoxic and EGFR-related biomarkers in recurrent glioblastoma multiforme treated with cetuximab, bevacizumab and irinotecan

Irinotecan and bevacizumab in recurrent glioblastoma multiforme

Inhibition of Notch signaling alters the phenotype of orthotopic tumors formed from glioblastoma multiforme neurosphere cells but does not hamper intracranial tumor growth regardless of endogene Notch pathway signature

Combined EGFR- and notch inhibition display additive inhibitory effect on glioblastoma cell viability and glioblastoma-induced endothelial cell sprouting in vitro

Cetuximab Insufficiently Inhibits Glioma Cell Growth Due to Persistent EGFR Downstream Signaling

Notch signaling and brain tumors

Regulation of the Notch target gene Hes-1 by TGFα induced Ras/MAPK signaling in human neuroblastoma cells

Comparison of (18)F-FET and (18)F-FLT small animal PET for the assessment of anti-VEGF treatment response in an orthotopic model of glioblastoma

sup.18F-FET MicroPET and MicroMRI for Anti-VEGF and Anti-PlGF Response Assessment in an Orthotopic Murine Model of Human Glioblastoma

The Use of Longitudinal .sup.18F-FET MicroPET Imaging to Evaluate Response to Irinotecan in Orthotopic Human Glioblastoma Multiforme Xenografts

18F-FET microPET and microMRI for anti-VEGF and anti-PlGF response assessment in an orthotopic murine model of human glioblastoma

Notch signaling in neuroblastoma

The use of longitudinal 18F-FET MicroPET imaging to evaluate response to irinotecan in orthotopic human glioblastoma multiforme xenografts

Improved Response by Co-targeting EGFR/EGFRvIII and Src Family Kinases in Human Cancer Cells

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