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Computational and statistical approaches to analyzing variants identified by exome sequencing

Genome biology, 2011-01, Vol.12 (9), p.227-227

2011

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Identification of Medically Actionable Secondary Findings in the 1000 Genomes

PloS one, 2015-09, Vol.10 (9), p.e0135193-e0135193

2015

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Polygenic Risk Score Identifies Subgroup With Higher Burden of Atherosclerosis and Greater Relative Benefit From Statin Therapy in the Primary Prevention Setting

Circulation (New York, N.Y.), 2017-05, Vol.135 (22), p.2091-2101

2017

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Human genetic insights into lipoproteins and risk of cardiometabolic disease

Current opinion in lipidology, 2017-04, Vol.28 (2), p.113-119

Stitziel, Nathan O

2017

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Clinical Genetic Testing for Familial Hypercholesterolemia: JACC Scientific Expert Panel

Journal of the American College of Cardiology, 2018-08, Vol.72 (6), p.662-680

2018

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Angiopoietin-like 3: An important protein in regulating lipoprotein levels

Baillière's best practice & research. Clinical endocrinology & metabolism, 2023-05, Vol.37 (3), p.101688-101688, Article 101688

2023

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Exome sequencing and the genetic basis of complex traits

Nature genetics, 2012-06, Vol.44 (6), p.623-630

2012

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Non-parametric Polygenic Risk Prediction via Partitioned GWAS Summary Statistics

American journal of human genetics, 2020-07, Vol.107 (1), p.46-59

2020

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Leveraging Human Genetics to Guide Drug Target Discovery

Trends in cardiovascular medicine, 2017-07, Vol.27 (5), p.352-359

2017

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Low LDL Cholesterol Is Not an Independent Risk Factor for Hepatic Steatosis

Digestive diseases and sciences, 2023-08, Vol.68 (8), p.3451-3457

2023

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Genetics of the extracellular matrix in aortic aneurysmal diseases

Matrix biology, 2018-10, Vol.71-72, p.128-143

2018

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Emerging Targets for Cardiovascular Disease Prevention in Diabetes

Trends in molecular medicine, 2020-08, Vol.26 (8), p.744-757

2020

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SVEP1 is an endogenous ligand for the orphan receptor PEAR1

Nature communications, 2023-02, Vol.14 (1), p.850-18, Article 850

2023

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ANGPTL3 deficiency impairs lipoprotein production and produces adaptive changes in hepatic lipid metabolism

Journal of lipid research, 2024-02, Vol.65 (2), p.100500-100500, Article 100500

2024

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Integrin α9β1 deficiency does not impact the development of atherosclerosis in mice

Heliyon, 2024-02, Vol.10 (4), p.e25760-e25760, Article e25760

2024

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Phenotypic Consequences of a Genetic Predisposition to Enhanced Nitric Oxide Signaling

Circulation (New York, N.Y.), 2018-01, Vol.137 (3), p.222-232

2018

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Loss of function mutation in LOX causes thoracic aortic aneurysm and dissection in humans

Proceedings of the National Academy of Sciences - PNAS, 2016-08, Vol.113 (31), p.8759-8764

2016

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The emerging Janus face of SVEP1 in development and disease

Trends in molecular medicine, 2023-11, Vol.29 (11), p.939-950

2023

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Capitalizing on Insights from Human Genetics to Identify Novel Therapeutic Targets for Coronary Artery Disease

Annual review of medicine, 2019-01, Vol.70 (1), p.19-32

2019

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Vascular smooth muscle- and myeloid cell-derived integrin α9β1 does not directly mediate the development of atherosclerosis in mice

Atherosclerosis, 2022-11, Vol.360, p.15-20

2022

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Gain-of-function mutations in the mechanically activated ion channel PIEZO2 cause a subtype of Distal Arthrogryposis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (12), p.4667-4672

2013

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SVEP1 is a human coronary artery disease locus that promotes atherosclerosis

Science translational medicine, 2021-03, Vol.13 (586)

2021

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Author Correction: SVEP1 is an endogenous ligand for the orphan receptor PEAR1

Nature communications, 2023-03, Vol.14 (1), p.1511-1511, Article 1511

2023

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Semi-supervised machine learning method for predicting homogeneous ancestry groups to assess Hardy-Weinberg equilibrium in diverse whole-genome sequencing studies

American journal of human genetics, 2024-10, Vol.111 (10), p.2129-2138

2024

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Genetic association studies in cardiovascular diseases: Do we have enough power?

Trends in cardiovascular medicine, 2017-08, Vol.27 (6), p.397-404

2017

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Computational and statistical approaches to analyzing variants identified by exome sequencing

Identification of Medically Actionable Secondary Findings in the 1000 Genomes

Polygenic Risk Score Identifies Subgroup With Higher Burden of Atherosclerosis and Greater Relative Benefit From Statin Therapy in the Primary Prevention Setting

Human genetic insights into lipoproteins and risk of cardiometabolic disease

Clinical Genetic Testing for Familial Hypercholesterolemia: JACC Scientific Expert Panel

Angiopoietin-like 3: An important protein in regulating lipoprotein levels

Exome sequencing and the genetic basis of complex traits

Non-parametric Polygenic Risk Prediction via Partitioned GWAS Summary Statistics

Leveraging Human Genetics to Guide Drug Target Discovery

Low LDL Cholesterol Is Not an Independent Risk Factor for Hepatic Steatosis

Genetics of the extracellular matrix in aortic aneurysmal diseases

Emerging Targets for Cardiovascular Disease Prevention in Diabetes

SVEP1 is an endogenous ligand for the orphan receptor PEAR1

ANGPTL3 deficiency impairs lipoprotein production and produces adaptive changes in hepatic lipid metabolism

Integrin α9β1 deficiency does not impact the development of atherosclerosis in mice

Phenotypic Consequences of a Genetic Predisposition to Enhanced Nitric Oxide Signaling

Loss of function mutation in LOX causes thoracic aortic aneurysm and dissection in humans

The emerging Janus face of SVEP1 in development and disease

Capitalizing on Insights from Human Genetics to Identify Novel Therapeutic Targets for Coronary Artery Disease

Vascular smooth muscle- and myeloid cell-derived integrin α9β1 does not directly mediate the development of atherosclerosis in mice

Gain-of-function mutations in the mechanically activated ion channel PIEZO2 cause a subtype of Distal Arthrogryposis

SVEP1 is a human coronary artery disease locus that promotes atherosclerosis

Author Correction: SVEP1 is an endogenous ligand for the orphan receptor PEAR1

Semi-supervised machine learning method for predicting homogeneous ancestry groups to assess Hardy-Weinberg equilibrium in diverse whole-genome sequencing studies

Genetic association studies in cardiovascular diseases: Do we have enough power?

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