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Intracellular Ca2+ release contributes to automaticity in cat atrial pacemaker cells

The Journal of physiology, 2000-04, Vol.524 (2), p.415-422

2000

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Local calcium gradients during excitation–contraction coupling and alternans in atrial myocytes

The Journal of physiology, 2003-01, Vol.546 (1), p.19-31

2003

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Functional coupling between glycolysis and excitation—contraction coupling underlies alternans in cat heart cells

The Journal of physiology, 2000-05, Vol.524 (3), p.795-806

2000

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Effects of mitochondrial uncoupling on Ca(2+) signaling during excitation-contraction coupling in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2013-04, Vol.304 (7), p.H983-H993

2013

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Acute exposure to thyroid hormone increases Na+ current and intracellular Ca2+ in cat atrial myocytes

The Journal of physiology, 2003-01, Vol.546 (2), p.491-499

2003

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Mitochondrial calcium uptake stimulates nitric oxide production in mitochondria of bovine vascular endothelial cells

American Journal of Physiology: Cell Physiology, 2004-02, Vol.286 (2), p.C406-C415

2004

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Reactive oxygen species contribute to the development of arrhythmogenic Ca2+ waves during β‐adrenergic receptor stimulation in rabbit cardiomyocytes

The Journal of physiology, 2012-07, Vol.590 (14), p.3291-3304

2012

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Nitric oxide signaling mediates stimulation of L-type Ca2+ current elicited by withdrawal of acetylcholine in cat atrial myocytes

The Journal of general physiology, 1998-01, Vol.111 (1), p.113-125

1998

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Activation and propagation of Ca(2+) release during excitation-contraction coupling in atrial myocytes

Biophysical journal, 2001-11, Vol.81 (5), p.2590-2605

2001

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Activation and Propagation of Ca2+ Release during Excitation-Contraction Coupling in Atrial Myocytes

Biophysical journal, 2001-11, Vol.81 (5), p.2590-2605

2001

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Signaling Mechanisms That Mediate Nitric Oxide Production Induced by Acetylcholine Exposure and Withdrawal in Cat Atrial Myocytes

Circulation research, 2003-12, Vol.93 (12), p.1233-1240

2003

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Laminin binding to β1‐integrins selectively alters β1‐ and β2‐adrenoceptor signalling in cat atrial myocytes

The Journal of physiology, 2000-08, Vol.527 (1), p.3-9

2000

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Premature beats elicit a phase reversal of mechanoelectrical alternans in cat ventricular myocytes. A possible mechanism for reentrant arrhythmias

Circulation (New York, N.Y.), 1995-01, Vol.91 (1), p.201-214

1995

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Effects of mitochondrial uncoupling on Ca2+ signaling during excitation-contraction coupling in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2013-04, Vol.304 (7), p.H983-H993

2013

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Reactive oxygen species contribute to the development of arrhythmogenic Ca 2+ waves during β‐adrenergic receptor stimulation in rabbit cardiomyocytes

The Journal of physiology, 2012-07, Vol.590 (14), p.3291-3304

2012

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Reactive oxygen species contribute to the development of arrhythmogenic Ca2+ waves during [beta]-adrenergic receptor stimulation in rabbit cardiomyocytes

The Journal of physiology, 2012-07, Vol.590 (14), p.3291

2012

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Diastolic SR Ca efflux in atrial pacemaker cells and Ca-overloaded myocytes

American journal of physiology. Heart and circulatory physiology, 1997-08, Vol.273 (2), p.H886-H892

1997

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Nitric oxide signalling by selective β2‐adrenoceptor stimulation prevents ach‐induced inhibition of β2‐stimulated Ca2+ current in cat atrial myocytes

The Journal of physiology, 2002-08, Vol.542 (3), p.711-723

2002

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Inhibition of Camp-Dependent PKA Activates β2-Adrenergic Receptor Stimulation of Cytosolic Phospholipase A2 via Raf-1/Mek/Erk and Ip3-Dependent Ca2+ Signaling in Atrial Myocytes

Biophysical journal, 2014-01, Vol.106 (2), p.305a-305a

2014

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Effects of mitochondrial uncoupling on Ca^sup 2+^ signaling during excitation-contraction coupling in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2013-04, Vol.304 (7), p.H983

2013

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Effects of mitochondrial uncoupling on Ca 2+ signaling during excitation-contraction coupling in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2013-04, Vol.304 (7), p.H983-H993

2013

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Laminin acts via β1 integrin signalling to alter cholinergic regulation of L‐type Ca2+ current in cat atrial myocytes

The Journal of physiology, 2000-07, Vol.526 (1), p.57-68

2000

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A Cellular Mechanism Contributing to Postvagal Tachycardia Studied in Isolated Pacemaker Cells From Cat Right Atrium

Circulation research, 1996-07, Vol.79 (1), p.109-114

1996

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β-Adrenergic stimulation induces acetylcholine to activate ATP-sensitive K+ current in cat atrial myocytes

Circulation research, 1995-09, Vol.77 (3), p.565-574

1995

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β-Adrenergic Receptor Stimulation of ROS Production Generates Spontaneous Ca2+ Waves in Rabbit Ventricular Myocytes

Biophysical journal, 2011-02, Vol.100 (3), p.559-560a

2011

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Intracellular Ca2+ release contributes to automaticity in cat atrial pacemaker cells

Local calcium gradients during excitation–contraction coupling and alternans in atrial myocytes

Functional coupling between glycolysis and excitation—contraction coupling underlies alternans in cat heart cells

Effects of mitochondrial uncoupling on Ca(2+) signaling during excitation-contraction coupling in atrial myocytes

Acute exposure to thyroid hormone increases Na+ current and intracellular Ca2+ in cat atrial myocytes

Mitochondrial calcium uptake stimulates nitric oxide production in mitochondria of bovine vascular endothelial cells

Reactive oxygen species contribute to the development of arrhythmogenic Ca2+ waves during β‐adrenergic receptor stimulation in rabbit cardiomyocytes

Nitric oxide signaling mediates stimulation of L-type Ca2+ current elicited by withdrawal of acetylcholine in cat atrial myocytes

Activation and propagation of Ca(2+) release during excitation-contraction coupling in atrial myocytes

Activation and Propagation of Ca2+ Release during Excitation-Contraction Coupling in Atrial Myocytes

Signaling Mechanisms That Mediate Nitric Oxide Production Induced by Acetylcholine Exposure and Withdrawal in Cat Atrial Myocytes

Laminin binding to β1‐integrins selectively alters β1‐ and β2‐adrenoceptor signalling in cat atrial myocytes

Premature beats elicit a phase reversal of mechanoelectrical alternans in cat ventricular myocytes. A possible mechanism for reentrant arrhythmias

Effects of mitochondrial uncoupling on Ca2+ signaling during excitation-contraction coupling in atrial myocytes

Reactive oxygen species contribute to the development of arrhythmogenic Ca 2+ waves during β‐adrenergic receptor stimulation in rabbit cardiomyocytes

Reactive oxygen species contribute to the development of arrhythmogenic Ca2+ waves during [beta]-adrenergic receptor stimulation in rabbit cardiomyocytes

Diastolic SR Ca efflux in atrial pacemaker cells and Ca-overloaded myocytes

Nitric oxide signalling by selective β2‐adrenoceptor stimulation prevents ach‐induced inhibition of β2‐stimulated Ca2+ current in cat atrial myocytes

Inhibition of Camp-Dependent PKA Activates β2-Adrenergic Receptor Stimulation of Cytosolic Phospholipase A2 via Raf-1/Mek/Erk and Ip3-Dependent Ca2+ Signaling in Atrial Myocytes

Effects of mitochondrial uncoupling on Ca^sup 2+^ signaling during excitation-contraction coupling in atrial myocytes

Effects of mitochondrial uncoupling on Ca 2+ signaling during excitation-contraction coupling in atrial myocytes

Laminin acts via β1 integrin signalling to alter cholinergic regulation of L‐type Ca2+ current in cat atrial myocytes

A Cellular Mechanism Contributing to Postvagal Tachycardia Studied in Isolated Pacemaker Cells From Cat Right Atrium

β-Adrenergic stimulation induces acetylcholine to activate ATP-sensitive K+ current in cat atrial myocytes

β-Adrenergic Receptor Stimulation of ROS Production Generates Spontaneous Ca2+ Waves in Rabbit Ventricular Myocytes

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