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Lung vascular development: implications for the pathogenesis of bronchopulmonary dysplasia

Annual review of physiology, 2005-01, Vol.67 (1), p.623-661

2005

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Cellular and molecular pathobiology of pulmonary arterial hypertension

Journal of the American College of Cardiology, 2004-06, Vol.43 (12), p.S13-S24

2004

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Role of the Adventitia in Pulmonary Vascular Remodeling

Physiology (Bethesda, Md.), 2006-04, Vol.21 (2), p.134-145

2006

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Hypoxia-Induced Pulmonary Vascular Remodeling Requires Recruitment of Circulating Mesenchymal Precursors of a Monocyte/Macrophage Lineage

The American journal of pathology, 2006-02, Vol.168 (2), p.659-669

2006

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Vascular Remodeling Versus Vasoconstriction in Chronic Hypoxic Pulmonary Hypertension: A Time for Reappraisal?

Circulation research, 2005-07, Vol.97 (2), p.95-98

2005

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Distensibility of the normal human lung circulation during exercise

American journal of physiology. Lung cellular and molecular physiology, 2005-03, Vol.288 (3), p.L419-L425

2005

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Mature Vascular Endothelium Can Give Rise to Smooth Muscle Cells via Endothelial-Mesenchymal Transdifferentiation: In Vitro Analysis

Circulation research, 2002-06, Vol.90 (11), p.1189-1196

2002

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Hypoxic activation of adventitial fibroblasts: Role in vascular remodeling

Chest, 2002-12, Vol.122 (6), p.326-334

2002

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Hypoxia-induced pulmonary artery adventitial remodeling and neovascularization: contribution of progenitor cells

American journal of physiology. Lung cellular and molecular physiology, 2004-04, Vol.286 (4), p.668-L678

2004

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Hypoxia, leukocytes, and the pulmonary circulation

Journal of applied physiology (1985), 2005-02, Vol.98 (2), p.715-721

2005

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Cellular and molecular mechanisms of pulmonary vascular remodeling

Annual review of physiology, 1997-01, Vol.59 (1), p.89-144

1997

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Activation of phosphatidylinositol 3-kinase, Akt, and mammalian target of rapamycin is necessary for hypoxia-induced pulmonary artery adventitial fibroblast proliferation

Journal of applied physiology (1985), 2005-02, Vol.98 (2), p.722-731

2005

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Hypoxia induces differentiation of pulmonary artery adventitial fibroblasts into myofibroblasts

American Journal of Physiology: Cell Physiology, 2004-02, Vol.286 (2), p.C416-C425

2004

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Extracellular ATP is an autocrine/paracrine regulator of hypoxia-induced adventitial fibroblast growth. Signaling through extracellular signal-regulated kinase-1/2 and the Egr-1 transcription factor

The Journal of biological chemistry, 2002-11, Vol.277 (47), p.44638-44650

2002

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Unique, highly proliferative growth phenotype expressed by embryonic and neointimal smooth muscle cells is driven by constitutive Akt, mTOR, and p70S6K signaling and is actively repressed by PTEN

Circulation (New York, N.Y.), 2004-03, Vol.109 (10), p.1299-1306

2004

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Endothelin-mediated increases in lung VEGF content promote vascular leak in young rats exposed to viral infection and hypoxia

American journal of physiology. Lung cellular and molecular physiology, 2005-12, Vol.289 (6), p.L1075-L1082

2005

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cAMP Response Element-binding Protein Content Is a Molecular Determinant of Smooth Muscle Cell Proliferation and Migration

The Journal of biological chemistry, 2001-12, Vol.276 (49), p.46132-46141

2001

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Extracellular ATP-induced Proliferation of Adventitial Fibroblasts Requires Phosphoinositide 3-Kinase, Akt, Mammalian Target of Rapamycin, and p70 S6 Kinase Signaling Pathways

The Journal of biological chemistry, 2005-01, Vol.280 (3), p.1838-1848

2005

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Smooth Muscle Cells Isolated From Discrete Compartments of the Mature Vascular Media Exhibit Unique Phenotypes and Distinct Growth Capabilities

Circulation research, 1997-12, Vol.81 (6), p.940-952

1997

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Hypoxia decreases lung neprilysin expression and increases pulmonary vascular leak

American journal of physiology. Lung cellular and molecular physiology, 2001-10, Vol.281 (4), p.941-L948

2001

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Hypoxia-induced Proliferative Response of Vascular Adventitial Fibroblasts Is Dependent on G Protein-mediated Activation of Mitogen-activated Protein Kinases

The Journal of biological chemistry, 2001-05, Vol.276 (19), p.15631-15640

2001

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Angiogenic therapy for bronchopulmonary dysplasia: rationale and promise

Circulation (New York, N.Y.), 2005-10, Vol.112 (16), p.2383-2385

2005

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Induction of SM-α-actin expression by mechanical strain in adult vascular smooth muscle cells is mediated through activation of JNK and p38 MAP kinase

Biochemical and biophysical research communications, 2003-02, Vol.301 (4), p.1116-1121

2003

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Lung vascular development: breathing new life into an old problem

American journal of respiratory cell and molecular biology, 2003-02, Vol.28 (2), p.133-137

2003

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Egr-1 antisense oligonucleotides inhibit hypoxia-induced proliferation of pulmonary artery adventitial fibroblasts

Journal of applied physiology (1985), 2005-02, Vol.98 (2), p.732-738

2005

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Lung vascular development: implications for the pathogenesis of bronchopulmonary dysplasia

Cellular and molecular pathobiology of pulmonary arterial hypertension

Role of the Adventitia in Pulmonary Vascular Remodeling

Hypoxia-Induced Pulmonary Vascular Remodeling Requires Recruitment of Circulating Mesenchymal Precursors of a Monocyte/Macrophage Lineage

Vascular Remodeling Versus Vasoconstriction in Chronic Hypoxic Pulmonary Hypertension: A Time for Reappraisal?

Distensibility of the normal human lung circulation during exercise

Mature Vascular Endothelium Can Give Rise to Smooth Muscle Cells via Endothelial-Mesenchymal Transdifferentiation: In Vitro Analysis

Hypoxic activation of adventitial fibroblasts: Role in vascular remodeling

Hypoxia-induced pulmonary artery adventitial remodeling and neovascularization: contribution of progenitor cells

Hypoxia, leukocytes, and the pulmonary circulation

Cellular and molecular mechanisms of pulmonary vascular remodeling

Activation of phosphatidylinositol 3-kinase, Akt, and mammalian target of rapamycin is necessary for hypoxia-induced pulmonary artery adventitial fibroblast proliferation

Hypoxia induces differentiation of pulmonary artery adventitial fibroblasts into myofibroblasts

Extracellular ATP is an autocrine/paracrine regulator of hypoxia-induced adventitial fibroblast growth. Signaling through extracellular signal-regulated kinase-1/2 and the Egr-1 transcription factor

Unique, highly proliferative growth phenotype expressed by embryonic and neointimal smooth muscle cells is driven by constitutive Akt, mTOR, and p70S6K signaling and is actively repressed by PTEN

Endothelin-mediated increases in lung VEGF content promote vascular leak in young rats exposed to viral infection and hypoxia

cAMP Response Element-binding Protein Content Is a Molecular Determinant of Smooth Muscle Cell Proliferation and Migration

Extracellular ATP-induced Proliferation of Adventitial Fibroblasts Requires Phosphoinositide 3-Kinase, Akt, Mammalian Target of Rapamycin, and p70 S6 Kinase Signaling Pathways

Smooth Muscle Cells Isolated From Discrete Compartments of the Mature Vascular Media Exhibit Unique Phenotypes and Distinct Growth Capabilities

Hypoxia decreases lung neprilysin expression and increases pulmonary vascular leak

Hypoxia-induced Proliferative Response of Vascular Adventitial Fibroblasts Is Dependent on G Protein-mediated Activation of Mitogen-activated Protein Kinases

Angiogenic therapy for bronchopulmonary dysplasia: rationale and promise

Induction of SM-α-actin expression by mechanical strain in adult vascular smooth muscle cells is mediated through activation of JNK and p38 MAP kinase

Lung vascular development: breathing new life into an old problem

Egr-1 antisense oligonucleotides inhibit hypoxia-induced proliferation of pulmonary artery adventitial fibroblasts

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