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Excess Brain Protein Oxidation and Enzyme Dysfunction in Normal Aging and in Alzheimer Disease

Proceedings of the National Academy of Sciences - PNAS, 1991-12, Vol.88 (23), p.10540-10543

1991

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Reversal of Age-Related Increase in Brain Protein Oxidation, Decrease in Enzyme Activity, and Loss in Temporal and Spatial Memory by Chronic Administration of the Spin-Trapping Compound N-Tert-Butyl-α- Phenylnitrone

Proceedings of the National Academy of Sciences - PNAS, 1991-05, Vol.88 (9), p.3633-3636

1991

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Oxidative Damage to Brain Proteins, Loss of Glutamine Synthetase Activity, and Production of Free Radicals During Ischemia/Reperfusion-Induced Injury to Gerbil Brain

Proceedings of the National Academy of Sciences - PNAS, 1990-07, Vol.87 (13), p.5144-5147

1990

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Neurobiology of eating behavior, nutrition and health

Journal of internal medicine, 2023-11, Vol.294 (5), p.582-604

2023

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Human implications of caloric restriction's effects on aging in laboratory animals: an overview of opportunities for research

The journals of gerontology. Series A, Biological sciences and medical sciences, 2001-03, Vol.56 Spec No 1 (Supplement 1), p.5-6

2001

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Protein oxidation and proteolysis during aging and oxidative stress

Archives of biochemistry and biophysics, 1989-12, Vol.275 (2), p.559-567

1989

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Exercise, oxidative damage and effects of antioxidant manipulation

The Journal of nutrition, 1992-03, Vol.122 (3S), p.766-773

1992

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Free radical scavenging is involved in the protective effect of l-propionyl-carnitine against ischemia-reperfusion injury of the heart

Archives of biochemistry and biophysics, 1991-08, Vol.288 (2), p.533-537

1991

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Funds for nutrition related research. Aging

The Journal of nutrition, 1996-10, Vol.126 (10), p.2593-2594

Starke-Reed, P E

1996

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Green tea extract suppresses the age-related increase in Collagen crosslinking and fluorescent products in C57BL/6 mice

International journal for vitamin and nutrition research, 2003-11, Vol.73 (6), p.453-460

2003

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Summary of the 2000 Surgeon General's Listening Session: Toward a National Action Plan on Overweight and Obesity

Obesity (Silver Spring, Md.), 2002-12, Vol.10 (12), p.1299-1305

2002

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Protein oxidation and myelinolysis occur in brain following rapid correction of hyponatremia

Biochemical and biophysical research communications, 1990-10, Vol.172 (1), p.92-97

1990

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Antioxidants: strategies for interventions in aging and age-related diseases. A workshop sponsored by the National Institute on Aging and by the Office of Dietary Supplements

Antioxidants & redox signaling, 2000, Vol.2 (3), p.375-377

2000

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Age-Related Oxidation Reaction in Proteins

Toxicology and industrial health, 1993-01, Vol.9 (1-2), p.187-196

1993

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Inhibition of postcardiac arrest brain protein oxidation by acetyl-l-carnitine

Free radical biology & medicine, 1993-12, Vol.15 (6), p.667-670

1993

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The role of oxidative modification in cellular protein turnover and aging

Progress in clinical and biological research, 1989, Vol.287, p.269

Starke-Reed, P E

1989

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Protein modification in aging

EXS (Basel), 1992, Vol.62, p.64

1992

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Oxidative modification of enzymes during aging and acute oxidative stress

Basic life sciences, 1988, Vol.49, p.537

1988

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Daily administration of α-phneyl-N-tert-butyl nitrone (PBN) decreases the level of oxidized brain protein and increases brain glutamine synthetase activity in aged gerbils

Free radical biology & medicine, 1990, Vol.9, p.84-84

1990

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Reversal of age-related increase in brain protein oxidation, decrease in enzyme activity, and loss in temporal and spatial memory by chronic administration of the spintrapping compound n-tert-butyl-α-phenylnitrone

Proceedings of the National Academy of Sciences - PNAS, 1991, Vol.88 (9), p.3633-3636

1991

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Rapid correction of severe hyponatremia results in myelinolysis, brain protein oxidation, and altered blood chemistries

Annals of emergency medicine, 1989-04, Vol.18 (4), p.460-461

1989

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Optimization of Electrospray Ionization Source Parameters for Lipidomics To Reduce Misannotation of In-Source Fragments as Precursor Ions

Analytical chemistry (Washington), 2018-11, Vol.90 (22), p.13523-13532

2018

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National Institutes of Health state-of-the-science conference statement : Multivitamin/mineral supplements and chronic disease prevention

The American journal of clinical nutrition, 2007, Vol.85 (1), p.257S-264S

National Institutes of Health State-of-the-Science Panel

2007

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Excess Brain Protein Oxidation and Enzyme Dysfunction in Normal Aging and in Alzheimer Disease

Reversal of Age-Related Increase in Brain Protein Oxidation, Decrease in Enzyme Activity, and Loss in Temporal and Spatial Memory by Chronic Administration of the Spin-Trapping Compound N-Tert-Butyl-α- Phenylnitrone

Oxidative Damage to Brain Proteins, Loss of Glutamine Synthetase Activity, and Production of Free Radicals During Ischemia/Reperfusion-Induced Injury to Gerbil Brain

Neurobiology of eating behavior, nutrition and health

Human implications of caloric restriction's effects on aging in laboratory animals: an overview of opportunities for research

Protein oxidation and proteolysis during aging and oxidative stress

Exercise, oxidative damage and effects of antioxidant manipulation

Free radical scavenging is involved in the protective effect of l-propionyl-carnitine against ischemia-reperfusion injury of the heart

Funds for nutrition related research. Aging

Green tea extract suppresses the age-related increase in Collagen crosslinking and fluorescent products in C57BL/6 mice

Summary of the 2000 Surgeon General's Listening Session: Toward a National Action Plan on Overweight and Obesity

Protein oxidation and myelinolysis occur in brain following rapid correction of hyponatremia

Antioxidants: strategies for interventions in aging and age-related diseases. A workshop sponsored by the National Institute on Aging and by the Office of Dietary Supplements

Age-Related Oxidation Reaction in Proteins

Inhibition of postcardiac arrest brain protein oxidation by acetyl-l-carnitine

The role of oxidative modification in cellular protein turnover and aging

Protein modification in aging

Oxidative modification of enzymes during aging and acute oxidative stress

Daily administration of α-phneyl-N-tert-butyl nitrone (PBN) decreases the level of oxidized brain protein and increases brain glutamine synthetase activity in aged gerbils

Reversal of age-related increase in brain protein oxidation, decrease in enzyme activity, and loss in temporal and spatial memory by chronic administration of the spintrapping compound n-tert-butyl-α-phenylnitrone

Rapid correction of severe hyponatremia results in myelinolysis, brain protein oxidation, and altered blood chemistries

Optimization of Electrospray Ionization Source Parameters for Lipidomics To Reduce Misannotation of In-Source Fragments as Precursor Ions

National Institutes of Health state-of-the-science conference statement : Multivitamin/mineral supplements and chronic disease prevention

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