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MiR-486 and miR-92a Identified in Circulating HDL Discriminate between Stable and Vulnerable Coronary Artery Disease Patients

PloS one, 2015-10, Vol.10 (10), p.e0140958-e0140958

2015

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Elevated Levels of Circulating lncRNAs LIPCAR and MALAT1 Predict an Unfavorable Outcome in Acute Coronary Syndrome Patients

International journal of molecular sciences, 2023-08, Vol.24 (15), p.12076

2023

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Aggregated LDL turn human macrophages into foam cells and induce mitochondrial dysfunction without triggering oxidative or endoplasmic reticulum stress

PloS one, 2021-01, Vol.16 (1), p.e0245797-e0245797

2021

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LHCOPN and LHCONE: Status and Future Evolution

Journal of physics. Conference series, 2015-12, Vol.664 (5), p.52025

2015

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Increased miR-142 Levels in Plasma and Atherosclerotic Plaques from Peripheral Artery Disease Patients with Post-Surgery Cardiovascular Events

International journal of molecular sciences, 2020-12, Vol.21 (24), p.9600

2020

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Regulation of microRNAs in high-fat diet induced hyperlipidemic hamsters

Scientific reports, 2020-11, Vol.10 (1), p.20549-20549, Article 20549

2020

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Mitochondrial DNA Together with miR-142-3p in Plasma Can Predict Unfavorable Outcomes in Patients after Acute Myocardial Infarction

International journal of molecular sciences, 2022-09, Vol.23 (17), p.9947

2022

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Hyperglycemia Determines Increased Specific MicroRNAs Levels in Sera and HDL of Acute Coronary Syndrome Patients and Stimulates MicroRNAs Production in Human Macrophages

PloS one, 2016-08, Vol.11 (8), p.e0161201-e0161201

2016

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Dysfunctional high-density lipoproteins have distinct composition, diminished anti-inflammatory potential and discriminate acute coronary syndrome from stable coronary artery disease patients

Scientific reports, 2017-08, Vol.7 (1), p.7295-13, Article 7295

2017

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Oscillating Glucose Induces the Increase in Inflammatory Stress through Ninjurin-1 Up-Regulation and Stimulation of Transport Proteins in Human Endothelial Cells

Biomolecules (Basel, Switzerland), 2023-03, Vol.13 (4), p.626

2023

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Irreversibly glycated LDL induce oxidative and inflammatory state in human endothelial cells; added effect of high glucose

Biochemical and biophysical research communications, 2009-12, Vol.390 (3), p.877-882

2009

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Anti-oxidant and anti-inflammatory mechanisms of amlodipine action to improve endothelial cell dysfunction induced by irreversibly glycated LDL

Biochemical and biophysical research communications, 2011-07, Vol.411 (1), p.202-207

2011

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CRISPR/dCas9 Transcriptional Activation of Endogenous Apolipoprotein AI and Paraoxonase 1 in Enterocytes Alleviates Endothelial Cell Dysfunction

Biomolecules (Basel, Switzerland), 2021-11, Vol.11 (12), p.1769

2021

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The Carrier Status of Streptococcus pneumoniae in a Multi-National Medical Student Population

Annali di igiene, 2020-01, Vol.32 (1), p.65-71

2020

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Nutritional or Active Vitamin D for the Correction of Mineral Metabolism Abnormalities in Non-Dialysis Chronic Kidney Disease Patients?

Acta endocrinologica (Bucharest, Romania : 2005), 2018-10, Vol.14 (4), p.503-513

Stancu, S

2018

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AB0470 Pauciimmune vasculitis and renal failure- 14 years experience in the “c.davila” nephrology teaching hospital- bucharest

Annals of the rheumatic diseases, 2013-06, Vol.72 (Suppl 3), p.A933-A933

2013

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Vascular endothelium in atherosclerosis

Cell and tissue research, 2009, Vol.335 (1), p.191-203

2009

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Comparative Analysis of Immediate Postoperative Complications Following Total Gastrectomy

Polski przeglad chirurgiczny, 2017-06, Vol.89 (3), p.1-6

2017

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Dual role of lipoproteins in endothelial cell dysfunction in atherosclerosis

Cell and tissue research, 2012-08, Vol.349 (2), p.433-446

2012

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NUTRITIONAL OR ACTIVE VITAMIN D FOR THE CORRECTION OF MINERAL METABOLISM ABNORMALITIES IN NON-DIALYSIS CHRONIC KIDNEY DISEASE PATIENTS?

Acta endocrinologica (Bucharest, Romania : 2005), 2018-10, Vol.14 (4), p.505-513

2018

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Novel molecular mechanisms by which ginger extract reduces the inflammatory stress in TNFα – activated human endothelial cells; decrease of Ninjurin-1, TNFR1 and NADPH oxidase subunits expression

Journal of functional foods, 2018-09, Vol.48, p.654-664

2018

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Ninjurin-1 upregulated by TNFα receptor 1 stimulates monocyte adhesion to human TNFα-activated endothelial cells; benefic effects of amlodipine

Life sciences (1973), 2020-05, Vol.249, p.117518-117518, Article 117518

2020

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Probiotics administration or the high-fat diet arrest modulates microRNAs levels in hyperlipidemic hamsters

Journal of functional foods, 2019-05, Vol.56, p.295-302

2019

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Integrating network and transfer metrics to optimize transfer efficiency and experiment workflows

Journal of physics. Conference series, 2015-12, Vol.664 (5), p.52003

2015

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Caffeic acid attenuates the inflammatory stress induced by glycated LDL in human endothelial cells by mechanisms involving inhibition of AGE‐receptor, oxidative, and endoplasmic reticulum stress

BioFactors (Oxford), 2017-09, Vol.43 (5), p.685-697

2017

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MiR-486 and miR-92a Identified in Circulating HDL Discriminate between Stable and Vulnerable Coronary Artery Disease Patients

Elevated Levels of Circulating lncRNAs LIPCAR and MALAT1 Predict an Unfavorable Outcome in Acute Coronary Syndrome Patients

Aggregated LDL turn human macrophages into foam cells and induce mitochondrial dysfunction without triggering oxidative or endoplasmic reticulum stress

LHCOPN and LHCONE: Status and Future Evolution

Increased miR-142 Levels in Plasma and Atherosclerotic Plaques from Peripheral Artery Disease Patients with Post-Surgery Cardiovascular Events

Regulation of microRNAs in high-fat diet induced hyperlipidemic hamsters

Mitochondrial DNA Together with miR-142-3p in Plasma Can Predict Unfavorable Outcomes in Patients after Acute Myocardial Infarction

Hyperglycemia Determines Increased Specific MicroRNAs Levels in Sera and HDL of Acute Coronary Syndrome Patients and Stimulates MicroRNAs Production in Human Macrophages

Dysfunctional high-density lipoproteins have distinct composition, diminished anti-inflammatory potential and discriminate acute coronary syndrome from stable coronary artery disease patients

Oscillating Glucose Induces the Increase in Inflammatory Stress through Ninjurin-1 Up-Regulation and Stimulation of Transport Proteins in Human Endothelial Cells

Irreversibly glycated LDL induce oxidative and inflammatory state in human endothelial cells; added effect of high glucose

Anti-oxidant and anti-inflammatory mechanisms of amlodipine action to improve endothelial cell dysfunction induced by irreversibly glycated LDL

CRISPR/dCas9 Transcriptional Activation of Endogenous Apolipoprotein AI and Paraoxonase 1 in Enterocytes Alleviates Endothelial Cell Dysfunction

The Carrier Status of Streptococcus pneumoniae in a Multi-National Medical Student Population

Nutritional or Active Vitamin D for the Correction of Mineral Metabolism Abnormalities in Non-Dialysis Chronic Kidney Disease Patients?

AB0470 Pauciimmune vasculitis and renal failure- 14 years experience in the “c.davila” nephrology teaching hospital- bucharest

Vascular endothelium in atherosclerosis

Comparative Analysis of Immediate Postoperative Complications Following Total Gastrectomy

Dual role of lipoproteins in endothelial cell dysfunction in atherosclerosis

NUTRITIONAL OR ACTIVE VITAMIN D FOR THE CORRECTION OF MINERAL METABOLISM ABNORMALITIES IN NON-DIALYSIS CHRONIC KIDNEY DISEASE PATIENTS?

Novel molecular mechanisms by which ginger extract reduces the inflammatory stress in TNFα – activated human endothelial cells; decrease of Ninjurin-1, TNFR1 and NADPH oxidase subunits expression

Ninjurin-1 upregulated by TNFα receptor 1 stimulates monocyte adhesion to human TNFα-activated endothelial cells; benefic effects of amlodipine

Probiotics administration or the high-fat diet arrest modulates microRNAs levels in hyperlipidemic hamsters

Integrating network and transfer metrics to optimize transfer efficiency and experiment workflows

Caffeic acid attenuates the inflammatory stress induced by glycated LDL in human endothelial cells by mechanisms involving inhibition of AGE‐receptor, oxidative, and endoplasmic reticulum stress

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