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Critical data-based re-evaluation of minocycline as a putative specific microglia inhibitor

Glia, 2016-10, Vol.64 (10), p.1788-1794

2016

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Fluorescent False Neurotransmitters Visualize Dopamine Release from Individual Presynaptic Terminals

Science (American Association for the Advancement of Science), 2009-06, Vol.324 (5933), p.1441-1444

2009

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Inhibition of P2X7 receptors by Lu AF27139 diminishes colonic hypersensitivity and CNS prostanoid levels in a rat model of visceral pain

Purinergic signalling, 2022-12, Vol.18 (4), p.499-514

2022

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Senicapoc: Repurposing a Drug to Target Microglia KCa3.1 in Stroke

Neurochemical research, 2017-09, Vol.42 (9), p.2639-2645

2017

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KCa3.1-a microglial target ready for drug repurposing?

Glia, 2016-10, Vol.64 (10), p.1733-1741

2016

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Alpha-synuclein overexpression increases cytosolic catecholamine concentration

The Journal of neuroscience, 2006-09, Vol.26 (36), p.9304-9311

2006

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Microglial Drug Targets in AD: Opportunities and Challenges in Drug Discovery and Development

Frontiers in pharmacology, 2019-08, Vol.10, p.840-840

2019

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PKC θ activity maintains normal quantal size in chromaffin cells

Journal of neurochemistry, 2008-06, Vol.105 (5), p.1635-1641

2008

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Sorting of Vesicular Monoamine Transporter 2 to the Regulated Secretory Pathway Confers the Somatodendritic Exocytosis of Monoamines

Neuron (Cambridge, Mass.), 2005-11, Vol.48 (4), p.619-633

2005

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Dopamine neurons release transmitter via a flickering fusion pore

Nature neuroscience, 2004-04, Vol.7 (4), p.341-346

2004

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Dopamine release at individual presynaptic terminals visualized with FFNs

Journal of visualized experiments, 2009-08 (30)

2009

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K Ca 3.1—a microglial target ready for drug repurposing?

Glia, 2016-10, Vol.64 (10), p.1733-1741

2016

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Senicapoc: Repurposing a Drug to Target Microglia K^sub Ca^3.1 in Stroke

Neurochemical research, 2017-09, Vol.42 (9), p.2639

2017

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Inhibition of the potassium channel KCa3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury

European journal of pharmacology, 2017-01, Vol.795, p.1-7

2017

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Analysis of VMAT2 Binding After Methamphetamine or MPTP Treatment

Journal of neurochemistry, 2000-05, Vol.74 (5), p.2217-2220

2000

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Inhibition of the potassium channel K Ca 3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury

European journal of pharmacology, 2017-01, Vol.795, p.1-7

2017

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KCa 3.1-a microglial target ready for drug repurposing?

Glia, 2016-10, Vol.64 (10), p.1733-1741

2016

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Senicapoc: Repurposing a Drug to Target Microglia K Ca 3.1 in Stroke

Neurochemical research, 2017-09, Vol.42 (9), p.2639

2017

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K sub(Ca)3.1-a microglial target ready for drug repurposing?

Glia, 2016-10, Vol.64 (10), p.1733-1741

2016

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Analysis of VMAT2 binding after methamphetamine or MPTP treatment: disparity between homogenates and vesicle preparations

Journal of neurochemistry, 2000-05, Vol.74 (5), p.2217-2220

2000

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PKC 0 activity maintains normal quantal size in chromaffin cells

Journal of neurochemistry, 2008, Vol.105 (5), p.1635-1641

2008

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Recent Developments in Targeting Neuroinflammation in Disease

Annual Reports in Medicinal Chemistry, 2012, Vol.47, p.37-53

2012

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PKC [theta] activity maintains normal quantal size in chromaffin cells

Journal of neurochemistry, 2008-06, Vol.105 (5), p.1635-1641

2008

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Inhibition of brain vesicular monoamine transporter (VMAT2) enhances 1-methyl-4-phenylpyridinium neurotoxicity in vivo in rat striata

The Journal of pharmacology and experimental therapeutics, 2000-05, Vol.293 (2), p.336

2000

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Dopamine Release at Individual Presynaptic Terminals Visualized with FFNs

Journal of visualized experiments, 2009-08 (30)

2009

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Critical data-based re-evaluation of minocycline as a putative specific microglia inhibitor

Fluorescent False Neurotransmitters Visualize Dopamine Release from Individual Presynaptic Terminals

Inhibition of P2X7 receptors by Lu AF27139 diminishes colonic hypersensitivity and CNS prostanoid levels in a rat model of visceral pain

Senicapoc: Repurposing a Drug to Target Microglia KCa3.1 in Stroke

KCa3.1-a microglial target ready for drug repurposing?

Alpha-synuclein overexpression increases cytosolic catecholamine concentration

Microglial Drug Targets in AD: Opportunities and Challenges in Drug Discovery and Development

PKC θ activity maintains normal quantal size in chromaffin cells

Sorting of Vesicular Monoamine Transporter 2 to the Regulated Secretory Pathway Confers the Somatodendritic Exocytosis of Monoamines

Dopamine neurons release transmitter via a flickering fusion pore

Dopamine release at individual presynaptic terminals visualized with FFNs

K Ca 3.1—a microglial target ready for drug repurposing?

Senicapoc: Repurposing a Drug to Target Microglia K^sub Ca^3.1 in Stroke

Inhibition of the potassium channel KCa3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury

Analysis of VMAT2 Binding After Methamphetamine or MPTP Treatment

Inhibition of the potassium channel K Ca 3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury

KCa 3.1-a microglial target ready for drug repurposing?

Senicapoc: Repurposing a Drug to Target Microglia K Ca 3.1 in Stroke

K sub(Ca)3.1-a microglial target ready for drug repurposing?

Analysis of VMAT2 binding after methamphetamine or MPTP treatment: disparity between homogenates and vesicle preparations

PKC 0 activity maintains normal quantal size in chromaffin cells

Recent Developments in Targeting Neuroinflammation in Disease

PKC [theta] activity maintains normal quantal size in chromaffin cells

Inhibition of brain vesicular monoamine transporter (VMAT2) enhances 1-methyl-4-phenylpyridinium neurotoxicity in vivo in rat striata

Dopamine Release at Individual Presynaptic Terminals Visualized with FFNs

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