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Soluble oligomers of amyloid-beta peptide induce neuronal apoptosis by activating a cPLA2-dependent sphingomyelinase-ceramide pathway

Neurobiology of disease, 2006-07, Vol.23 (1), p.178-189

2006

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The BMDC model, a performant cell‐based test to assess the sensitizing potential and potency of chemicals including pre/pro‐haptens

Contact dermatitis, 2024-03, Vol.90 (3), p.211-234

2024

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In vitro detection of chemical allergens: an optimized assay using mouse bone marrow‐derived dendritic cells

Contact dermatitis, 2017-11, Vol.77 (5), p.311-322

2017

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Evaluation of chromium in red blood cells as an indicator of exposure to hexavalent chromium: An in vitro study

Toxicology letters, 2016-07, Vol.255, p.63-70

2016

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A new cytometry-based method reveals an accumulation of Nrf2 in dendritic cells exposed to two respiratory sensitizers

Toxicology research (Cambridge), 2021-12, Vol.10 (6), p.1223-1227

2021

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Activation of T cells by dendritic cells exposed to a reference sensitizer: Towards a promising model to assess the allergenic potential of chemicals

Contact dermatitis, 2018-08, Vol.79 (2), p.67-75

2018

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De nouveaux tests pour évaluer le caractère sensibilisant d’une substance industrielle

Archives des maladies professionnelles et de l'environnement, 2020-10, Vol.81 (5), p.489-489

2020

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H2S induced hypometabolism in mice is missing in sedated sheep

Respiratory physiology & neurobiology, 2008, Vol.160 (1), p.109-115

2008

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Development of an exposure indicator to hexavalent chromium: An in vitro evaluation with human blood

Toxicology letters, 2017-10, Vol.280, p.S178-S178

2017

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Docosahexaenoic acid prevents neuronal apoptosis induced by soluble amyloid‐β oligomers

Journal of neurochemistry, 2006-01, Vol.96 (2), p.385-395

2006

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Microtubule-associated protein MAP1A, MAP1B, and MAP2 proteolysis during soluble amyloid beta-peptide-induced neuronal apoptosis. Synergistic involvement of calpain and caspase-3

The Journal of biological chemistry, 2006-01, Vol.281 (1), p.229-240

2006

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Inhibition of vitamin B12 metabolism by OH-cobalamin c-lactam in rat oligodendrocytes in culture: a model for studying neuropathy due to vitamin B12 deficiency

Neuroscience letters, 2000-07, Vol.288 (3), p.191-194

2000

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Cytosolic phospholipase A2 mediates neuronal apoptosis induced by soluble oligomers of the amyloid‐β peptide

The FASEB journal, 2005-01, Vol.19 (1), p.85-87

2005

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Membrane cholesterol interferes with neuronal apoptosis induced by soluble oligomers but not fibrils of the amyloid‐β peptide

The FASEB journal, 2004-05, Vol.18 (7), p.836-838

2004

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Humanin rescues cortical neurons from prion-peptide-induced apoptosis

Molecular and cellular neuroscience, 2004-01, Vol.25 (1), p.95-102

2004

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Apoptotic neuronal cell death induced by the non-fibrillar amyloid-beta peptide proceeds through an early reactive oxygen species-dependent cytoskeleton perturbation

The Journal of biological chemistry, 2003-01, Vol.278 (5), p.3437-3445

2003

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In Vivo and In Vitro Neurotoxicity of the Human Prion Protein (PrP) Fragment P118-135 Independently of PrP Expression

The Journal of neuroscience, 2003-01, Vol.23 (2), p.462-469

2003

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ALTERED KINETICS OF HYPOXIA INDUCED HYPOMETABOLISM IN LEPTIN DEFICIENT MICE

The FASEB journal, 2007, Vol.21 (6), p.LB113-LB113

2007

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H 2S induced hypometabolism in mice is missing in sedated sheep

Respiratory physiology & neurobiology, 2008, Vol.160 (1), p.109-115

2008

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H2 S induced hypometabolism in mice is missing in sedated sheep

Respiratory physiology & neurobiology, 2007, Vol.160 (1), p.109-115

2007

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Microtubule-associated Protein MAP1A, MAP1B, and MAP2 Proteolysis during Soluble Amyloid β-Peptide-induced Neuronal Apoptosis

The Journal of biological chemistry, 2006-01, Vol.281 (1), p.229-240

2006

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Microtubule-associated Protein MAP1A, MAP1B, and MAP2 Proteolysis during Soluble Amyloid Î²-Peptide-induced Neuronal Apoptosis

The Journal of biological chemistry, 2006-01, Vol.281 (1), p.229

2006

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Soluble oligomers of amyloid-β peptide induce neuronal apoptosis by activating a cPLA 2-dependent sphingomyelinase-ceramide pathway

Neurobiology of disease, 2006, Vol.23 (1), p.178-189

2006

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Oligodendrocytes are susceptible to apoptotic cell death induced by prion protein-derived peptides

Glia, 2004-07, Vol.47 (1), p.1-8

2004

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Cytosolic phospholipase A 2 mediates neuronal apoptosis induced by soluble oligomers of the amyloid‐β peptide

The FASEB journal, 2005-01, Vol.19 (1), p.85-87

2005

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Soluble oligomers of amyloid-beta peptide induce neuronal apoptosis by activating a cPLA2-dependent sphingomyelinase-ceramide pathway

The BMDC model, a performant cell‐based test to assess the sensitizing potential and potency of chemicals including pre/pro‐haptens

In vitro detection of chemical allergens: an optimized assay using mouse bone marrow‐derived dendritic cells

Evaluation of chromium in red blood cells as an indicator of exposure to hexavalent chromium: An in vitro study

A new cytometry-based method reveals an accumulation of Nrf2 in dendritic cells exposed to two respiratory sensitizers

Activation of T cells by dendritic cells exposed to a reference sensitizer: Towards a promising model to assess the allergenic potential of chemicals

De nouveaux tests pour évaluer le caractère sensibilisant d’une substance industrielle

H2S induced hypometabolism in mice is missing in sedated sheep

Development of an exposure indicator to hexavalent chromium: An in vitro evaluation with human blood

Docosahexaenoic acid prevents neuronal apoptosis induced by soluble amyloid‐β oligomers

Microtubule-associated protein MAP1A, MAP1B, and MAP2 proteolysis during soluble amyloid beta-peptide-induced neuronal apoptosis. Synergistic involvement of calpain and caspase-3

Inhibition of vitamin B12 metabolism by OH-cobalamin c-lactam in rat oligodendrocytes in culture: a model for studying neuropathy due to vitamin B12 deficiency

Cytosolic phospholipase A2 mediates neuronal apoptosis induced by soluble oligomers of the amyloid‐β peptide

Membrane cholesterol interferes with neuronal apoptosis induced by soluble oligomers but not fibrils of the amyloid‐β peptide

Humanin rescues cortical neurons from prion-peptide-induced apoptosis

Apoptotic neuronal cell death induced by the non-fibrillar amyloid-beta peptide proceeds through an early reactive oxygen species-dependent cytoskeleton perturbation

In Vivo and In Vitro Neurotoxicity of the Human Prion Protein (PrP) Fragment P118-135 Independently of PrP Expression

ALTERED KINETICS OF HYPOXIA INDUCED HYPOMETABOLISM IN LEPTIN DEFICIENT MICE

H 2S induced hypometabolism in mice is missing in sedated sheep

H2 S induced hypometabolism in mice is missing in sedated sheep

Microtubule-associated Protein MAP1A, MAP1B, and MAP2 Proteolysis during Soluble Amyloid β-Peptide-induced Neuronal Apoptosis

Microtubule-associated Protein MAP1A, MAP1B, and MAP2 Proteolysis during Soluble Amyloid Î²-Peptide-induced Neuronal Apoptosis

Soluble oligomers of amyloid-β peptide induce neuronal apoptosis by activating a cPLA 2-dependent sphingomyelinase-ceramide pathway

Oligodendrocytes are susceptible to apoptotic cell death induced by prion protein-derived peptides

Cytosolic phospholipase A 2 mediates neuronal apoptosis induced by soluble oligomers of the amyloid‐β peptide

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