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A Population-Based Analysis of Application of WHO Nomenclature in Pathology Reports of Pulmonary Neuroendocrine Tumors

Journal of thoracic oncology, 2016-04, Vol.11 (4), p.593-602

2016

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Clinical features of large cell neuroendocrine carcinoma: a population-based overview

The European respiratory journal, 2016-02, Vol.47 (2), p.615-624

2016

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Amplification Methods to Increase the Sensitivity of In Situ Hybridization: Play CARD(S)

Journal of Histochemistry & Cytochemistry, 1999-03, Vol.47 (3), p.281-288

1999

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Prevalence of high-risk human papillomavirus infection and cancer gene mutations in nonmalignant tonsils

Oral oncology, 2017-10, Vol.73, p.77-82

2017

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Sensitive Multicolor Fluorescence In Situ Hybridization Using Catalyzed Reporter Deposition (CARD) Amplification

The journal of histochemistry and cytochemistry, 1997-10, Vol.45 (10), p.1439-1446

1997

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Rapid Synthesis of Biotin-, Digoxigenin-, Trinitrophenyl-, and Fluorochrome-labeled Tyramides and Their Application for In Situ Hybridization Using CARD Amplification

The journal of histochemistry and cytochemistry, 1998-06, Vol.46 (6), p.771-777

1998

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Losses of Chromosomes 1p and 3q Are Early Genetic Events in the Development of Sporadic Pheochromocytomas

The American journal of pathology, 2000-08, Vol.157 (2), p.353-359

2000

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Limited additive value of the Ki‐67 proliferative index on patient survival in World Health Organization‐classified pulmonary carcinoids

Histopathology, 2017-02, Vol.70 (3), p.412-422

2017

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Multicenter Evaluation of the Fully Automated PCR-Based Idylla EGFR Mutation Assay on Formalin-Fixed, Paraffin-Embedded Tissue of Human Lung Cancer

The Journal of molecular diagnostics : JMD, 2019-11, Vol.21 (6), p.1010-1024

2019

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Differential Loss of Chromosome 11q in Familial and Sporadic Parasympathetic Paragangliomas Detected by Comparative Genomic Hybridization

The American journal of pathology, 2001-06, Vol.158 (6), p.1937-1942

2001

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Germ-line variants identified by next generation sequencing in a panel of estrogen and cancer associated genes correlate with poor clinical outcome in Lynch syndrome patients

Oncotarget, 2015-12, Vol.6 (38), p.41108-41122

2015

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A subset of head and neck squamous cell carcinomas exhibits integration of HPV 16/18 DNA and overexpression of p16INK4A and p53 in the absence of mutations in p53 exons 5–8

International journal of cancer, 2003-11, Vol.107 (3), p.394-400

2003

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Mutations and Allelic Deletions of the MEN1 Gene Are Associated with a Subset of Sporadic Endocrine Pancreatic and Neuroendocrine Tumors and Not Restricted to Foregut Neoplasms

The American journal of pathology, 1999-02, Vol.154 (2), p.429-436

1999

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Genetic Differences in Endocrine Pancreatic Tumor Subtypes Detected by Comparative Genomic Hybridization

The American journal of pathology, 1999-12, Vol.155 (6), p.1787-1794

1999

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Tyramide signal amplification for DNA and mRNA in situ hybridization

Methods in molecular biology (Clifton, N.J.), 2006, Vol.326, p.33

2006

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Putative Tumor Suppressor Loci at 6q22 and 6q23-q24 Are Involved in the Malignant Progression of Sporadic Endocrine Pancreatic Tumors

The American journal of pathology, 2001-06, Vol.158 (6), p.1903-1911

2001

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Gene expression profiling of pulmonary neuroendocrine neoplasms: A comprehensive overview

Cancer treatment communications, 2015, Vol.4, p.148-160

2015

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Role of human papillomavirus in the development of head and neck squamous cell carcinomas

Acta oto-laryngologica, 2004-05, Vol.124 (4), p.520-526

2004

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Genetic evidence for early divergence of small functioning and nonfunctioning endocrine pancreatic tumors: gain of 9Q34 is an early event in insulinomas

Cancer research (Chicago, Ill.), 2001-07, Vol.61 (13), p.5186-5192

2001

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Gain of chromosome 8q is a frequent finding in pleuropulmonary blastoma

Modern pathology, 2007-11, Vol.20 (11), p.1191-1199

2007

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Mixed Medullary-Follicular Thyroid Carcinoma : Molecular Evidence for a Dual Origin of Tumor Components

The American journal of pathology, 1999-11, Vol.155 (5), p.1499-1509

1999

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Mixed Medullary-Follicular Thyroid Carcinoma

The American journal of pathology, 1999-11, Vol.155 (5), p.1499-1509

1999

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Deletion at 3p25.3-p23 is frequently encountered in endocrine pancreatic tumours and is associated with metastatic progression

The Journal of pathology, 2001-08, Vol.194 (4), p.451-458

2001

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Analysis of genomic alterations in sporadic adrenocortical lesions : Gain of chromosome 17 is an early event in adrenocortical tumorigenesis

The American journal of pathology, 1999-10, Vol.155 (4), p.1039-1045

1999

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Identification of Chromosome 9 Alterations and p53 Accumulation in Isolated Carcinoma in Situ of the Urinary Bladder versus Carcinoma in Situ Associated with Carcinoma

The American journal of pathology, 2002-10, Vol.161 (4), p.1119-1125

2002

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A Population-Based Analysis of Application of WHO Nomenclature in Pathology Reports of Pulmonary Neuroendocrine Tumors

Clinical features of large cell neuroendocrine carcinoma: a population-based overview

Amplification Methods to Increase the Sensitivity of In Situ Hybridization: Play CARD(S)

Prevalence of high-risk human papillomavirus infection and cancer gene mutations in nonmalignant tonsils

Sensitive Multicolor Fluorescence In Situ Hybridization Using Catalyzed Reporter Deposition (CARD) Amplification

Rapid Synthesis of Biotin-, Digoxigenin-, Trinitrophenyl-, and Fluorochrome-labeled Tyramides and Their Application for In Situ Hybridization Using CARD Amplification

Losses of Chromosomes 1p and 3q Are Early Genetic Events in the Development of Sporadic Pheochromocytomas

Limited additive value of the Ki‐67 proliferative index on patient survival in World Health Organization‐classified pulmonary carcinoids

Multicenter Evaluation of the Fully Automated PCR-Based Idylla EGFR Mutation Assay on Formalin-Fixed, Paraffin-Embedded Tissue of Human Lung Cancer

Differential Loss of Chromosome 11q in Familial and Sporadic Parasympathetic Paragangliomas Detected by Comparative Genomic Hybridization

Germ-line variants identified by next generation sequencing in a panel of estrogen and cancer associated genes correlate with poor clinical outcome in Lynch syndrome patients

A subset of head and neck squamous cell carcinomas exhibits integration of HPV 16/18 DNA and overexpression of p16INK4A and p53 in the absence of mutations in p53 exons 5–8

Mutations and Allelic Deletions of the MEN1 Gene Are Associated with a Subset of Sporadic Endocrine Pancreatic and Neuroendocrine Tumors and Not Restricted to Foregut Neoplasms

Genetic Differences in Endocrine Pancreatic Tumor Subtypes Detected by Comparative Genomic Hybridization

Tyramide signal amplification for DNA and mRNA in situ hybridization

Putative Tumor Suppressor Loci at 6q22 and 6q23-q24 Are Involved in the Malignant Progression of Sporadic Endocrine Pancreatic Tumors

Gene expression profiling of pulmonary neuroendocrine neoplasms: A comprehensive overview

Role of human papillomavirus in the development of head and neck squamous cell carcinomas

Genetic evidence for early divergence of small functioning and nonfunctioning endocrine pancreatic tumors: gain of 9Q34 is an early event in insulinomas

Gain of chromosome 8q is a frequent finding in pleuropulmonary blastoma

Mixed Medullary-Follicular Thyroid Carcinoma : Molecular Evidence for a Dual Origin of Tumor Components

Mixed Medullary-Follicular Thyroid Carcinoma

Deletion at 3p25.3-p23 is frequently encountered in endocrine pancreatic tumours and is associated with metastatic progression

Analysis of genomic alterations in sporadic adrenocortical lesions : Gain of chromosome 17 is an early event in adrenocortical tumorigenesis

Identification of Chromosome 9 Alterations and p53 Accumulation in Isolated Carcinoma in Situ of the Urinary Bladder versus Carcinoma in Situ Associated with Carcinoma

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