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Both Transcriptional Regulation and Translational Control of ATF4 Are Central to the Integrated Stress Response

The Journal of biological chemistry, 2010-10, Vol.285 (43), p.33165-33174

2010

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Impact of Age and Insulin-Like Growth Factor-1 on DNA Damage Responses in UV-Irradiated Human Skin

Molecules (Basel, Switzerland), 2017-02, Vol.22 (3), p.356

2017

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IL-4 impairs wound healing potential in the skin by repressing fibronectin expression

Journal of allergy and clinical immunology, 2017-01, Vol.139 (1), p.142-151.e5

2017

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Primary cilia signaling mediates intraocular pressure sensation

Proceedings of the National Academy of Sciences - PNAS, 2014-09, Vol.111 (35), p.12871-12876

2014

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Insulin-like Growth Factor 1 Receptor Signaling Is Required for Optimal ATR-CHK1 Kinase Signaling in Ultraviolet B (UVB)-irradiated Human Keratinocytes

The Journal of biological chemistry, 2017-01, Vol.292 (4), p.1231-1239

2017

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Translational Repression Protects Human Keratinocytes from UVB-Induced Apoptosis through a Discordant eIF2 Kinase Stress Response

Journal of investigative dermatology, 2015-10, Vol.135 (10), p.2502-2511

2015

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Pten Loss Induces Autocrine FGF Signaling to Promote Skin Tumorigenesis

Cell reports (Cambridge), 2014-03, Vol.6 (5), p.818-826

2014

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Creatine and Nicotinamide Prevent Oxidant-Induced Senescence in Human Fibroblasts

Nutrients, 2021-11, Vol.13 (11), p.4102

2021

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Wounding Therapies for Prevention of Photocarcinogenesis

Frontiers in oncology, 2022-01, Vol.11, p.813132

2022

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Estrogen modulates mesenchyme-epidermis interactions in the adult nipple

Development (Cambridge), 2017-04, Vol.144 (8), p.1498-1509

2017

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UVB-induced senescence in human keratinocytes requires a functional insulin-like growth factor-1 receptor and p53

Molecular biology of the cell, 2008-04, Vol.19 (4), p.1346-1353

2008

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Insulin-like growth factor-1 receptor regulates repair of ultraviolet B-induced DNA damage in human keratinocytes in vivo

Molecular oncology, 2016-10, Vol.10 (8), p.1245-1254

2016

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Radiation therapy generates platelet-activating factor agonists

Oncotarget, 2016-04, Vol.7 (15), p.20788-20800

2016

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Multiple mechanisms activate GCN2 eIF2 kinase in response to diverse stress conditions

Nucleic acids research, 2024-02, Vol.52 (4), p.1830-1846

2024

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Fibronectin Promotes Wound Healing in an Atopic Human Skin Xenografting Model

Journal of investigative dermatology, 2024-06, Vol.144 (6), p.1415-1418

2024

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γδ T Cell‒Mediated Wound Healing Is Diminished by Allergic Skin Inflammation

Journal of investigative dermatology, 2022-10, Vol.142 (10), p.2805-2816.e4

2022

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Translational control of a human CDKN1A mRNA splice variant regulates the fate of UVB-irradiated human keratinocytes

Molecular biology of the cell, 2018-01, Vol.29 (1), p.29-41

2018

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E5 Protein of Human Papillomavirus Type 16 Protects Human Foreskin Keratinocytes from UV B-Irradiation-Induced Apoptosis

Journal of Virology, 2002-01, Vol.76 (1), p.220-231

2002

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Human Keratinocyte Differentiation Requires Translational Control by the eIF2α Kinase GCN2

Journal of investigative dermatology, 2017-09, Vol.137 (9), p.1924-1934

2017

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UVB activation of NF-κB in normal human keratinocytes occurs via a unique mechanism

Archives of Dermatological Research, 2007-05, Vol.299 (2), p.93-101

2007

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The eIF2 kinase GCN2 directs keratinocyte collective cell migration during wound healing via coordination of reactive oxygen species and amino acids

The Journal of biological chemistry, 2021-11, Vol.297 (5), p.101257-101257, Article 101257

2021

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Single Ablative Fractional Resurfacing Laser Treatment For Forearm Actinic Keratoses: 6‐Month Follow‐Up Data From An Intrapatient Comparison Between Treated and Untreated Sites

Lasers in surgery and medicine, 2020-01, Vol.52 (1), p.84-87

2020

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Randomized controlled trial of fractionated laser resurfacing on aged skin as prophylaxis against actinic neoplasia

The Journal of clinical investigation, 2021-10, Vol.131 (19), p.1-12

2021

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Ex vivo culture of mouse skin activates an interleukin 1 alpha‐dependent inflammatory response

Experimental dermatology, 2020-01, Vol.29 (1), p.102-106

2020

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Fractionated Laser Resurfacing Corrects the Inappropriate UVB Response in Geriatric Skin

Journal of investigative dermatology, 2012-06, Vol.132 (6), p.1591-1596

2012

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Both Transcriptional Regulation and Translational Control of ATF4 Are Central to the Integrated Stress Response

Impact of Age and Insulin-Like Growth Factor-1 on DNA Damage Responses in UV-Irradiated Human Skin

IL-4 impairs wound healing potential in the skin by repressing fibronectin expression

Primary cilia signaling mediates intraocular pressure sensation

Insulin-like Growth Factor 1 Receptor Signaling Is Required for Optimal ATR-CHK1 Kinase Signaling in Ultraviolet B (UVB)-irradiated Human Keratinocytes

Translational Repression Protects Human Keratinocytes from UVB-Induced Apoptosis through a Discordant eIF2 Kinase Stress Response

Pten Loss Induces Autocrine FGF Signaling to Promote Skin Tumorigenesis

Creatine and Nicotinamide Prevent Oxidant-Induced Senescence in Human Fibroblasts

Wounding Therapies for Prevention of Photocarcinogenesis

Estrogen modulates mesenchyme-epidermis interactions in the adult nipple

UVB-induced senescence in human keratinocytes requires a functional insulin-like growth factor-1 receptor and p53

Insulin-like growth factor-1 receptor regulates repair of ultraviolet B-induced DNA damage in human keratinocytes in vivo

Radiation therapy generates platelet-activating factor agonists

Multiple mechanisms activate GCN2 eIF2 kinase in response to diverse stress conditions

Fibronectin Promotes Wound Healing in an Atopic Human Skin Xenografting Model

γδ T Cell‒Mediated Wound Healing Is Diminished by Allergic Skin Inflammation

Translational control of a human CDKN1A mRNA splice variant regulates the fate of UVB-irradiated human keratinocytes

E5 Protein of Human Papillomavirus Type 16 Protects Human Foreskin Keratinocytes from UV B-Irradiation-Induced Apoptosis

Human Keratinocyte Differentiation Requires Translational Control by the eIF2α Kinase GCN2

UVB activation of NF-κB in normal human keratinocytes occurs via a unique mechanism

The eIF2 kinase GCN2 directs keratinocyte collective cell migration during wound healing via coordination of reactive oxygen species and amino acids

Single Ablative Fractional Resurfacing Laser Treatment For Forearm Actinic Keratoses: 6‐Month Follow‐Up Data From An Intrapatient Comparison Between Treated and Untreated Sites

Randomized controlled trial of fractionated laser resurfacing on aged skin as prophylaxis against actinic neoplasia

Ex vivo culture of mouse skin activates an interleukin 1 alpha‐dependent inflammatory response

Fractionated Laser Resurfacing Corrects the Inappropriate UVB Response in Geriatric Skin

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