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The Tg.AC (v-Ha-ras) Transgenic Mouse: Nature of the Model

Toxicologic pathology, 2001, Vol.29 (1_suppl), p.51-59

2001

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Tg.AC Genetically Altered Mouse: Assay Working Group Overview of Available Data

Toxicologic pathology, 2001, Vol.29 (1_suppl), p.60-80

2001

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Biological, cellular, and molecular characteristics of an inducible transgenic skin tumor model: a review

Oncogene, 2005-12, Vol.24 (56), p.8217-8228

2005

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THE USE OF TRANSGENIC ANIMALS IN CANCER TESTING

Inhalation toxicology, 1999-06, Vol.11 (6-7), p.541-544

1999

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Transformation of BALB/c-3T3 Cells: V. Transformation Responses of 168 Chemicals Compared with Mutagenicity in Salmonella and Carcinogenicity in Rodent Bioassays

Environmental health perspectives, 1993-07, Vol.101 (Suppl 2), p.347-482

1993

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Age-dependent skin tumorigenesis and transgene expression in the Tg.AC (v-Ha-ras) transgenic mouse

Carcinogenesis (New York), 2001-04, Vol.22 (4), p.651-659

2001

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Kinetics of wound-induced v-Ha-ras transgene expression and papilloma development in transgenic Tg.AC mice

Molecular carcinogenesis, 1997-09, Vol.20 (1), p.108-114

1997

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The transgenic Tg.AC mouse model for identification of chemical carcinogens

Toxicology letters, 1998-12, Vol.102-103 (1-3), p.465-471

1998

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Genetic alterations cooperate with v-Ha-ras to accelerate multistage carcinogenesis in TG.AC transgenic mouse skin

Cancer research (Chicago, Ill.), 1995-07, Vol.55 (14), p.3171-3178

1995

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Oral administration of dimethylvinyl chloride increases frequency of forestomach papillomas in Tg.AC mice

Molecular carcinogenesis, 2000-12, Vol.29 (4), p.229-235

2000

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Identifying Chemical Carcinogens and Assessing Potential Risk in Short-Term Bioassays Using Transgenic Mouse Models

Environmental health perspectives, 1995-10, Vol.103 (10), p.942-950

1995

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Transformation of BALB/c-3T3 Cells: IV. Rank-Ordered Potency of 24 Chemical Responses Detected in a Sensitive New Assay Procedure

Environmental health perspectives, 1993-07, Vol.101 (suppl 2), p.319-345

1993

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Responses of Transgenic Mouse Lines p53+/− and Tg•AC to Agents Tested in Conventional Carcinogenicity Bioassays

Toxicological sciences, 2000-02, Vol.53 (2), p.213-223

2000

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Examination of genotoxicity, toxicity and morphologic alterations in hepatocytes following in vivo or in vitro exposure to methapyrilene

Carcinogenesis (New York), 1988-06, Vol.9 (6), p.959-963

1988

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Development of a transgenic mouse model for carcinogenesis bioassays : Evaluation of chemically induced skin tumors in Tg.AC mice

Toxicological sciences, 1999-06, Vol.49 (2), p.241-254

1999

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Induction of hepatic cell proliferation and unscheduled DNA synthesis in mouse hepatocytes following in vivo treatment

Carcinogenesis (New York), 1985-10, Vol.6 (10), p.1521-1524

1985

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Chemically induced skin carcinogenesis in a transgenic mouse line (TG·AC) carrying a v-Ha-ras gene

Carcinogenesis (New York), 1993-07, Vol.14 (7), p.1335-1341

1993

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Comparative expression of novel vascular endothelial growth factor/vascular permeability factor transcripts in skin, papillomas, and carcinomas of v-Ha-ras Tg.AC transgenic mice and FVB/N mice

Biochemical and biophysical research communications, 1998-06, Vol.247 (3), p.644-653

1998

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Induction of transgene expression in Tg.AC (v-Ha-ras) transgenic mice concomitant with DNA hypomethylation

Molecular carcinogenesis, 1998-04, Vol.21 (4), p.244-250

1998

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Hemizygous Tg.AC transgenic mouse as a potential alternative to the two-year mouse carcinogenicity bioassay: evaluation of husbandry and housing factors

Journal of applied toxicology, 1998-01, Vol.18 (1), p.19-24

1998

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Measurement of unscheduled DNA synthesis and S-phase synthesis in rodent hepatocytes following in vivo treatment: Testing of 24 compounds

Environmental and molecular mutagenesis, 1989, Vol.14 (3), p.155-164

1989

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Predicting Chemical Carcinogenesis in Rodents

Environmental health perspectives, 1993-10, Vol.101 (5), p.444-445

1993

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Comparative Expression of Novel Vascular Endothelial Growth Factor/Vascular Permeability Factor Transcripts in Skin, Papillomas, and Carcinomas of v-Ha- rasTg.AC Transgenic Mice and FVB/N Mice

Biochemical and biophysical research communications, 1998-06, Vol.247 (3), p.644-653

1998

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Transformation of BALB/c-3T3 Cells: IV. Rank-Ordered Potency of 24 Chemical Responses Detected in a Sensitive New Assay Procedure

Environmental health perspectives, 1993-07, Vol.101, p.319

1993

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Transformation of BALB/c-3T3 Cells: V. Transformation Responses of 168 Chemicals Compared with Mutagenicity in Salmonella and Carcinogenicity in Rodent Bioassays

Environmental health perspectives, 1993-07, Vol.101, p.347

1993

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The Tg.AC (v-Ha-ras) Transgenic Mouse: Nature of the Model

Tg.AC Genetically Altered Mouse: Assay Working Group Overview of Available Data

Biological, cellular, and molecular characteristics of an inducible transgenic skin tumor model: a review

THE USE OF TRANSGENIC ANIMALS IN CANCER TESTING

Transformation of BALB/c-3T3 Cells: V. Transformation Responses of 168 Chemicals Compared with Mutagenicity in Salmonella and Carcinogenicity in Rodent Bioassays

Age-dependent skin tumorigenesis and transgene expression in the Tg.AC (v-Ha-ras) transgenic mouse

Kinetics of wound-induced v-Ha-ras transgene expression and papilloma development in transgenic Tg.AC mice

The transgenic Tg.AC mouse model for identification of chemical carcinogens

Genetic alterations cooperate with v-Ha-ras to accelerate multistage carcinogenesis in TG.AC transgenic mouse skin

Oral administration of dimethylvinyl chloride increases frequency of forestomach papillomas in Tg.AC mice

Identifying Chemical Carcinogens and Assessing Potential Risk in Short-Term Bioassays Using Transgenic Mouse Models

Transformation of BALB/c-3T3 Cells: IV. Rank-Ordered Potency of 24 Chemical Responses Detected in a Sensitive New Assay Procedure

Responses of Transgenic Mouse Lines p53+/− and Tg•AC to Agents Tested in Conventional Carcinogenicity Bioassays

Examination of genotoxicity, toxicity and morphologic alterations in hepatocytes following in vivo or in vitro exposure to methapyrilene

Development of a transgenic mouse model for carcinogenesis bioassays : Evaluation of chemically induced skin tumors in Tg.AC mice

Induction of hepatic cell proliferation and unscheduled DNA synthesis in mouse hepatocytes following in vivo treatment

Chemically induced skin carcinogenesis in a transgenic mouse line (TG·AC) carrying a v-Ha-ras gene

Comparative expression of novel vascular endothelial growth factor/vascular permeability factor transcripts in skin, papillomas, and carcinomas of v-Ha-ras Tg.AC transgenic mice and FVB/N mice

Induction of transgene expression in Tg.AC (v-Ha-ras) transgenic mice concomitant with DNA hypomethylation

Hemizygous Tg.AC transgenic mouse as a potential alternative to the two-year mouse carcinogenicity bioassay: evaluation of husbandry and housing factors

Measurement of unscheduled DNA synthesis and S-phase synthesis in rodent hepatocytes following in vivo treatment: Testing of 24 compounds

Predicting Chemical Carcinogenesis in Rodents

Comparative Expression of Novel Vascular Endothelial Growth Factor/Vascular Permeability Factor Transcripts in Skin, Papillomas, and Carcinomas of v-Ha- rasTg.AC Transgenic Mice and FVB/N Mice

Transformation of BALB/c-3T3 Cells: IV. Rank-Ordered Potency of 24 Chemical Responses Detected in a Sensitive New Assay Procedure

Transformation of BALB/c-3T3 Cells: V. Transformation Responses of 168 Chemicals Compared with Mutagenicity in Salmonella and Carcinogenicity in Rodent Bioassays

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