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Molecular characterization of the translocation breakpoints in the Down syndrome mouse model Ts65Dn

Mammalian genome, 2011-12, Vol.22 (11-12), p.685-691

2011

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Profiling molecular regulators of recurrence in chemorefractory triple-negative breast cancers

Breast cancer research : BCR, 2019-08, Vol.21 (1), p.87-87, Article 87

2019

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Next-generation sequencing of circulating tumor DNA to predict recurrence in triple-negative breast cancer patients with residual disease after neoadjuvant chemotherapy

NPJ breast cancer, 2017-07, Vol.3 (1), p.24-6, Article 24

2017

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A large microRNA cluster on chromosome 19 is a transcriptional hallmark of WHO type A and AB thymomas

British journal of cancer, 2016-02, Vol.114 (4), p.477-484

2016

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Dual PI3K and Wnt pathway inhibition is a synergistic combination against triple negative breast cancer

NPJ breast cancer, 2017-04, Vol.3 (1), p.17-8, Article 17

2017

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Association of Circulating Tumor DNA and Circulating Tumor Cells After Neoadjuvant Chemotherapy With Disease Recurrence in Patients With Triple-Negative Breast Cancer: Preplanned Secondary Analysis of the BRE12-158 Randomized Clinical Trial

JAMA oncology, 2020-09, Vol.6 (9), p.1410

2020

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Initial Phase I Safety Study of Gedatolisib plus Cofetuzumab Pelidotin for Patients with Metastatic Triple-Negative Breast Cancer

Clinical cancer research, 2022-08, Vol.28 (15), p.3235-3241

2022

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Commonality in Down and fetal alcohol syndromes

Birth defects research. A Clinical and molecular teratology, 2013-04, Vol.97 (4), p.187-197

2013

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Characterizing the heterogeneity of triple-negative breast cancers using microdissected normal ductal epithelium and RNA-sequencing

Breast cancer research and treatment, 2014-01, Vol.143 (1), p.57-68

2014

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Abstract P4-01-07: Insights into the compensation mechanism behind targeting PI3K and Wnt in triple-negative breast cancer

Cancer research (Chicago, Ill.), 2022-02, Vol.82 (4_Supplement), p.P4-P4-01-07

2022

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Abstract 2066: Targeting NUPR1: Response to a common compensatory pathway in triple negative breast cancer

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.2066-2066

2019

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Abstract LB-303: Transposable element insertions impact the mutational landscape of breast cancer genomes and disease onset

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.LB-303-LB-303

2019

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Abstract P3-10-12: Targeting a common drug compensation pathway using NUPR1 inhibition in triple negative breast cancer

Cancer research (Chicago, Ill.), 2020-02, Vol.80 (4_Supplement), p.P3-P3-10-12

2020

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Abstract 2525: Comprehensive analysis of thymic epithelial tumors using an integrated bioinformatics platform reveals novel biological characteristics

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.2525-2525

2019

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Erratum to: Molecular characterization of the translocation breakpoints in the Down syndrome mouse model Ts65Dn

Mammalian genome, 2011-12, Vol.22 (11-12), p.692-692

2011

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Abstract 2280: Co-detection of circulating tumor DNA and RNA in the plasma of patients with breast cancer increases the detectable number of mutated molecules

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.2280-2280

2019

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Abstract 711: Co-detection of circulating tumor DNA and RNA for enhanced detection of minimal residual disease in patients with chemorefractory triple-negative breast cancer

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16_Supplement), p.711-711

2020

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Abstract PS10-26: An initial safety study of gedatolisib plus cofetuzumab pelidotin for metastatic triple-negative breast cancer

Cancer research (Chicago, Ill.), 2021-02, Vol.81 (4_Supplement), p.PS10-26-PS10-26

2021

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Abstract 5340: Dual PI3K and Wnt pathway inhibition is a synergistic combination against triple-negative breast cancer

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.5340-5340

2015

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Abstract P4-01-16: 4-marker positive selection system for improved circulating tumor cell analysis of metastatic triple-negative breast cancer

Cancer research (Chicago, Ill.), 2020-02, Vol.80 (4_Supplement), p.P4-P4-01-16

2020

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Abstract 2029: Precision therapeutic combinations are synergistic against triple negative breast cancer using compensatory pathways

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.2029-2029

2017

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Abstract 4403: Next-generation RNA sequencing identifies UHRF1 as a key regulatory protein in triple-negative breast cancer survival

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.4403-4403

2013

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Molecular characterization of the translocation breakpoints in the Down syndrome mouse model Ts65Dn

Profiling molecular regulators of recurrence in chemorefractory triple-negative breast cancers

Next-generation sequencing of circulating tumor DNA to predict recurrence in triple-negative breast cancer patients with residual disease after neoadjuvant chemotherapy

A large microRNA cluster on chromosome 19 is a transcriptional hallmark of WHO type A and AB thymomas

Dual PI3K and Wnt pathway inhibition is a synergistic combination against triple negative breast cancer

Association of Circulating Tumor DNA and Circulating Tumor Cells After Neoadjuvant Chemotherapy With Disease Recurrence in Patients With Triple-Negative Breast Cancer: Preplanned Secondary Analysis of the BRE12-158 Randomized Clinical Trial

Initial Phase I Safety Study of Gedatolisib plus Cofetuzumab Pelidotin for Patients with Metastatic Triple-Negative Breast Cancer

BRE12-158: A Postneoadjuvant, Randomized Phase II Trial of Personalized Therapy Versus Treatment of Physician's Choice for Patients With Residual Triple-Negative Breast Cancer

Commonality in Down and fetal alcohol syndromes

Characterizing the heterogeneity of triple-negative breast cancers using microdissected normal ductal epithelium and RNA-sequencing

Abstract P4-01-07: Insights into the compensation mechanism behind targeting PI3K and Wnt in triple-negative breast cancer

Abstract 2066: Targeting NUPR1: Response to a common compensatory pathway in triple negative breast cancer

Abstract LB-303: Transposable element insertions impact the mutational landscape of breast cancer genomes and disease onset

Abstract P3-10-12: Targeting a common drug compensation pathway using NUPR1 inhibition in triple negative breast cancer

Abstract 2525: Comprehensive analysis of thymic epithelial tumors using an integrated bioinformatics platform reveals novel biological characteristics

Erratum to: Molecular characterization of the translocation breakpoints in the Down syndrome mouse model Ts65Dn

Abstract 2280: Co-detection of circulating tumor DNA and RNA in the plasma of patients with breast cancer increases the detectable number of mutated molecules

Abstract 711: Co-detection of circulating tumor DNA and RNA for enhanced detection of minimal residual disease in patients with chemorefractory triple-negative breast cancer

Abstract PS10-26: An initial safety study of gedatolisib plus cofetuzumab pelidotin for metastatic triple-negative breast cancer

Abstract 5340: Dual PI3K and Wnt pathway inhibition is a synergistic combination against triple-negative breast cancer

Abstract P4-01-16: 4-marker positive selection system for improved circulating tumor cell analysis of metastatic triple-negative breast cancer

Abstract 2029: Precision therapeutic combinations are synergistic against triple negative breast cancer using compensatory pathways

Abstract 4403: Next-generation RNA sequencing identifies UHRF1 as a key regulatory protein in triple-negative breast cancer survival

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