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Advances in understanding the state of titin truncation variants in dilated cardiomyopathy

Pflügers Archiv, 2022-03, Vol.474 (3), p.265-266

Solaro, R. John

2022

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Novel insights into sarcomere regulatory systems control of cardiac thin filament activation

The Journal of general physiology, 2021-07, Vol.153 (7), p.1

2021

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Coronary arterial vasculature in the pathophysiology of hypertrophic cardiomyopathy

Pflügers Archiv, 2019-05, Vol.471 (5), p.769-780

2019

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Ranolazine Improves Cardiac Diastolic Dysfunction Through Modulation of Myofilament Calcium Sensitivity

Circulation research, 2012-03, Vol.110 (6), p.841-850

2012

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Protein Phosphorylation and Signal Transduction in Cardiac Thin Filaments

The Journal of biological chemistry, 2011-03, Vol.286 (12), p.9935-9940

2011

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Long-Term Biased β-Arrestin Signaling Improves Cardiac Structure and Function in Dilated Cardiomyopathy

Circulation (New York, N.Y.), 2017-03, Vol.135 (11), p.1056-1070

2017

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Binding of calcium and magnesium to human cardiac troponin C

The Journal of biological chemistry, 2021-01, Vol.296, p.100350-100350, Article 100350

2021

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The Troponin C G159D Mutation Blunts Myofilament Desensitization Induced by Troponin I Ser23/24 Phosphorylation

Circulation research, 2007-05, Vol.100 (10), p.1486-1493

2007

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S-Glutathionylation of Cryptic Cysteines Enhances Titin Elasticity by Blocking Protein Folding

Cell, 2014-03, Vol.156 (6), p.1235-1246

2014

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Into the Golden Anniversary of the Yellow Journal

Journal of molecular and cellular cardiology, 2017, Vol.102, p.117-118

John Solaro, R

2017

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N-acetylcysteine reverses diastolic dysfunction and hypertrophy in familial hypertrophic cardiomyopathy

American journal of physiology. Heart and circulatory physiology, 2015-11, Vol.309 (10), p.H1720-H1730

2015

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The significance of regulatory light chain phosphorylation in cardiac physiology

Archives of biochemistry and biophysics, 2011-06, Vol.510 (2), p.129-134

2011

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Computational Studies of the Effect of the S23D/S24D Troponin I Mutation on Cardiac Troponin Structural Dynamics

Biophysical journal, 2014-10, Vol.107 (7), p.1675-1685

2014

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Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathway

Scientific reports, 2021-10, Vol.11 (1), p.21154-21154, Article 21154

2021

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Truncation of the N-terminus of cardiac troponin I initiates adaptive remodeling of the myocardial proteosome via phosphorylation of mechano-sensitive signaling pathways

Molecular and cellular biochemistry, 2022-06, Vol.477 (6), p.1803-1815

2022

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Myocardial infarction in mice alters sarcomeric function via post-translational protein modification

Molecular and cellular biochemistry, 2012-04, Vol.363 (1-2), p.203-215

2012

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Mechanisms and use of calcium-sensitizing agents in the failing heart

Circulation (New York, N.Y.), 2006-01, Vol.113 (2), p.305-315

2006

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The unique functions of cardiac troponin I in the control of cardiac muscle contraction and relaxation

Biochemical and biophysical research communications, 2008-04, Vol.369 (1), p.82-87

2008

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Increased Ca2+ Affinity of Cardiac Thin Filaments Reconstituted with Cardiomyopathy-related Mutant Cardiac Troponin I

The Journal of biological chemistry, 2006-05, Vol.281 (19), p.13471-13477

2006

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Deletion of P21-activated kinase-1 induces age-dependent increased visceral adiposity and cardiac dysfunction in female mice

Molecular and cellular biochemistry, 2021-03, Vol.476 (3), p.1337-1349

2021

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Widely cited publications of Michael Bárány in 1964 and 1967 as tipping points in understanding myosin molecular motors

Archives of biochemistry and biophysics, 2022-09, Vol.727, p.109319-109319, Article 109319

Solaro, R. John

2022

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Loss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity

Heart rhythm, 2018-08, Vol.15 (8), p.1233-1241

2018

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In Situ Time-Resolved FRET Reveals Effects of Sarcomere Length on Cardiac Thin-Filament Activation

Biophysical journal, 2014-08, Vol.107 (3), p.682-693

2014

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On ‘Comparative studies of rabit cardiac and skeletal myosins’ by M.Bárány, E.Gaetjens, K.Bárány, E.Karp

Archives of biochemistry and biophysics, 2022-09, Vol.726, p.109300-109300, Article 109300

Solaro, R. John

2022

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The β-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

American journal of physiology. Heart and circulatory physiology, 2013-09, Vol.305 (6), p.H856-H866

2013

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Advances in understanding the state of titin truncation variants in dilated cardiomyopathy

Novel insights into sarcomere regulatory systems control of cardiac thin filament activation

Coronary arterial vasculature in the pathophysiology of hypertrophic cardiomyopathy

Ranolazine Improves Cardiac Diastolic Dysfunction Through Modulation of Myofilament Calcium Sensitivity

Protein Phosphorylation and Signal Transduction in Cardiac Thin Filaments

Long-Term Biased β-Arrestin Signaling Improves Cardiac Structure and Function in Dilated Cardiomyopathy

Binding of calcium and magnesium to human cardiac troponin C

The Troponin C G159D Mutation Blunts Myofilament Desensitization Induced by Troponin I Ser23/24 Phosphorylation

S-Glutathionylation of Cryptic Cysteines Enhances Titin Elasticity by Blocking Protein Folding

Into the Golden Anniversary of the Yellow Journal

N-acetylcysteine reverses diastolic dysfunction and hypertrophy in familial hypertrophic cardiomyopathy

The significance of regulatory light chain phosphorylation in cardiac physiology

Computational Studies of the Effect of the S23D/S24D Troponin I Mutation on Cardiac Troponin Structural Dynamics

Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathway

Truncation of the N-terminus of cardiac troponin I initiates adaptive remodeling of the myocardial proteosome via phosphorylation of mechano-sensitive signaling pathways

Myocardial infarction in mice alters sarcomeric function via post-translational protein modification

Mechanisms and use of calcium-sensitizing agents in the failing heart

The unique functions of cardiac troponin I in the control of cardiac muscle contraction and relaxation

Increased Ca2+ Affinity of Cardiac Thin Filaments Reconstituted with Cardiomyopathy-related Mutant Cardiac Troponin I

Deletion of P21-activated kinase-1 induces age-dependent increased visceral adiposity and cardiac dysfunction in female mice

Widely cited publications of Michael Bárány in 1964 and 1967 as tipping points in understanding myosin molecular motors

Loss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity

In Situ Time-Resolved FRET Reveals Effects of Sarcomere Length on Cardiac Thin-Filament Activation

On ‘Comparative studies of rabit cardiac and skeletal myosins’ by M.Bárány, E.Gaetjens, K.Bárány, E.Karp

The β-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

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