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Pre‐synaptic C‐terminal truncated tau is released from cortical synapses in Alzheimer's disease

Journal of neurochemistry, 2015-05, Vol.133 (3), p.368-379

2015

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Co-Localization of Amyloid Beta and Tau Pathology in Alzheimer's Disease Synaptosomes

The American journal of pathology, 2008-06, Vol.172 (6), p.1683-1692

2008

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Intracellular Dyssynchrony of Diastolic Cytosolic [Ca2+] Decay in Ventricular Cardiomyocytes in Cardiac Remodeling and Human Heart Failure

Circulation research, 2013-08, Vol.113 (5), p.527-538

2013

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Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex

Neurobiology of disease, 2012-01, Vol.45 (1), p.381-387

2012

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Na+ -Ca2+ exchanger (NCX3) knock-out mice display an impairment in hippocampal long-term potentiation and spatial learning and memory

The Journal of neuroscience, 2011-05, Vol.31 (20), p.7312-7321

2011

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Extensive p-Tau Pathology and SDS-Stable p-Tau Oligomers in Alzheimer's Cortical Synapses

Brain pathology (Zurich, Switzerland), 2012-11, Vol.22 (6), p.826-833

2012

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Targeted Disruption of Na+/Ca2+ Exchanger 3 (NCX3) Gene Leads to a Worsening of Ischemic Brain Damage

The Journal of neuroscience, 2008-01, Vol.28 (5), p.1179-1184

2008

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The Na+/Ca2+ exchanger NCX3 mediates Ca2+ entry into matrix vesicles to facilitate initial steps of mineralization in osteoblasts

Journal of extracellular vesicles, 2024-06, Vol.13 (6), p.e12450-n/a

2024

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Prescribing cholinesterase inhibitors in mild cognitive impairment—Observations from the Alzheimer's Disease Neuroimaging Initiative

Alzheimer's & dementia : translational research & clinical interventions, 2021, Vol.7 (1), p.e12168-n/a

2021

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NCX3 knockout mice exhibit increased hippocampal CA1 and CA2 neuronal damage compared to wild-type mice following global cerebral ischemia

Experimental neurology, 2008-03, Vol.210 (1), p.268-273

2008

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Ncx3 gene ablation impairs oligodendrocyte precursor response and increases susceptibility to experimental autoimmune encephalomyelitis

Glia, 2016-07, Vol.64 (7), p.1124-1137

2016

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The Na + /Ca 2+ exchanger NCX3 mediates Ca 2+ entry into matrix vesicles to facilitate initial steps of mineralization in osteoblasts

Journal of extracellular vesicles, 2024-06, Vol.13 (6)

2024

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Silencing or knocking out the Na(+)/Ca(2+) exchanger-3 (NCX3) impairs oligodendrocyte differentiation

Cell death and differentiation, 2012-04, Vol.19 (4), p.562-572

2012

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P2‐003: ACETYLCHOLINESTERASE INHIBITOR THERAPY IN MILD COGNITIVE IMPAIRMENT: YES OR NO?

Alzheimer's & dementia, 2018-07, Vol.14 (7S_Part_12), p.P665-P666

2018

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ACETYLCHOLINESTERASE INHIBITOR THERAPY IN MILD COGNITIVE IMPAIRMENT: YES OR NO?

Alzheimer's & dementia, 2018-07, Vol.14 (7), p.P665-P666

2018

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Deleterious Effect of Butyrylcholinesterase K-Variant in Donepezil Treatment of Mild Cognitive Impairment

Journal of Alzheimer's disease, 2017-01, Vol.56 (1), p.229

2017

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AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assembly

Neurobiology of aging, 2012-08, Vol.33 (8), p.1545-1555

2012

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Metal(loid)s in Cucurbita pepo in a Uranium Mining Impacted Area in Northwestern New Mexico, USA

International journal of environmental research and public health, 2019-07, Vol.16 (14), p.2569

2019

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Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy

Human molecular genetics, 2008-08, Vol.17 (15), p.2280-2292

2008

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Presynaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's disease

Journal of neurochemistry, 2015-01, Vol.133 (3), p.368-379

2015

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P1‐052: Damaging Effect of the Butyrylcholinesterase K‐Variant in Donepezil Therapy of Mild Cognitive Impairment

Alzheimer's & dementia, 2016-07, Vol.12 (7S_Part_8), p.P421-P421

2016

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High levels of synaptosomal Na(+)-Ca(2+) exchangers (NCX1, NCX2, NCX3) co-localized with amyloid-beta in human cerebral cortex affected by Alzheimer's disease

Cell calcium (Edinburgh), 2011-04, Vol.49 (4), p.208-216

2011

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High levels of synaptosomal Na+ –Ca2+ exchangers (NCX1, NCX2, NCX3) co-localized with amyloid-beta in human cerebral cortex affected by Alzheimer's disease

Cell calcium (Edinburgh), 2011-04, Vol.49 (4), p.208-216

2011

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Heterozygous Inactivation of the Na/Ca Exchanger Increases Glucose-Induced Insulin Release, β-Cell Proliferation, and Mass

Diabetes (New York, N.Y.), 2011-08, Vol.60 (8), p.2076-2085

2011

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Pre‐synaptic C‐terminal truncated tau is released from cortical synapses in Alzheimer's disease

Co-Localization of Amyloid Beta and Tau Pathology in Alzheimer's Disease Synaptosomes

Intracellular Dyssynchrony of Diastolic Cytosolic [Ca2+] Decay in Ventricular Cardiomyocytes in Cardiac Remodeling and Human Heart Failure

Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex

Na+ -Ca2+ exchanger (NCX3) knock-out mice display an impairment in hippocampal long-term potentiation and spatial learning and memory

Extensive p-Tau Pathology and SDS-Stable p-Tau Oligomers in Alzheimer's Cortical Synapses

Targeted Disruption of Na+/Ca2+ Exchanger 3 (NCX3) Gene Leads to a Worsening of Ischemic Brain Damage

The Na+/Ca2+ exchanger NCX3 mediates Ca2+ entry into matrix vesicles to facilitate initial steps of mineralization in osteoblasts

Prescribing cholinesterase inhibitors in mild cognitive impairment—Observations from the Alzheimer's Disease Neuroimaging Initiative

NCX3 knockout mice exhibit increased hippocampal CA1 and CA2 neuronal damage compared to wild-type mice following global cerebral ischemia

Ncx3 gene ablation impairs oligodendrocyte precursor response and increases susceptibility to experimental autoimmune encephalomyelitis

The Na + /Ca 2+ exchanger NCX3 mediates Ca 2+ entry into matrix vesicles to facilitate initial steps of mineralization in osteoblasts

Silencing or knocking out the Na(+)/Ca(2+) exchanger-3 (NCX3) impairs oligodendrocyte differentiation

Sodium-calcium exchanger-3 regulates pain “wind-up”: From human psychophysics to spinal mechanisms

P2‐003: ACETYLCHOLINESTERASE INHIBITOR THERAPY IN MILD COGNITIVE IMPAIRMENT: YES OR NO?

ACETYLCHOLINESTERASE INHIBITOR THERAPY IN MILD COGNITIVE IMPAIRMENT: YES OR NO?

Deleterious Effect of Butyrylcholinesterase K-Variant in Donepezil Treatment of Mild Cognitive Impairment

AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assembly

Metal(loid)s in Cucurbita pepo in a Uranium Mining Impacted Area in Northwestern New Mexico, USA

Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy

Presynaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's disease

P1‐052: Damaging Effect of the Butyrylcholinesterase K‐Variant in Donepezil Therapy of Mild Cognitive Impairment

High levels of synaptosomal Na(+)-Ca(2+) exchangers (NCX1, NCX2, NCX3) co-localized with amyloid-beta in human cerebral cortex affected by Alzheimer's disease

High levels of synaptosomal Na+ –Ca2+ exchangers (NCX1, NCX2, NCX3) co-localized with amyloid-beta in human cerebral cortex affected by Alzheimer's disease

Heterozygous Inactivation of the Na/Ca Exchanger Increases Glucose-Induced Insulin Release, β-Cell Proliferation, and Mass

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