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Activation of microglia acidifies lysosomes and leads to degradation of Alzheimer amyloid fibrils

Molecular biology of the cell, 2007-04, Vol.18 (4), p.1490-1496

2007

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Systemic administration of tripeptidyl peptidase I in a mouse model of late infantile neuronal ceroid lipofuscinosis: effect of glycan modification

PloS one, 2012-07, Vol.7 (7), p.e40509

2012

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Crystal Structure and Autoactivation Pathway of the Precursor Form of Human Tripeptidyl-peptidase 1, the Enzyme Deficient in Late Infantile Ceroid Lipofuscinosis

The Journal of biological chemistry, 2009-02, Vol.284 (6), p.3985-3997

2009

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The Human CLN2 Protein/Tripeptidyl-Peptidase I Is a Serine Protease That Autoactivates at Acidic pH

The Journal of biological chemistry, 2001-01, Vol.276 (3), p.2249-2255

2001

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Identification and Validation of Mannose 6-Phosphate Glycoproteins in Human Plasma Reveal a Wide Range of Lysosomal and Non-lysosomal Proteins

Molecular & cellular proteomics, 2006-10, Vol.5 (10), p.1942-1956

2006

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Loss of Niemann-Pick C1 or C2 protein results in similar biochemical changes suggesting that these proteins function in a common lysosomal pathway

PloS one, 2011-08, Vol.6 (8), p.e23677

2011

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Quantitation of oxidative damage to tissue proteins

The international journal of biochemistry & cell biology, 1999-07, Vol.31 (7), p.751-757

1999

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Determination of the Substrate Specificity of Tripeptidyl-peptidase I Using Combinatorial Peptide Libraries and Development of Improved Fluorogenic Substrates

The Journal of biological chemistry, 2006-03, Vol.281 (10), p.6559-6572

2006

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Significance and prognostic value of lysosomal enzyme activities measured in surgically operated adenocarcinomas of the gastroesophageal junction and squamous cell carcinomas of the lower third of esophagus

World journal of gastroenterology : WJG, 2005-10, Vol.11 (37), p.5751

2005

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Enzyme-based diagnosis of classical late infantile neuronal ceroid lipofuscinosis: Comparison of tripeptidyl peptidase I and pepstatin-insensitive protease assays

Clinical chemistry (Baltimore, Md.), 2000-07, Vol.46 (7), p.1005-1008

2000

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Demonstration of Lysosomal Localization for the Mammalian Ependymin-related Protein Using Classical Approaches Combined with a Novel Density Shift Method

The Journal of biological chemistry, 2006-11, Vol.281 (46), p.35436-35445

2006

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Rat Brain Contains High Levels of Mannose-6-phosphorylated Glycoproteins Including Lysosomal Enzymes and Palmitoyl-Protein Thioesterase, an Enzyme Implicated in Infantile Neuronal Lipofuscinosis

The Journal of biological chemistry, 1996-08, Vol.271 (32), p.19191-19198

1996

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Metabolic changes in the lower esophageal sphincter influencing the result of anti-reflux surgical interventions in chronic gastroesophageal reflux disease

World journal of gastroenterology : WJG, 2005-03, Vol.11 (11), p.1623

2005

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Proteomic Analysis of Brain and Cerebrospinal Fluid from the Three Major Forms of Neuronal Ceroid Lipofuscinosis Reveals Potential Biomarkers

Journal of proteome research, 2017-10, Vol.16 (10), p.3787-3804

2017

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Large-volume Intrathecal Enzyme Delivery Increases Survival of a Mouse Model of Late Infantile Neuronal Ceroid Lipofuscinosis

Molecular therapy, 2011-10, Vol.19 (10), p.1842-1848

2011

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Effective Intravenous Therapy for Neurodegenerative Disease With a Therapeutic Enzyme and a Peptide That Mediates Delivery to the Brain

Molecular therapy, 2014-03, Vol.22 (3), p.547-553

2014

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Mouse mutants lacking the cation-independent mannose 6-phosphate/insulin-like growth factor II receptor are impaired in lysosomal enzyme transport: comparison of cation-independent and cation-dependent mannose 6-phosphate receptor-deficient mice

Biochemical journal, 1998-03, Vol.330 ( Pt 2) (2), p.903-908

1998

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Intrathecal tripeptidyl-peptidase 1 reduces lysosomal storage in a canine model of late infantile neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2011-11, Vol.104 (3), p.325-337

2011

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Proteomic analysis of mouse models of Niemann-Pick C disease reveals alterations in the steady-state levels of lysosomal proteins within the brain

Proteomics (Weinheim), 2012-12, Vol.12 (23-24), p.3499-3509

2012

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A mutation in the cathepsin D gene ( CTSD) in American Bulldogs with neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2006-04, Vol.87 (4), p.341-348

2006

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Subcellular Localization of Mannose 6-Phosphate Glycoproteins in Rat Brain

The Journal of biological chemistry, 1999-07, Vol.274 (30), p.21104-21113

1999

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Association of Mutations in a Lysosomal Protein with Classical Late- Infantile Neuronal Ceroid Lipofuscinosis

Science (American Association for the Advancement of Science), 1997-09, Vol.277 (5333), p.1802-1805

1997

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A frame shift mutation in canine TPP1 (the ortholog of human CLN2) in a juvenile Dachshund with neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2006-11, Vol.89 (3), p.254-260

2006

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A mutation in the ovine cathepsin D gene causes a congenital lysosomal storage disease with profound neurodegeneration

The EMBO journal, 2000-06, Vol.19 (12), p.2786-2792

2000

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The human brain mannose 6-phosphate glycoproteome: A complex mixture composed of multiple isoforms of many soluble lysosomal proteins

Proteomics (Weinheim), 2005-04, Vol.5 (6), p.1520-1532

2005

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Activation of microglia acidifies lysosomes and leads to degradation of Alzheimer amyloid fibrils

Systemic administration of tripeptidyl peptidase I in a mouse model of late infantile neuronal ceroid lipofuscinosis: effect of glycan modification

Crystal Structure and Autoactivation Pathway of the Precursor Form of Human Tripeptidyl-peptidase 1, the Enzyme Deficient in Late Infantile Ceroid Lipofuscinosis

The Human CLN2 Protein/Tripeptidyl-Peptidase I Is a Serine Protease That Autoactivates at Acidic pH

Identification and Validation of Mannose 6-Phosphate Glycoproteins in Human Plasma Reveal a Wide Range of Lysosomal and Non-lysosomal Proteins

Loss of Niemann-Pick C1 or C2 protein results in similar biochemical changes suggesting that these proteins function in a common lysosomal pathway

Quantitation of oxidative damage to tissue proteins

Determination of the Substrate Specificity of Tripeptidyl-peptidase I Using Combinatorial Peptide Libraries and Development of Improved Fluorogenic Substrates

Significance and prognostic value of lysosomal enzyme activities measured in surgically operated adenocarcinomas of the gastroesophageal junction and squamous cell carcinomas of the lower third of esophagus

Enzyme-based diagnosis of classical late infantile neuronal ceroid lipofuscinosis: Comparison of tripeptidyl peptidase I and pepstatin-insensitive protease assays

Demonstration of Lysosomal Localization for the Mammalian Ependymin-related Protein Using Classical Approaches Combined with a Novel Density Shift Method

Rat Brain Contains High Levels of Mannose-6-phosphorylated Glycoproteins Including Lysosomal Enzymes and Palmitoyl-Protein Thioesterase, an Enzyme Implicated in Infantile Neuronal Lipofuscinosis

Metabolic changes in the lower esophageal sphincter influencing the result of anti-reflux surgical interventions in chronic gastroesophageal reflux disease

Proteomic Analysis of Brain and Cerebrospinal Fluid from the Three Major Forms of Neuronal Ceroid Lipofuscinosis Reveals Potential Biomarkers

Large-volume Intrathecal Enzyme Delivery Increases Survival of a Mouse Model of Late Infantile Neuronal Ceroid Lipofuscinosis

Effective Intravenous Therapy for Neurodegenerative Disease With a Therapeutic Enzyme and a Peptide That Mediates Delivery to the Brain

Mouse mutants lacking the cation-independent mannose 6-phosphate/insulin-like growth factor II receptor are impaired in lysosomal enzyme transport: comparison of cation-independent and cation-dependent mannose 6-phosphate receptor-deficient mice

Intrathecal tripeptidyl-peptidase 1 reduces lysosomal storage in a canine model of late infantile neuronal ceroid lipofuscinosis

Proteomic analysis of mouse models of Niemann-Pick C disease reveals alterations in the steady-state levels of lysosomal proteins within the brain

A mutation in the cathepsin D gene ( CTSD) in American Bulldogs with neuronal ceroid lipofuscinosis

Subcellular Localization of Mannose 6-Phosphate Glycoproteins in Rat Brain

Association of Mutations in a Lysosomal Protein with Classical Late- Infantile Neuronal Ceroid Lipofuscinosis

A frame shift mutation in canine TPP1 (the ortholog of human CLN2) in a juvenile Dachshund with neuronal ceroid lipofuscinosis

A mutation in the ovine cathepsin D gene causes a congenital lysosomal storage disease with profound neurodegeneration

The human brain mannose 6-phosphate glycoproteome: A complex mixture composed of multiple isoforms of many soluble lysosomal proteins

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