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Insulin Therapy, Insulin Resistance and Vascular Dysfunction

Current vascular pharmacology, 2019-01, Vol.17 (5), p.429

2019

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A Hypothesis for Preeclampsia: Adenosine and Inducible Nitric Oxide Synthase in Human Placental Microvascular Endothelium

Placenta (Eastbourne), 2008-06, Vol.29 (6), p.469-483

2008

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Cross-sectional and longitudinal lipid determination studies in pregnant women reveal an association between increased maternal LDL cholesterol concentrations and reduced human umbilical vein relaxation

Placenta (Eastbourne), 2015-08, Vol.36 (8), p.895-902

2015

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Human supraphysiological gestational weight gain and fetoplacental vascular dysfunction

International Journal of Obesity, 2015-08, Vol.39 (8), p.1264-1273

2015

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Insulin recovers endothelial dysfunction requiring A2B adenosine receptor activation in human umbilical vein endothelium from late-onset preeclampsia

Placenta (Eastbourne), 2017-03, Vol.51, p.129-129

2017

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Gestational diabetes and the adenosine/ l-Arginine/nitric oxide (ALANO) pathway in human umbilical vein endothelium

Placenta (Eastbourne), 2006, Vol.27 (1), p.1-10

2006

Volltextzugriff (PDF)

Insulin receptor isoforms: an integrated view focused on gestational diabetes mellitus

Diabetes/metabolism research and reviews, 2016-05, Vol.32 (4), p.350-365

2016

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Maternal supraphysiological hypercholesterolemia leads to reduced nitric oxide synthase activity associated with reduced levels of tetrahydrobiopterin in huvec

Atherosclerosis, 2014-08, Vol.235 (2), p.e37-e37

2014

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Human maternal supraphysiological hypercholesterolemia leads to endothelial dysfunction of the placental microvasculature

Placenta (Eastbourne), 2017-03, Vol.51, p.126-126

2017

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NHE1 modulates intracellular pH and cell proliferation in human ovarian cancer

Placenta (Eastbourne), 2017-03, Vol.51, p.130-130

2017

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Review: Differential placental macrovascular and microvascular endothelial dysfunction in gestational diabetes

Placenta (Eastbourne), 2011-03, Vol.32, p.S159-S164

2011

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Human Equilibrative Nucleoside Transporters 1 and 2 may be Differentially Modulated by A2B Adenosine Receptors in Placenta Microvascular Endothelial Cells from Pre-eclampsia

Placenta (Eastbourne), 2008-09, Vol.29 (9), p.816-825

2008

Volltextzugriff (PDF)

Reck expression is induced in placentas from preeclampsia and reduces migration, invasion, and endovascular remodelling of first trimester human trophoblast

Placenta (Eastbourne), 2017-03, Vol.51, p.125-126

2017

Volltextzugriff (PDF)

Modulation of intracellular pH in human ovarian cancer

Current molecular medicine, 2016-01, Vol.16 (1), p.23

2016

Volltextzugriff (PDF)

Insulin restores l -arginine transport requiring adenosine receptors activation in umbilical vein endothelium from late-onset preeclampsia

Placenta (Eastbourne), 2015-03, Vol.36 (3), p.287-296

2015

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Prevalence of maternal supraphysiological dyslipidemia in a group of Chilean pregnant women with gestational diabetes mellitus

Placenta (Eastbourne), 2017-03, Vol.51, p.127-127

2017

Volltextzugriff (PDF)

Modulation of the placental HDL and LDL cholesterol uptake by the maternal lipids level in human trophoblast

Placenta (Eastbourne), 2017-03, Vol.51, p.128-129

2017

Volltextzugriff (PDF)

Neonatal high-density lipoprotein from pregnancies with maternal supraphysiological hypercholesterolemia impairs nitric oxide synthase activity in human umbilical vein endothelial cells

Placenta (Eastbourne), 2017-03, Vol.51, p.126-127

2017

Volltextzugriff (PDF)

Excessive gestational weight gain reduces the response to vasoactive molecules in human fetoplacental microvessels

Placenta (Eastbourne), 2017-03, Vol.51, p.111-111

2017

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Maternal supraphysiological dyslipidemia in pregnancy worse vascular response of umbilical vein rings from gestational diabetes mellitus

Placenta (Eastbourne), 2017-03, Vol.51, p.128-128

2017

Volltextzugriff (PDF)

Effect of MgSO4 on protein expression and exosomes release from human placental microvascular endothelial cells from late-onset preeclampsia

Placenta (Eastbourne), 2017-03, Vol.51, p.127-128

2017

Volltextzugriff (PDF)

Sodium/proton exchanger subtype 1 regulates intracellular pH in human umbilical vein endothelial cells from gestational diabetes mellitus

Placenta (Eastbourne), 2017-03, Vol.51, p.129-130

2017

Volltextzugriff (PDF)

Hypoxia-reduced nitric oxide synthase activity is partially explained by higher arginase-2 activity and cellular redistribution in human umbilical vein endothelium

Placenta (Eastbourne), 2011-12, Vol.32 (12), p.932-940

2011

Volltextzugriff (PDF)

Insulin therapy restores the equilibrative nucleoside transporter 1 expression in human umbilical vein endothelial cells from gestational diabetes mellitus

Placenta (Eastbourne), 2017-03, Vol.51, p.130-130

2017

Volltextzugriff (PDF)

Endoplasmic reticulum stress in human umbilical vein endothelial cells from pre-gestational maternal obesity

Placenta (Eastbourne), 2017-03, Vol.51, p.116-116

2017

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Insulin Therapy, Insulin Resistance and Vascular Dysfunction

A Hypothesis for Preeclampsia: Adenosine and Inducible Nitric Oxide Synthase in Human Placental Microvascular Endothelium

Cross-sectional and longitudinal lipid determination studies in pregnant women reveal an association between increased maternal LDL cholesterol concentrations and reduced human umbilical vein relaxation

Human supraphysiological gestational weight gain and fetoplacental vascular dysfunction

Insulin recovers endothelial dysfunction requiring A2B adenosine receptor activation in human umbilical vein endothelium from late-onset preeclampsia

Gestational diabetes and the adenosine/ l-Arginine/nitric oxide (ALANO) pathway in human umbilical vein endothelium

Insulin receptor isoforms: an integrated view focused on gestational diabetes mellitus

Maternal supraphysiological hypercholesterolemia leads to reduced nitric oxide synthase activity associated with reduced levels of tetrahydrobiopterin in huvec

Human maternal supraphysiological hypercholesterolemia leads to endothelial dysfunction of the placental microvasculature

NHE1 modulates intracellular pH and cell proliferation in human ovarian cancer

Review: Differential placental macrovascular and microvascular endothelial dysfunction in gestational diabetes

Human Equilibrative Nucleoside Transporters 1 and 2 may be Differentially Modulated by A2B Adenosine Receptors in Placenta Microvascular Endothelial Cells from Pre-eclampsia

Reck expression is induced in placentas from preeclampsia and reduces migration, invasion, and endovascular remodelling of first trimester human trophoblast

Modulation of intracellular pH in human ovarian cancer

Insulin restores l -arginine transport requiring adenosine receptors activation in umbilical vein endothelium from late-onset preeclampsia

Prevalence of maternal supraphysiological dyslipidemia in a group of Chilean pregnant women with gestational diabetes mellitus

Modulation of the placental HDL and LDL cholesterol uptake by the maternal lipids level in human trophoblast

Neonatal high-density lipoprotein from pregnancies with maternal supraphysiological hypercholesterolemia impairs nitric oxide synthase activity in human umbilical vein endothelial cells

Excessive gestational weight gain reduces the response to vasoactive molecules in human fetoplacental microvessels

Maternal supraphysiological dyslipidemia in pregnancy worse vascular response of umbilical vein rings from gestational diabetes mellitus

Effect of MgSO4 on protein expression and exosomes release from human placental microvascular endothelial cells from late-onset preeclampsia

Sodium/proton exchanger subtype 1 regulates intracellular pH in human umbilical vein endothelial cells from gestational diabetes mellitus

Hypoxia-reduced nitric oxide synthase activity is partially explained by higher arginase-2 activity and cellular redistribution in human umbilical vein endothelium

Insulin therapy restores the equilibrative nucleoside transporter 1 expression in human umbilical vein endothelial cells from gestational diabetes mellitus

Endoplasmic reticulum stress in human umbilical vein endothelial cells from pre-gestational maternal obesity

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