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Exercise stimulates beneficial adaptations to diminish doxorubicin-induced cellular toxicity

American journal of physiology. Regulatory, integrative and comparative physiology, 2019-11, Vol.317 (5), p.R662-R672

Smuder, Ashley J

2019

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Redox control of skeletal muscle atrophy

Free radical biology & medicine, 2016-09, Vol.98, p.208-217

2016

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Oxidative stress and disuse muscle atrophy: cause or consequence?

Current opinion in clinical nutrition and metabolic care, 2012-05, Vol.15 (3), p.240-245

2012

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Doxorubicin-induced oxidative stress differentially regulates proteolytic signaling in cardiac and skeletal muscle

American journal of physiology. Regulatory, integrative and comparative physiology, 2020-02, Vol.318 (2), p.R227-R233

2020

Volltextzugriff (PDF)

Doxorubicin-related effects on cardiorespiratory function and body composition

American heart journal plus, 2024-02, Vol.38, p.100360, Article 100360

Smuder, Ashley J.

2024

Volltextzugriff (PDF)

Effects of short-term endurance exercise training on acute doxorubicin-induced FoxO transcription in cardiac and skeletal muscle

Journal of applied physiology (1985), 2014-08, Vol.117 (3), p.223-230

2014

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Mechanistic links between oxidative stress and disuse muscle atrophy

Antioxidants & redox signaling, 2011-11, Vol.15 (9), p.2519

2011

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Critical Illness Myopathy Alters Diaphragm Neuromuscular Junction Protein and Gene Expression

American journal of respiratory and critical care medicine, 2023-02, Vol.207 (3), p.358-361

2023

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Crosstalk between autophagy and oxidative stress regulates proteolysis in the diaphragm during mechanical ventilation

Free radical biology & medicine, 2018-02, Vol.115, p.179-190

2018

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Global Proteome Changes in the Rat Diaphragm Induced by Endurance Exercise Training

PloS one, 2017-01, Vol.12 (1), p.e0171007-e0171007

2017

Volltextzugriff (PDF)

Targeting Mitochondria and Oxidative Stress in Cancer- and Chemotherapy-Induced Muscle Wasting

Antioxidants & redox signaling, 2023-02, Vol.38 (4-6), p.352

2023

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Partial Support Ventilation and Mitochondrial-Targeted Antioxidants Protect against Ventilator-Induced Decreases in Diaphragm Muscle Protein Synthesis

PloS one, 2015-09, Vol.10 (9), p.e0137693

2015

Volltextzugriff (PDF)

Effects of Exercise Preconditioning on Doxorubicin-Induced Liver and Kidney Toxicity in Male and Female Rats

International journal of molecular sciences, 2023-06, Vol.24 (12), p.10222

2023

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Ventilator-induced diaphragm dysfunction: cause and effect

American journal of physiology. Regulatory, integrative and comparative physiology, 2013-09, Vol.305 (5), p.R464-R477

2013

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Mitochondrial-targeted antioxidants protect skeletal muscle against immobilization-induced muscle atrophy

Journal of applied physiology (1985), 2011-11, Vol.111 (5), p.1459-1466

2011

Volltextzugriff (PDF)

Mitochondria-targeted antioxidants protect against mechanical ventilation-induced diaphragm weakness

Critical care medicine, 2011-07, Vol.39 (7), p.1749-1759

2011

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Both high level pressure support ventilation and controlled mechanical ventilation induce diaphragm dysfunction and atrophy

Critical care medicine, 2012-04, Vol.40 (4), p.1254-1260

2012

Volltextzugriff (PDF)

Cross-talk between the calpain and caspase-3 proteolytic systems in the diaphragm during prolonged mechanical ventilation

Critical care medicine, 2012-06, Vol.40 (6), p.1857-1863

2012

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Increased mitochondrial emission of reactive oxygen species and calpain activation are required for doxorubicin‐induced cardiac and skeletal muscle myopathy

The Journal of physiology, 2015-04, Vol.593 (8), p.2017-2036

2015

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Mechanisms of exercise-induced cardioprotection

Physiology (Bethesda, Md.), 2014-01, Vol.29 (1), p.27-38

2014

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Exercise Training Prevents Doxorubicin-induced Mitochondrial Dysfunction of the Liver

Medicine and science in sports and exercise, 2019-06, Vol.51 (6), p.1106-1115

2019

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Exercise protects against doxorubicin-induced oxidative stress and proteolysis in skeletal muscle

Journal of applied physiology (1985), 2011-04, Vol.110 (4), p.935-942

2011

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Effects of doxorubicin on cardiac muscle subsarcolemmal and intermyofibrillar mitochondria

Mitochondrion, 2017-05, Vol.34, p.9-19

2017

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Recovery from FOLFOX Chemotherapy-induced Systemic and Skeletal Muscle Metabolic Dysfunction in Mice

American journal of physiology: endocrinology and metabolism, 2023-08, Vol.325 (2), p.E132-E151

2023

Volltextzugriff (PDF)

Calpain and caspase-3 play required roles in immobilization-induced limb muscle atrophy

Journal of applied physiology (1985), 2013-05, Vol.114 (10), p.1482-1489

2013

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Exercise stimulates beneficial adaptations to diminish doxorubicin-induced cellular toxicity

Redox control of skeletal muscle atrophy

Oxidative stress and disuse muscle atrophy: cause or consequence?

Doxorubicin-induced oxidative stress differentially regulates proteolytic signaling in cardiac and skeletal muscle

Doxorubicin-related effects on cardiorespiratory function and body composition

Effects of short-term endurance exercise training on acute doxorubicin-induced FoxO transcription in cardiac and skeletal muscle

Mechanistic links between oxidative stress and disuse muscle atrophy

Critical Illness Myopathy Alters Diaphragm Neuromuscular Junction Protein and Gene Expression

Crosstalk between autophagy and oxidative stress regulates proteolysis in the diaphragm during mechanical ventilation

Global Proteome Changes in the Rat Diaphragm Induced by Endurance Exercise Training

Targeting Mitochondria and Oxidative Stress in Cancer- and Chemotherapy-Induced Muscle Wasting

Partial Support Ventilation and Mitochondrial-Targeted Antioxidants Protect against Ventilator-Induced Decreases in Diaphragm Muscle Protein Synthesis

Effects of Exercise Preconditioning on Doxorubicin-Induced Liver and Kidney Toxicity in Male and Female Rats

Ventilator-induced diaphragm dysfunction: cause and effect

Mitochondrial-targeted antioxidants protect skeletal muscle against immobilization-induced muscle atrophy

Mitochondria-targeted antioxidants protect against mechanical ventilation-induced diaphragm weakness

Both high level pressure support ventilation and controlled mechanical ventilation induce diaphragm dysfunction and atrophy

Cross-talk between the calpain and caspase-3 proteolytic systems in the diaphragm during prolonged mechanical ventilation

Increased mitochondrial emission of reactive oxygen species and calpain activation are required for doxorubicin‐induced cardiac and skeletal muscle myopathy

Mechanisms of exercise-induced cardioprotection

Exercise Training Prevents Doxorubicin-induced Mitochondrial Dysfunction of the Liver

Exercise protects against doxorubicin-induced oxidative stress and proteolysis in skeletal muscle

Effects of doxorubicin on cardiac muscle subsarcolemmal and intermyofibrillar mitochondria

Recovery from FOLFOX Chemotherapy-induced Systemic and Skeletal Muscle Metabolic Dysfunction in Mice

Calpain and caspase-3 play required roles in immobilization-induced limb muscle atrophy

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