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B and T Cells Driving Multiple Sclerosis: Identity, Mechanisms and Potential Triggers

Frontiers in immunology, 2020-05, Vol.11, p.760

2020

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Letter to the Editor: Vitamin D deficiency in COVID-19: Mixing up cause and consequence

Metabolism, clinical and experimental, 2021-02, Vol.115, p.154434-154434, Article 154434

2021

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Should anti-CD20 be used as an immune reconstitution therapy?

Multiple sclerosis, 2023-02, Vol.29 (2), p.308-310

2023

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Tissue-resident memory T cells populate the human brain

Nature communications, 2018-11, Vol.9 (1), p.4593-14, Article 4593

2018

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Benefits of early highly effective versus escalation treatment strategies in relapsing multiple sclerosis estimated using a treatment-sequence model

Multiple sclerosis, 2024-07, Vol.30 (8), p.1016-1025

2024

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T cell composition and polygenic multiple sclerosis risk: A population‐based study in children

European journal of neurology, 2021-11, Vol.28 (11), p.3731-3741

2021

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Vitamin D Status Is Positively Correlated with Regulatory T Cell Function in Patients with Multiple Sclerosis

PloS one, 2009-08, Vol.4 (8), p.e6635-e6635

2009

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Vitamin D related genetic polymorphisms affect serological response to high-dose vitamin D supplementation in multiple sclerosis

PloS one, 2021-12, Vol.16 (12), p.e0261097-e0261097

2021

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Editorial: Environmental factors influencing the immune functions during multiple sclerosis

Frontiers in immunology, 2023-01, Vol.14, p.1141014-1141014

2023

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Safety and T cell modulating effects of high dose vitamin D3 supplementation in multiple sclerosis

PloS one, 2010-12, Vol.5 (12), p.e15235-e15235

2010

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Pregnancy-induced effects on memory B-cell development in multiple sclerosis

Scientific reports, 2021-06, Vol.11 (1), p.12126-12126, Article 12126

2021

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GPCRomics of Homeostatic and Disease-Associated Human Microglia

Frontiers in immunology, 2021-05, Vol.12, p.674189-674189

2021

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Effects of vitamin D on the peripheral adaptive immune system: A review

Autoimmunity reviews, 2011-10, Vol.10 (12), p.733-743

2011

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Editorial: Vitamin D in Neurological Diseases: From Pathophysiology to Therapy

Frontiers in neurology, 2021-03, Vol.12, p.614900-614900

2021

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Profiling of microglia nodules in multiple sclerosis reveals propensity for lesion formation

Nature communications, 2024-02, Vol.15 (1), p.1667-1667, Article 1667

2024

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Disease activity in primary progressive multiple sclerosis: a systematic review and meta-analysis

Frontiers in neurology, 2023-11, Vol.14, p.1277477-1277477

2023

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Vitamin D and Multiple Sclerosis: Correlation, Causality, and Controversy

Autoimmune Diseases, 2011, Vol.2011 (2011), p.137-139

2011

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Exploring the effect of vitamin D3 supplementation on the anti-EBV antibody response in relapsing-remitting multiple sclerosis

Multiple sclerosis, 2018-09, Vol.24 (10), p.1280-1287

2018

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Expression of Vitamin D Receptor and Metabolizing Enzymes in Multiple Sclerosis–Affected Brain Tissue

Journal of neuropathology and experimental neurology, 2013-02, Vol.72 (2), p.91-105

2013

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Multiple sclerosis: why we should focus on both sides of the (auto)antibody

Neural regeneration research, 2021-12, Vol.16 (12), p.2422-2424

2021

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Phenotyping primary human microglia: Tight regulation of LPS responsiveness

Glia, 2012-10, Vol.60 (10), p.1506-1517

2012

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Fok-I vitamin D receptor gene polymorphism (rs10735810) and vitamin D metabolism in multiple sclerosis

Journal of neuroimmunology, 2009-02, Vol.207 (1), p.117-121

2009

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Pediatric MS patients with a primary progressive-like disease may still have relevant inflammatory activity and may benefit from regular MS treatment

Multiple sclerosis, 2020-12, Vol.26 (14), p.1962-1964

2020

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Post‐mortem multiple sclerosis lesion pathology is influenced by single nucleotide polymorphisms

Brain pathology (Zurich, Switzerland), 2020-01, Vol.30 (1), p.106-119

2020

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Vitamin D Status Does Not Affect Disability Progression of Patients with Multiple Sclerosis over Three Year Follow-Up

PloS one, 2016-06, Vol.11 (6), p.e0156122-e0156122

2016

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B and T Cells Driving Multiple Sclerosis: Identity, Mechanisms and Potential Triggers

Letter to the Editor: Vitamin D deficiency in COVID-19: Mixing up cause and consequence

Should anti-CD20 be used as an immune reconstitution therapy?

Tissue-resident memory T cells populate the human brain

Benefits of early highly effective versus escalation treatment strategies in relapsing multiple sclerosis estimated using a treatment-sequence model

T cell composition and polygenic multiple sclerosis risk: A population‐based study in children

Vitamin D Status Is Positively Correlated with Regulatory T Cell Function in Patients with Multiple Sclerosis

Vitamin D related genetic polymorphisms affect serological response to high-dose vitamin D supplementation in multiple sclerosis

Editorial: Environmental factors influencing the immune functions during multiple sclerosis

Safety and T cell modulating effects of high dose vitamin D3 supplementation in multiple sclerosis

Pregnancy-induced effects on memory B-cell development in multiple sclerosis

GPCRomics of Homeostatic and Disease-Associated Human Microglia

Effects of vitamin D on the peripheral adaptive immune system: A review

Editorial: Vitamin D in Neurological Diseases: From Pathophysiology to Therapy

Profiling of microglia nodules in multiple sclerosis reveals propensity for lesion formation

Disease activity in primary progressive multiple sclerosis: a systematic review and meta-analysis

Vitamin D and Multiple Sclerosis: Correlation, Causality, and Controversy

Exploring the effect of vitamin D3 supplementation on the anti-EBV antibody response in relapsing-remitting multiple sclerosis

Expression of Vitamin D Receptor and Metabolizing Enzymes in Multiple Sclerosis–Affected Brain Tissue

Multiple sclerosis: why we should focus on both sides of the (auto)antibody

Phenotyping primary human microglia: Tight regulation of LPS responsiveness

Fok-I vitamin D receptor gene polymorphism (rs10735810) and vitamin D metabolism in multiple sclerosis

Pediatric MS patients with a primary progressive-like disease may still have relevant inflammatory activity and may benefit from regular MS treatment

Post‐mortem multiple sclerosis lesion pathology is influenced by single nucleotide polymorphisms

Vitamin D Status Does Not Affect Disability Progression of Patients with Multiple Sclerosis over Three Year Follow-Up

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