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Msx1 loss suppresses formation of the ectopic crypts developed in the Apc-deficient small intestinal epithelium

Scientific reports, 2019-02, Vol.9 (1), p.1629-1629, Article 1629

2019

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Unique Gene Expression Signatures in the Intestinal Mucosa and Organoids Derived from Germ-Free and Monoassociated Mice

International journal of molecular sciences, 2019-03, Vol.20 (7), p.1581

2019

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Wnt Effector TCF4 Is Dispensable for Wnt Signaling in Human Cancer Cells

Genes, 2018-09, Vol.9 (9), p.439

2018

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HIC1 Expression Distinguishes Intestinal Carcinomas Sensitive to Chemotherapy

Translational oncology, 2016-04, Vol.9 (2), p.99-107

2016

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Regulation of c-Src activity by the expression of wild-type v-Src and its kinase-dead double Y416F-K295N mutant

Archives of biochemistry and biophysics, 2006-11, Vol.455 (2), p.136-143

2006

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Troy, a Tumor Necrosis Factor Receptor Family Member, Interacts With Lgr5 to Inhibit Wnt Signaling in Intestinal Stem Cells

Gastroenterology (New York, N.Y. 1943), 2013-02, Vol.144 (2), p.381-391

2013

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Monensin inhibits canonical Wnt signaling in human colorectal cancer cells and suppresses tumor growth in multiple intestinal neoplasia mice

Molecular cancer therapeutics, 2014-04, Vol.13 (4), p.812-822

2014

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Regulation of mTORC1 Signaling by Src Kinase Activity Is Akt1-Independent in RSV-Transformed Cells

Neoplasia (New York, N.Y.), 2008-02, Vol.10 (2), p.99-107

2008

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Author Correction: Msx1 loss suppresses formation of the ectopic crypts developed in the Apc-deficient small intestinal epithelium

Scientific reports, 2019-12, Vol.9 (1), p.20188-1, Article 20188

2019

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HIC1 Tumor Suppressor Loss Potentiates TLR2/NF-κB Signaling and Promotes Tissue Damage-Associated Tumorigenesis

Molecular cancer research, 2015-07, Vol.13 (7), p.1139-1148

2015

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Wnt Signaling Inhibition Deprives Small Intestinal Stem Cells of Clonogenic Capacity

Genesis (New York, N.Y. : 2000), 2016-03, Vol.54 (3), p.101-114

2016

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Characterization of four clones derived from human adenocarcinoma cell line, HT29, and analysis of their response to sodium butyrate

International journal of oncology, 2006-02, Vol.28 (2), p.559-565

2006

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Focal Adhesion Kinase Functions as an Akt Downstream Target in Migration of Colorectal Cancer Cells

Translational oncology, 2009-12, Vol.2 (4), p.281-290

2009

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Cocaine- and amphetamine-regulated transcript (CART) peptide specific binding in pheochromocytoma cells PC12

European journal of pharmacology, 2007-03, Vol.559 (2), p.109-114

2007

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W nt Signaling Inhibition Deprives Small Intestinal Stem Cells of Clonogenic Capacity

Genesis (New York, N.Y. : 2000), 2016-03, Vol.54 (3), p.101-114

2016

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Generation of two modified mouse alleles of the Hic1 tumor suppressor gene

Genesis (New York, N.Y. : 2000), 2011-03, Vol.49 (3), p.142-151

2011

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HIC1 Expression Distinguishes Intestinal Carcinomas Sensitive to Chemotherapy12

Translational oncology, 2016-03, Vol.9 (2), p.99-107

2016

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Transactivation of E-cadherin is not involved in the activity of EGF receptor in colorectal carcinoma cells

International journal of oncology, 2004-11, Vol.25 (5), p.1459

2004

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Response of HT115, a highly invasive human colorectal adenocarcinoma cell line, to sodium butyrate treatment and glucose deprivation

International journal of oncology, 2005-03, Vol.26 (3), p.793

2005

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Alterations in mitochondria function and morphology in HT29 cells upon conditions inducing differentiation and apoptosis

International journal of oncology, 2003-12, Vol.23 (6), p.1755

2003

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Expression of β-catenin is regulated by PI-3 kinase and sodium butyrate in colorectal cancer cells

International journal of molecular medicine, 2006-01, Vol.17 (1), p.69-75

2006

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IL2-dependent phosphorylation of 40S ribosomal protein S6 is controlled by PI-3K/mTOR signalling in CTLL2 cells

International journal of molecular medicine, 2004-04, Vol.13 (4), p.601

2004

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Regulation of mTORC1 Signaling by Src Kinase Activity Is Akt1-Independent in RSV-Transformed Cells1

Neoplasia (New York, N.Y.), 2008-02, Vol.10 (2), p.99-107

2008

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Focal Adhesion Kinase Functions as an Akt Downstream Target in Migration of Colorectal Cancer Cells1

Translational oncology, 2009-12, Vol.2 (4), p.281-290

2009

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Msx1 loss suppresses formation of the ectopic crypts developed in the Apc-deficient small intestinal epithelium

Unique Gene Expression Signatures in the Intestinal Mucosa and Organoids Derived from Germ-Free and Monoassociated Mice

Wnt Effector TCF4 Is Dispensable for Wnt Signaling in Human Cancer Cells

HIC1 Expression Distinguishes Intestinal Carcinomas Sensitive to Chemotherapy

TROP2 Represents a Negative Prognostic Factor in Colorectal Adenocarcinoma and Its Expression Is Associated with Features of Epithelial–Mesenchymal Transition and Invasiveness

Regulation of c-Src activity by the expression of wild-type v-Src and its kinase-dead double Y416F-K295N mutant

Troy, a Tumor Necrosis Factor Receptor Family Member, Interacts With Lgr5 to Inhibit Wnt Signaling in Intestinal Stem Cells

Monensin inhibits canonical Wnt signaling in human colorectal cancer cells and suppresses tumor growth in multiple intestinal neoplasia mice

Regulation of mTORC1 Signaling by Src Kinase Activity Is Akt1-Independent in RSV-Transformed Cells

Author Correction: Msx1 loss suppresses formation of the ectopic crypts developed in the Apc-deficient small intestinal epithelium

HIC1 Tumor Suppressor Loss Potentiates TLR2/NF-κB Signaling and Promotes Tissue Damage-Associated Tumorigenesis

Wnt Signaling Inhibition Deprives Small Intestinal Stem Cells of Clonogenic Capacity

Characterization of four clones derived from human adenocarcinoma cell line, HT29, and analysis of their response to sodium butyrate

Focal Adhesion Kinase Functions as an Akt Downstream Target in Migration of Colorectal Cancer Cells

Cocaine- and amphetamine-regulated transcript (CART) peptide specific binding in pheochromocytoma cells PC12

W nt Signaling Inhibition Deprives Small Intestinal Stem Cells of Clonogenic Capacity

Generation of two modified mouse alleles of the Hic1 tumor suppressor gene

HIC1 Expression Distinguishes Intestinal Carcinomas Sensitive to Chemotherapy12

Transactivation of E-cadherin is not involved in the activity of EGF receptor in colorectal carcinoma cells

Response of HT115, a highly invasive human colorectal adenocarcinoma cell line, to sodium butyrate treatment and glucose deprivation

Alterations in mitochondria function and morphology in HT29 cells upon conditions inducing differentiation and apoptosis

Expression of β-catenin is regulated by PI-3 kinase and sodium butyrate in colorectal cancer cells

IL2-dependent phosphorylation of 40S ribosomal protein S6 is controlled by PI-3K/mTOR signalling in CTLL2 cells

Regulation of mTORC1 Signaling by Src Kinase Activity Is Akt1-Independent in RSV-Transformed Cells1

Focal Adhesion Kinase Functions as an Akt Downstream Target in Migration of Colorectal Cancer Cells1

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