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Virus-Induced Neuronal Apoptosis Blocked by the Herpes Simplex Virus Latency-Associated Transcript

Science (American Association for the Advancement of Science), 2000-02, Vol.287 (5457), p.1500-1503

2000

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A Gene Capable of Blocking Apoptosis Can Substitute for the Herpes Simplex Virus Type 1 Latency-Associated Transcript Gene and Restore Wild-Type Reactivation Levels

Journal of Virology, 2002-02, Vol.76 (3), p.1224-1235

2002

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The Role of Interleukin (IL)-2 and IL-4 in Herpes Simplex Virus Type 1 Ocular Replication and Eye Disease

The Journal of infectious diseases, 1999-05, Vol.179 (5), p.1086-1093

1999

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The Latency-Associated Transcript Gene Enhances Establishment of Herpes Simplex Virus Type 1 Latency in Rabbits

Journal of Virology, 2000-02, Vol.74 (4), p.1885-1891

2000

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Three Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutants with Distinct and Asymmetric Effects on Virulence in Mice Compared with Rabbits

Journal of Virology, 2001-10, Vol.75 (19), p.9018-9028

2001

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The effect of latency-associated transcript on the herpes simplex virus type 1 latency-reactivation phenotype is mouse strain-dependent

Journal of general virology, 2001-05, Vol.82 (5), p.1117-1122

2001

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A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant with Increased Virulence and Reduced Spontaneous Reactivation

Journal of Virology, 1999-02, Vol.73 (2), p.920-929

1999

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Herpes Simplex Virus Type 1 Serum Neutralizing Antibody Titers Increase during Latency in Rabbits Latently Infected with Latency-Associated Transcript (LAT)-Positive but Not LAT-Negative Viruses

Journal of Virology, 1999-11, Vol.73 (11), p.9669-9672

1999

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The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency

Journal of Virology, 1994-12, Vol.68 (12), p.8045-8055

1994

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The spontaneous reactivation function of the herpes simplex virus type 1 LAT gene resides completely within the first 1.5 kilobases of the 8.3-kilobase primary transcript

Journal of Virology, 1996-02, Vol.70 (2), p.976-984

1996

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A therapeutic vaccine that reduces recurrent herpes simplex virus type 1 corneal disease

Investigative ophthalmology & visual science, 1998-06, Vol.39 (7), p.1163-1170

1998

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Activity of herpes simplex virus type 1 latency-associated transcript (LAT) promoter in neuron-derived cells: evidence for neuron specificity and for a large LAT transcript

Journal of Virology, 1990-10, Vol.64 (10), p.5019-5028

1990

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A 371-nucleotide region between the herpes simplex virus type 1 (HSV-1) LAT promoter and the 2-kilobase LAT is not essential for efficient spontaneous reactivation of latent HSV-1

Journal of Virology, 1996-03, Vol.70 (3), p.2014-2018

1996

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Therapeutic Periocular Vaccination with a Subunit Vaccine Induces Higher Levels of Herpes Simplex Virus-Specific Tear Secretory Immunoglobulin A Than Systemic Vaccination and Provides Protection against Recurrent Spontaneous Ocular Shedding of Virus in Latently Infected Rabbits

Virology (New York, N.Y.), 1998-12, Vol.252 (1), p.200-209

1998

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High-dose ocular infection with a herpes simplex virus type 1 ICP34.5 deletion mutant produces no corneal disease or neurovirulence yet results in wild-type levels of spontaneous reactivation

Journal of Virology, 1996-05, Vol.70 (5), p.2883-2893

1996

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The region of the herpes simplex virus type 1 LAT gene that is colinear with the ICP34.5 gene is not involved in spontaneous reactivation

Journal of Virology, 1996-01, Vol.70 (1), p.282-291

1996

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An avirulent ICP34.5 deletion mutant of herpes simplex virus type 1 is capable of in vivo spontaneous reactivation

Journal of Virology, 1995-05, Vol.69 (5), p.3033-3041

1995

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Insulin Suppresses Triiodothyronine-Induced Growth Hormone Secretion by GH 3 Rat Pituitary Cells

Endocrinology (Philadelphia), 1985-08, Vol.117 (2), p.532-537

1985

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Insulin suppresses triiodothyronine-induced growth hormone secretion by GH3 rat pituitary cells

Endocrinology (Philadelphia), 1985-08, Vol.117 (2), p.532-537

1985

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The enzyme specificity of acth stimulation of rabbit adrenal microsomal 17α-hydroxylase activity

Journal of steroid biochemistry, 1982, Vol.16 (1), p.93-99

1982

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ACTH-stimulated rabbit adrenal 17α-hydroxylase. Kinetic properties and a comparison with those of 3β-hydroxysteroid dehydrogenase

Journal of steroid biochemistry, 1978, Vol.9 (11), p.1033-1041

1978

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ACTH-stimulated rabbit adrenal 17α-hydroxylase. Kinetic properties and a comparison with those of 3β-hydroxysteroid dehydrogenase

Journal of steroid biochemistry, 1978-11, Vol.9 (11), p.1033-1041

1978

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2022 ESC Guidelines on cardio-oncology developed in collaboration with the European Hematology Association (EHA), the European Society for Therapeutic Radiology and Oncology (ESTRO) and the International Cardio-Oncology Society (IC-OS)

European heart journal, 2022-11, Vol.43 (41), p.4229-4361

2022

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Virus-Induced Neuronal Apoptosis Blocked by the Herpes Simplex Virus Latency-Associated Transcript

A Gene Capable of Blocking Apoptosis Can Substitute for the Herpes Simplex Virus Type 1 Latency-Associated Transcript Gene and Restore Wild-Type Reactivation Levels

The Role of Interleukin (IL)-2 and IL-4 in Herpes Simplex Virus Type 1 Ocular Replication and Eye Disease

The Latency-Associated Transcript Gene Enhances Establishment of Herpes Simplex Virus Type 1 Latency in Rabbits

Three Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutants with Distinct and Asymmetric Effects on Virulence in Mice Compared with Rabbits

The effect of latency-associated transcript on the herpes simplex virus type 1 latency-reactivation phenotype is mouse strain-dependent

A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant with Increased Virulence and Reduced Spontaneous Reactivation

Herpes Simplex Virus Type 1 Serum Neutralizing Antibody Titers Increase during Latency in Rabbits Latently Infected with Latency-Associated Transcript (LAT)-Positive but Not LAT-Negative Viruses

The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency

The spontaneous reactivation function of the herpes simplex virus type 1 LAT gene resides completely within the first 1.5 kilobases of the 8.3-kilobase primary transcript

A therapeutic vaccine that reduces recurrent herpes simplex virus type 1 corneal disease

Activity of herpes simplex virus type 1 latency-associated transcript (LAT) promoter in neuron-derived cells: evidence for neuron specificity and for a large LAT transcript

A 371-nucleotide region between the herpes simplex virus type 1 (HSV-1) LAT promoter and the 2-kilobase LAT is not essential for efficient spontaneous reactivation of latent HSV-1

Therapeutic Periocular Vaccination with a Subunit Vaccine Induces Higher Levels of Herpes Simplex Virus-Specific Tear Secretory Immunoglobulin A Than Systemic Vaccination and Provides Protection against Recurrent Spontaneous Ocular Shedding of Virus in Latently Infected Rabbits

High-dose ocular infection with a herpes simplex virus type 1 ICP34.5 deletion mutant produces no corneal disease or neurovirulence yet results in wild-type levels of spontaneous reactivation

The region of the herpes simplex virus type 1 LAT gene that is colinear with the ICP34.5 gene is not involved in spontaneous reactivation

An avirulent ICP34.5 deletion mutant of herpes simplex virus type 1 is capable of in vivo spontaneous reactivation

Insulin Suppresses Triiodothyronine-Induced Growth Hormone Secretion by GH 3 Rat Pituitary Cells

Insulin suppresses triiodothyronine-induced growth hormone secretion by GH3 rat pituitary cells

The enzyme specificity of acth stimulation of rabbit adrenal microsomal 17α-hydroxylase activity

ACTH-stimulated rabbit adrenal 17α-hydroxylase. Kinetic properties and a comparison with those of 3β-hydroxysteroid dehydrogenase

ACTH-stimulated rabbit adrenal 17α-hydroxylase. Kinetic properties and a comparison with those of 3β-hydroxysteroid dehydrogenase

2022 ESC Guidelines on cardio-oncology developed in collaboration with the European Hematology Association (EHA), the European Society for Therapeutic Radiology and Oncology (ESTRO) and the International Cardio-Oncology Society (IC-OS)

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