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Neuroprotective Actions of Dietary Choline

Nutrients, 2017-07, Vol.9 (8), p.815

2017

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Perinatal Choline Supplementation Reduces Amyloidosis and Increases Choline Acetyltransferase Expression in the Hippocampus of the APPswePS1dE9 Alzheimer's Disease Model Mice

PloS one, 2017-01, Vol.12 (1), p.e0170450-e0170450

2017

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BMP9 ameliorates amyloidosis and the cholinergic defect in a mouse model of Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (48), p.19567-19572

2013

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Zebrafish as a Genetic Model in Biological and Behavioral Gerontology: Where Development Meets Aging in Vertebrates – A Mini-Review

Gerontology (Basel), 2009-01, Vol.55 (4), p.430-441

2009

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Correction: Perinatal Choline Supplementation Reduces Amyloidosis and Increases Choline Acetyltransferase Expression in the Hippocampus of the APPswePS1dE9 Alzheimer's Disease Model Mice

PloS one, 2017-03, Vol.12 (3), p.e0174875-e0174875

2017

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Accelerated Breakdown of Phosphatidylcholine and Phosphatidylethanolamine Is a Predominant Brain Metabolic Defect in Alzheimer's Disease

Journal of Alzheimer's disease, 2023-01, Vol.93 (4), p.1285-1289

2023

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F-spondin/spon1b expression patterns in developing and adult zebrafish

PloS one, 2012-06, Vol.7 (6), p.e37593-e37593

2012

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Release of Alzheimer Amyloid Precursor Derivatives Stimulated by Activation of Muscarinic Acetylcholine Receptors

Science (American Association for the Advancement of Science), 1992-10, Vol.258 (5080), p.304-307

1992

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Bone Morphogenetic Protein 9 Induces the Transcriptome of Basal Forebrain Cholinergic Neurons

Proceedings of the National Academy of Sciences - PNAS, 2005-05, Vol.102 (19), p.6984-6989

2005

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Developmental pattern of expression of BMP receptors and Smads and activation of Smad1 and Smad5 by BMP9 in mouse basal forebrain

Brain research, 2006-05, Vol.1088 (1), p.49-56

2006

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Tyrosine Phosphorylation of Paxillin and Focal Adhesion Kinase by Activation of Muscarinic m3 Receptors is Dependent on Integrin Engagement by the Extracellular Matrix

Proceedings of the National Academy of Sciences - PNAS, 1998-06, Vol.95 (13), p.7281-7286

Slack, Barbara E.

1998

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Acceleration of phosphatidylcholine synthesis and breakdown by inhibitors of mitochondrial function in neuronal cells: a model of the membrane defect of Alzheimer's disease

The FASEB journal, 2000-11, Vol.14 (14), p.2198-2206

2000

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Adhesion-dependent redistribution of MAP kinase and MEK promotes muscarinic receptor-mediated signaling to the nucleus

Journal of cellular biochemistry, 2005-05, Vol.95 (2), p.366-378

2005

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The Extracellular Regulated Kinase-1 (ERK1) Controls Regulated α-Secretase-mediated Processing, Promoter Transactivation, and mRNA Levels of the Cellular Prion Protein

The Journal of biological chemistry, 2011-08, Vol.286 (33), p.29192-29206

2011

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A Substance P (Neurokinin-1) Receptor Mutant Carboxyl-Terminally Truncated to Resemble a Naturally Occurring Receptor Isoform Displays Enhanced Responsiveness and Resistance to Desensitization

Proceedings of the National Academy of Sciences - PNAS, 1997-08, Vol.94 (17), p.9475-9480

1997

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Differential regulation of mTOR-dependent S6 phosphorylation by muscarinic acetylcholine receptor subtypes

Journal of cellular biochemistry, 2008-08, Vol.104 (5), p.1818-1831

2008

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Collagen type I selectively activates ectodomain shedding of the discoidin domain receptor 1: Involvement of Src tyrosine kinase

Journal of cellular biochemistry, 2006-06, Vol.98 (3), p.672-684

2006

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Inhibition of dynamin-dependent endocytosis increases shedding of the amyloid precursor protein ectodomain and reduces generation of amyloid beta protein

BMC cell biology, 2005-08, Vol.6 (1), p.30, Article 30

2005

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Isoform-specific contribution of protein kinase C to prion processing

Molecular and cellular neuroscience, 2008-10, Vol.39 (3), p.400-410

2008

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Surface expression and limited proteolysis of ADAM10 are increased by a dominant negative inhibitor of dynamin

BMC cell biology, 2011-05, Vol.12 (1), p.20, Article 20

2011

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O4-06-01: APP and PRPC physiological metabolisms are both controlled by muscarinic receptors via ADAM17 phosphorylation

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T196-T196

2008

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The m3 muscarinic acetylcholine receptor is coupled to mitogen-activated protein kinase via protein kinase C and epidermal growth factor receptor kinase

Biochemical journal, 2000-06, Vol.348 (2), p.381-387

SLACK, Barbara E.

2000

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Tyrosine Phosphorylation-dependent Stimulation of Amyloid Precursor Protein Secretion by the m3 Muscarinic Acetylcholine Receptor (∗)

The Journal of biological chemistry, 1995-04, Vol.270 (14), p.8337-8344

1995

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Elevated intracellular calcium concentration increases secretory processing of the amyloid precursor protein by a tyrosine phosphorylation-dependent mechanism

Biochemical journal, 1996-12, Vol.320 ( Pt 3) (3), p.957-963

1996

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Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-alpha converting enzyme

Biochemical journal, 2001-08, Vol.357 (Pt 3), p.787-794

2001

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Neuroprotective Actions of Dietary Choline

Perinatal Choline Supplementation Reduces Amyloidosis and Increases Choline Acetyltransferase Expression in the Hippocampus of the APPswePS1dE9 Alzheimer's Disease Model Mice

BMP9 ameliorates amyloidosis and the cholinergic defect in a mouse model of Alzheimer’s disease

Zebrafish as a Genetic Model in Biological and Behavioral Gerontology: Where Development Meets Aging in Vertebrates – A Mini-Review

Correction: Perinatal Choline Supplementation Reduces Amyloidosis and Increases Choline Acetyltransferase Expression in the Hippocampus of the APPswePS1dE9 Alzheimer's Disease Model Mice

Accelerated Breakdown of Phosphatidylcholine and Phosphatidylethanolamine Is a Predominant Brain Metabolic Defect in Alzheimer's Disease

F-spondin/spon1b expression patterns in developing and adult zebrafish

Release of Alzheimer Amyloid Precursor Derivatives Stimulated by Activation of Muscarinic Acetylcholine Receptors

Bone Morphogenetic Protein 9 Induces the Transcriptome of Basal Forebrain Cholinergic Neurons

Developmental pattern of expression of BMP receptors and Smads and activation of Smad1 and Smad5 by BMP9 in mouse basal forebrain

Tyrosine Phosphorylation of Paxillin and Focal Adhesion Kinase by Activation of Muscarinic m3 Receptors is Dependent on Integrin Engagement by the Extracellular Matrix

Acceleration of phosphatidylcholine synthesis and breakdown by inhibitors of mitochondrial function in neuronal cells: a model of the membrane defect of Alzheimer's disease

Adhesion-dependent redistribution of MAP kinase and MEK promotes muscarinic receptor-mediated signaling to the nucleus

The Extracellular Regulated Kinase-1 (ERK1) Controls Regulated α-Secretase-mediated Processing, Promoter Transactivation, and mRNA Levels of the Cellular Prion Protein

A Substance P (Neurokinin-1) Receptor Mutant Carboxyl-Terminally Truncated to Resemble a Naturally Occurring Receptor Isoform Displays Enhanced Responsiveness and Resistance to Desensitization

Differential regulation of mTOR-dependent S6 phosphorylation by muscarinic acetylcholine receptor subtypes

Collagen type I selectively activates ectodomain shedding of the discoidin domain receptor 1: Involvement of Src tyrosine kinase

Inhibition of dynamin-dependent endocytosis increases shedding of the amyloid precursor protein ectodomain and reduces generation of amyloid beta protein

Isoform-specific contribution of protein kinase C to prion processing

Surface expression and limited proteolysis of ADAM10 are increased by a dominant negative inhibitor of dynamin

O4-06-01: APP and PRPC physiological metabolisms are both controlled by muscarinic receptors via ADAM17 phosphorylation

The m3 muscarinic acetylcholine receptor is coupled to mitogen-activated protein kinase via protein kinase C and epidermal growth factor receptor kinase

Tyrosine Phosphorylation-dependent Stimulation of Amyloid Precursor Protein Secretion by the m3 Muscarinic Acetylcholine Receptor (∗)

Elevated intracellular calcium concentration increases secretory processing of the amyloid precursor protein by a tyrosine phosphorylation-dependent mechanism

Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-alpha converting enzyme

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