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Glucagon-like peptide-1 (GLP-1) mediates cardioprotection by remote ischaemic conditioning

Cardiovascular research, 2016-12, Vol.112 (3), p.669-676

2016

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Arginase regulates red blood cell nitric oxide synthase and export of cardioprotective nitric oxide bioactivity

Proceedings of the National Academy of Sciences - PNAS, 2013-09, Vol.110 (37), p.15049-15054

2013

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Arginase inhibition mediates cardioprotection during ischaemia–reperfusion

Cardiovascular research, 2010-01, Vol.85 (1), p.147-154

2010

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Remote ischaemic pre‐ and delayed postconditioning – similar degree of cardioprotection but distinct mechanisms

Experimental physiology, 2012-08, Vol.97 (8), p.908-917

2012

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PPAR-alpha activation protects the type 2 diabetic myocardium against ischemia-reperfusion injury: involvement of the PI3-Kinase/Akt and NO pathway

American journal of physiology. Heart and circulatory physiology, 2009-03, Vol.296 (3), p.H719-H727

2009

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Cold Environment Exacerbates Brain Pathology and Oxidative Stress Following Traumatic Brain Injuries: Potential Therapeutic Effects of Nanowired Antioxidant Compound H-290/51

Molecular neurobiology, 2018, Vol.55 (1), p.276-285

2018

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Methamphetamine exacerbates pathophysiology of traumatic brain injury at high altitude. Neuroprotective effects of nanodelivery of a potent antioxidant compound H-290/51

Progress in brain research, 2021-01, Vol.266, p.123-193

2021

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Neuroprotective effects of insulin like growth factor-1 on engineered metal nanoparticles Ag, Cu and Al induced blood-brain barrier breakdown, edema formation, oxidative stress, upregulation of neuronal nitric oxide synthase and brain pathology

Progress in brain research, 2021-01, Vol.266, p.97-121

2021

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Reply: Glucagon-like peptide-1 mediates cardioprotection by remote ischaemic conditioning

Cardiovascular research, 2017-01, Vol.113 (1), p.13-14

2017

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Local arginase inhibition during early reperfusion mediates cardioprotection via increased nitric oxide production

PloS one, 2012-07, Vol.7 (7), p.e42038-e42038

2012

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Nanoparticles Exacerbate Both Ubiquitin and Heat Shock Protein Expressions in Spinal Cord Injury: Neuroprotective Effects of the Proteasome Inhibitor Carfilzomib and the Antioxidant Compound H-290/51

Molecular neurobiology, 2015-10, Vol.52 (2), p.882-898

2015

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Adiponectin protects against myocardial ischaemia-reperfusion injury via AMP-activated protein kinase, Akt, and nitric oxide

Cardiovascular research, 2008-04, Vol.78 (1), p.116-122

2008

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Exacerbation of Methamphetamine Neurotoxicity in Cold and Hot Environments: Neuroprotective Effects of an Antioxidant Compound H-290/51

Molecular neurobiology, 2015-10, Vol.52 (2), p.1023-1033

2015

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Arginase as a target for treatment of myocardial ischemia-reperfusion injury

European journal of pharmacology, 2013-11, Vol.720 (1-3), p.121-123

2013

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Inhibition of Rho kinase protects from ischaemia–reperfusion injury via regulation of arginase activity and nitric oxide synthase in type 1 diabetes

Diabetes & vascular disease research, 2017-05, Vol.14 (3), p.236-245

2017

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Arginase inhibition reduces infarct size via nitric oxide, protein kinase C epsilon and mitochondrial ATP-dependent K+ channels

European journal of pharmacology, 2013-07, Vol.712 (1-3), p.16-21

2013

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Cardiac-Specific Overexpression of Oxytocin Receptor Leads to Cardiomyopathy in Mice

Journal of cardiac failure, 2018-07, Vol.24 (7), p.470-478

2018

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Myocardial protection by co-administration of l -arginine and tetrahydrobiopterin during ischemia and reperfusion

International journal of cardiology, 2013-10, Vol.169 (1), p.83-88

2013

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Cardioprotective effect of rosuvastatin in vivo is dependent on inhibition of geranylgeranyl pyrophosphate and altered RhoA membrane translocation

American journal of physiology. Heart and circulatory physiology, 2007-06, Vol.292 (6), p.H3158-H3163

2007

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Pharmacological possibilities for protection against myocardial reperfusion injury

Cardiovascular research, 2002-07, Vol.55 (1), p.25-37

2002

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Diabetes exacerbates brain pathology following a focal blast brain injury: New role of a multimodal drug cerebrolysin and nanomedicine

Neuropharmacology of Neuroprotection, 2020, Vol.258, p.285

2020

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combination of l-arginine and ischaemic post-conditioning at the onset of reperfusion limits myocardial injury in the pig

Acta Physiologica, 2011-02, Vol.201 (2), p.219-226

2011

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Cardioprotection mediated by rosiglitazone, a peroxisome proliferator-activated receptor gamma ligand, in relation to nitric oxide

Basic research in cardiology, 2007-01, Vol.102 (1), p.80-89

2007

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Spinal cord injury induced heat shock protein expression is reduced by an antioxidant compound H-290/51. An experimental study using light and electron microscopy in the rat

J Neural Transm, 2006-04, Vol.113 (4), p.521-536

2006

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Arginase inhibition protects from myocardial ischemia-reperfusion injury in rats with type 1 diabetes

EUROPEAN HEART JOURNAL, 2015, Vol.36, p.526

2015

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Glucagon-like peptide-1 (GLP-1) mediates cardioprotection by remote ischaemic conditioning

Arginase regulates red blood cell nitric oxide synthase and export of cardioprotective nitric oxide bioactivity

Arginase inhibition mediates cardioprotection during ischaemia–reperfusion

Remote ischaemic pre‐ and delayed postconditioning – similar degree of cardioprotection but distinct mechanisms

PPAR-alpha activation protects the type 2 diabetic myocardium against ischemia-reperfusion injury: involvement of the PI3-Kinase/Akt and NO pathway

Cold Environment Exacerbates Brain Pathology and Oxidative Stress Following Traumatic Brain Injuries: Potential Therapeutic Effects of Nanowired Antioxidant Compound H-290/51

Methamphetamine exacerbates pathophysiology of traumatic brain injury at high altitude. Neuroprotective effects of nanodelivery of a potent antioxidant compound H-290/51

Neuroprotective effects of insulin like growth factor-1 on engineered metal nanoparticles Ag, Cu and Al induced blood-brain barrier breakdown, edema formation, oxidative stress, upregulation of neuronal nitric oxide synthase and brain pathology

Reply: Glucagon-like peptide-1 mediates cardioprotection by remote ischaemic conditioning

Local arginase inhibition during early reperfusion mediates cardioprotection via increased nitric oxide production

Nanoparticles Exacerbate Both Ubiquitin and Heat Shock Protein Expressions in Spinal Cord Injury: Neuroprotective Effects of the Proteasome Inhibitor Carfilzomib and the Antioxidant Compound H-290/51

Adiponectin protects against myocardial ischaemia-reperfusion injury via AMP-activated protein kinase, Akt, and nitric oxide

Exacerbation of Methamphetamine Neurotoxicity in Cold and Hot Environments: Neuroprotective Effects of an Antioxidant Compound H-290/51

Arginase as a target for treatment of myocardial ischemia-reperfusion injury

Inhibition of Rho kinase protects from ischaemia–reperfusion injury via regulation of arginase activity and nitric oxide synthase in type 1 diabetes

Arginase inhibition reduces infarct size via nitric oxide, protein kinase C epsilon and mitochondrial ATP-dependent K+ channels

Cardiac-Specific Overexpression of Oxytocin Receptor Leads to Cardiomyopathy in Mice

Myocardial protection by co-administration of l -arginine and tetrahydrobiopterin during ischemia and reperfusion

Cardioprotective effect of rosuvastatin in vivo is dependent on inhibition of geranylgeranyl pyrophosphate and altered RhoA membrane translocation

Pharmacological possibilities for protection against myocardial reperfusion injury

Diabetes exacerbates brain pathology following a focal blast brain injury: New role of a multimodal drug cerebrolysin and nanomedicine

combination of l-arginine and ischaemic post-conditioning at the onset of reperfusion limits myocardial injury in the pig

Cardioprotection mediated by rosiglitazone, a peroxisome proliferator-activated receptor gamma ligand, in relation to nitric oxide

Spinal cord injury induced heat shock protein expression is reduced by an antioxidant compound H-290/51. An experimental study using light and electron microscopy in the rat

Arginase inhibition protects from myocardial ischemia-reperfusion injury in rats with type 1 diabetes

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