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The Role of the Core Non-Homologous End Joining Factors in Carcinogenesis and Cancer

Cancers, 2017-07, Vol.9 (7), p.81

2017

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Carbon Ion Radiotherapy: A Review of Clinical Experiences and Preclinical Research, with an Emphasis on DNA Damage/Repair

Cancers, 2017-06, Vol.9 (6), p.66

2017

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Deficiency in Mammalian Histone H2B Ubiquitin Ligase Bre1 (Rnf20/Rnf40) Leads to Replication Stress and Chromosomal Instability

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8), p.2111-2119

2012

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Avasopasem manganese synergizes with hypofractionated radiation to ablate tumors through the generation of hydrogen peroxide

Science translational medicine, 2021-05, Vol.13 (593)

2021

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Current trends and future perspectives in hadron therapy: radiobiology

Health and technology, 2024-09, Vol.14 (5), p.867-872

2024

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Evaluation of the Response of HNSCC Cell Lines to γ-Rays and 12C Ions: Can Radioresistant Tumors Be Identified and Selected for 12C Ion Radiotherapy?

Frontiers in oncology, 2022-02, Vol.12, p.812961-812961

2022

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DNA-PK phosphorylation of RPA32 Ser4/Ser8 regulates replication stress checkpoint activation, fork restart, homologous recombination and mitotic catastrophe

DNA repair, 2014-09, Vol.21, p.131-139

2014

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The Need for Biological Countermeasures to Mitigate the Risk of Space Radiation-Induced Carcinogenesis, Cardiovascular Disease, and Central Nervous System Deficiencies

Life sciences in space research, 2022-11, Vol.35 (C), p.4-8

2022

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Breaking the limit: Biological countermeasures for space radiation exposure to enable long-duration spaceflight

Life sciences in space research, 2022-11, Vol.35, p.1-3

2022

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Radiation-Induced Reprogramming of Pre-Senescent Mammary Epithelial Cells Enriches Putative CD44(+)/CD24(-/low) Stem Cell Phenotype

Frontiers in oncology, 2016-06, Vol.6, p.138-138

2016

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Telomeres and Telomerase in the Radiation Response: Implications for Instability, Reprograming, and Carcinogenesis

Frontiers in oncology, 2015-11, Vol.5, p.257-257

2015

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Abstract C52: The radioprotector GC4419 enhances the response of PDAC tumors to high dose per fraction radiation exposure

Cancer research (Chicago, Ill.), 2019-12, Vol.79 (24_Supplement), p.C52-C52

2019

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Abstract 6284: GC4419 protects again radiation induced oral mucositis, enhances the response of squamous cell carcinoma of the head and neck tumors to ionizing radiation, and enhances radioimmune therapy

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16_Supplement), p.6284-6284

2020

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Tumor-treating fields elicit a conditional vulnerability to ionizing radiation via the downregulation of BRCA1 signaling and reduced DNA double-strand break repair capacity in non-small cell lung cancer cell lines

Cell death & disease, 2017-03, Vol.8 (3), p.e2711-e2711

2017

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Abstract B51: Examining the differential cellular response of pancreatic cancer cell lines to 12C vs. photon irradiation

Cancer research (Chicago, Ill.), 2019-12, Vol.79 (24_Supplement), p.B51-B51

2019

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Radiation-Induced Reprogramming of Pre-Senescent Mammary Epithelial Cells Enriches Putative CD44+/CD24−/low Stem Cell Phenotype

Frontiers in oncology, 2016-06, Vol.6

2016

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EXTH-24. EXPOSURE TO TUMOR TREATING FIELDS INHIBITS DNA REPAIR, INDUCES REPLICATION STRESS AND RENDERS TUMOR CELLS SENSITIVE TO AGENTS THAT IMPINGE UPON THESE PATHWAYS

Neuro-oncology (Charlottesville, Va.), 2018-11, Vol.20 (suppl_6), p.vi90-vi90

2018

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Abstract 2922: The radioprotector GC4419 enhances the response of squamous cell carcinoma of the head and neck tumors to ionizing radiation and enhances radioimmune therapy

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.2922-2922

2019

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Abstract 3939: Exploiting tumor treating fields induced downregulation of BRCA1 pathway for novel combination therapies

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.3939-3939

2019

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Abstract 3217: Newly identified role of tumor treating fields in DNA damage repair and replication stress pathways

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.3217-3217

2018

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Abstract 3296: Tumor treatment fields slow cell proliferation and enhance radiosensitivity in a model of non-small cell lung cancer

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.3296-3296

2015

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Abstract 2138: Tumor treatment fields downregulate the BRCA1/FA pathway genes leading to reduced DNA repair capacity, the inhibition of mitophagy and enhanced cell death

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.2138-2138

2017

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The Need for Biological Countermeasures to Mitigate the Risk of Space Radiation-Induced Carcinogenesis, Cardiovascular Disease, and Central Nervous System Deficiencies

Life sciences in space research, 2022-11, Vol.35 (C)

2022

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Abstract 667: The radioprotector GC4419 ameliorates radiation induced lung fibrosis while enhancing the response of non-small cell lung cancer tumors to high dose per fraction radiation exposures

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.667-667

2018

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Abstract 2929: GC4419 enhances the response of non-small cell lung carcinoma cell lines to cisplatin and cisplatin plus radiation through a ROS-mediated pathway

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.2929-2929

2018

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The Role of the Core Non-Homologous End Joining Factors in Carcinogenesis and Cancer

Carbon Ion Radiotherapy: A Review of Clinical Experiences and Preclinical Research, with an Emphasis on DNA Damage/Repair

Deficiency in Mammalian Histone H2B Ubiquitin Ligase Bre1 (Rnf20/Rnf40) Leads to Replication Stress and Chromosomal Instability

Avasopasem manganese synergizes with hypofractionated radiation to ablate tumors through the generation of hydrogen peroxide

Current trends and future perspectives in hadron therapy: radiobiology

Evaluation of the Response of HNSCC Cell Lines to γ-Rays and 12C Ions: Can Radioresistant Tumors Be Identified and Selected for 12C Ion Radiotherapy?

DNA-PK phosphorylation of RPA32 Ser4/Ser8 regulates replication stress checkpoint activation, fork restart, homologous recombination and mitotic catastrophe

The Need for Biological Countermeasures to Mitigate the Risk of Space Radiation-Induced Carcinogenesis, Cardiovascular Disease, and Central Nervous System Deficiencies

Breaking the limit: Biological countermeasures for space radiation exposure to enable long-duration spaceflight

Radiation-Induced Reprogramming of Pre-Senescent Mammary Epithelial Cells Enriches Putative CD44(+)/CD24(-/low) Stem Cell Phenotype

Telomeres and Telomerase in the Radiation Response: Implications for Instability, Reprograming, and Carcinogenesis

Abstract C52: The radioprotector GC4419 enhances the response of PDAC tumors to high dose per fraction radiation exposure

Abstract 6284: GC4419 protects again radiation induced oral mucositis, enhances the response of squamous cell carcinoma of the head and neck tumors to ionizing radiation, and enhances radioimmune therapy

Tumor-treating fields elicit a conditional vulnerability to ionizing radiation via the downregulation of BRCA1 signaling and reduced DNA double-strand break repair capacity in non-small cell lung cancer cell lines

Abstract B51: Examining the differential cellular response of pancreatic cancer cell lines to 12C vs. photon irradiation

Radiation-Induced Reprogramming of Pre-Senescent Mammary Epithelial Cells Enriches Putative CD44+/CD24−/low Stem Cell Phenotype

EXTH-24. EXPOSURE TO TUMOR TREATING FIELDS INHIBITS DNA REPAIR, INDUCES REPLICATION STRESS AND RENDERS TUMOR CELLS SENSITIVE TO AGENTS THAT IMPINGE UPON THESE PATHWAYS

Abstract 2922: The radioprotector GC4419 enhances the response of squamous cell carcinoma of the head and neck tumors to ionizing radiation and enhances radioimmune therapy

Abstract 3939: Exploiting tumor treating fields induced downregulation of BRCA1 pathway for novel combination therapies

Abstract 3217: Newly identified role of tumor treating fields in DNA damage repair and replication stress pathways

Abstract 3296: Tumor treatment fields slow cell proliferation and enhance radiosensitivity in a model of non-small cell lung cancer

Abstract 2138: Tumor treatment fields downregulate the BRCA1/FA pathway genes leading to reduced DNA repair capacity, the inhibition of mitophagy and enhanced cell death

The Need for Biological Countermeasures to Mitigate the Risk of Space Radiation-Induced Carcinogenesis, Cardiovascular Disease, and Central Nervous System Deficiencies

Abstract 667: The radioprotector GC4419 ameliorates radiation induced lung fibrosis while enhancing the response of non-small cell lung cancer tumors to high dose per fraction radiation exposures

Abstract 2929: GC4419 enhances the response of non-small cell lung carcinoma cell lines to cisplatin and cisplatin plus radiation through a ROS-mediated pathway

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