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American Thyroid Association Guide to investigating thyroid hormone economy and action in rodent and cell models

Thyroid (New York, N.Y.), 2014-01, Vol.24 (1), p.88

2014

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Cardiac Thyroid Hormone Metabolism and Heart Failure

European thyroid journal, 2017-01, Vol.6 (3), p.130-137

2017

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Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

International journal of molecular sciences, 2021-11, Vol.22 (21), p.11906

2021

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Hypoxia-inducible factor induces local thyroid hormone inactivation during hypoxic-ischemic disease in rats

The Journal of clinical investigation, 2008-03, Vol.118 (3), p.975-983

2008

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Antioxidant treatment attenuates pulmonary arterial hypertension-induced heart failure

American journal of physiology. Heart and circulatory physiology, 2010-03, Vol.298 (3), p.H1038-H1047

2010

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MicroRNA 214 Is a Potential Regulator of Thyroid Hormone Levels in the Mouse Heart Following Myocardial Infarction, by Targeting the Thyroid-Hormone-Inactivating Enzyme Deiodinase Type III

Frontiers in endocrinology (Lausanne), 2016-03, Vol.7, p.22-22

2016

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Right ventricular pacing improves right heart function in experimental pulmonary arterial hypertension: a study in the isolated heart

American journal of physiology. Heart and circulatory physiology, 2009-11, Vol.297 (5), p.H1752-H1759

2009

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Absence of Myocardial Thyroid Hormone Inactivating Deiodinase Results in Restrictive Cardiomyopathy in Mice

Molecular endocrinology (Baltimore, Md.), 2012-05, Vol.26 (5), p.809-818

2012

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Cellular and Molecular Basis of Deiodinase-Regulated Thyroid Hormone Signaling

Endocrine reviews, 2008-12, Vol.29 (7), p.898-938

2008

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Thyroid hormone as a determinant of metabolic and contractile phenotype of skeletal muscle

Thyroid (New York, N.Y.), 2008-02, Vol.18 (2), p.205

2008

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Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling

Frontiers in endocrinology (Lausanne), 2014-01, Vol.5, p.171-171

2014

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Microarray analysis reveals pivotal divergent mRNA expression profiles early in the development of either compensated ventricular hypertrophy or heart failure

Physiological genomics, 2005-05, Vol.21 (3), p.314-323

2005

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Induction of Thyroid Hormone-Degrading Deiodinase in Cardiac Hypertrophy and Failure

Endocrinology (Philadelphia), 2002-07, Vol.143 (7), p.2812-2815

2002

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Left-Ventricular Remodeling After Myocardial Infarction Is Associated with a Cardiomyocyte-Specific Hypothyroid Condition

Endocrinology (Philadelphia), 2011-02, Vol.152 (2), p.669-679

2011

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H9c2 cardiomyoblasts produce thyroid hormone

American Journal of Physiology: Cell Physiology, 2008-05, Vol.294 (5), p.C1227-C1233

2008

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Right-ventricular failure is associated with increased mitochondrial complex II activity and production of reactive oxygen species

Cardiovascular research, 2007-09, Vol.75 (4), p.770-781

2007

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A Global Loss of Dio2 Leads to Unexpected Changes in Function and Fiber Types of Slow Skeletal Muscle in Male Mice

Endocrinology (Philadelphia), 2019-05, Vol.160 (5), p.1205-1222

2019

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Cellular and Molecular Basis of Deiodinase-Regulated Thyroid Hormone Signaling1

Endocrine reviews, 2008-12, Vol.29 (7), p.898-938

2008

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The Cardioprotective Effect of Sevoflurane Depends on Protein Kinase C Activation, Opening of Mitochondrial K+ATP Channels, and the Production of Reactive Oxygen Species

Anesthesia and analgesia, 2003-11, Vol.97 (5), p.1370-1376

2003

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Cardiomyocyte-specific inactivation of thyroid hormone in pathologic ventricular hypertrophy: an adaptative response or part of the problem?

Heart failure reviews, 2010-03, Vol.15 (2), p.133-142

2010

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Distinct myocardial effects of beta-blocker therapy in heart failure with normal and reduced left ventricular ejection fraction

European heart journal, 2009-08, Vol.30 (15), p.1863-1872

2009

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Constitutive cardiac overexpression of sarcoplasmic/endoplasmic reticulum Ca2+-ATPase delays myocardial failure after myocardial infarction in rats at a cost of increased acute arrhythmias

Circulation (New York, N.Y.), 2004-04, Vol.109 (15), p.1898-1903

2004

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Evidence for neuroinflammation after myocardial infarction in a mouse model

Heart and mind (Mumbai, India), 2017-10, Vol.1 (4), p.134-140

2017

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Thyroid hormones and skeletal muscle--new insights and potential implications

Nature reviews. Endocrinology, 2014-04, Vol.10 (4), p.206

2014

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RhoA-ROCK signaling is involved in contraction-mediated inhibition of SERCA2a expression in cardiomyocytes

Pflügers Archiv, 2009-08, Vol.458 (4), p.785-793

2009

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American Thyroid Association Guide to investigating thyroid hormone economy and action in rodent and cell models

Cardiac Thyroid Hormone Metabolism and Heart Failure

Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

Hypoxia-inducible factor induces local thyroid hormone inactivation during hypoxic-ischemic disease in rats

Antioxidant treatment attenuates pulmonary arterial hypertension-induced heart failure

MicroRNA 214 Is a Potential Regulator of Thyroid Hormone Levels in the Mouse Heart Following Myocardial Infarction, by Targeting the Thyroid-Hormone-Inactivating Enzyme Deiodinase Type III

Right ventricular pacing improves right heart function in experimental pulmonary arterial hypertension: a study in the isolated heart

Absence of Myocardial Thyroid Hormone Inactivating Deiodinase Results in Restrictive Cardiomyopathy in Mice

Cellular and Molecular Basis of Deiodinase-Regulated Thyroid Hormone Signaling

Thyroid hormone as a determinant of metabolic and contractile phenotype of skeletal muscle

Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling

Microarray analysis reveals pivotal divergent mRNA expression profiles early in the development of either compensated ventricular hypertrophy or heart failure

Induction of Thyroid Hormone-Degrading Deiodinase in Cardiac Hypertrophy and Failure

Left-Ventricular Remodeling After Myocardial Infarction Is Associated with a Cardiomyocyte-Specific Hypothyroid Condition

H9c2 cardiomyoblasts produce thyroid hormone

Right-ventricular failure is associated with increased mitochondrial complex II activity and production of reactive oxygen species

A Global Loss of Dio2 Leads to Unexpected Changes in Function and Fiber Types of Slow Skeletal Muscle in Male Mice

Cellular and Molecular Basis of Deiodinase-Regulated Thyroid Hormone Signaling1

The Cardioprotective Effect of Sevoflurane Depends on Protein Kinase C Activation, Opening of Mitochondrial K+ATP Channels, and the Production of Reactive Oxygen Species

Cardiomyocyte-specific inactivation of thyroid hormone in pathologic ventricular hypertrophy: an adaptative response or part of the problem?

Distinct myocardial effects of beta-blocker therapy in heart failure with normal and reduced left ventricular ejection fraction

Constitutive cardiac overexpression of sarcoplasmic/endoplasmic reticulum Ca2+-ATPase delays myocardial failure after myocardial infarction in rats at a cost of increased acute arrhythmias

Evidence for neuroinflammation after myocardial infarction in a mouse model

Thyroid hormones and skeletal muscle--new insights and potential implications

RhoA-ROCK signaling is involved in contraction-mediated inhibition of SERCA2a expression in cardiomyocytes

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