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Crucial role for human Toll-like receptor 4 in the development of contact allergy to nickel

Nature immunology, 2010-09, Vol.11 (9), p.814

2010

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Antigen affinity and antigen dose exert distinct influences on CD4 T-cell differentiation

Proceedings of the National Academy of Sciences - PNAS, 2014-10, Vol.111 (41), p.14852-14857

2014

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Activation of murine macrophages via TLR2 and TLR4 is negatively regulated by a Lyn/PI3K module and promoted by SHIP1

The Journal of immunology (1950), 2010-05, Vol.184 (10), p.5809-5818

2010

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Cholinergic Signaling Attenuates Pro-Inflammatory Interleukin-8 Response in Colonic Epithelial Cells

Frontiers in immunology, 2022-01, Vol.12, p.781147-781147

2022

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Lipopolysaccharide sensing an important factor in the innate immune response to Gram-negative bacterial infections: Benefits and hazards of LPS hypersensitivity

Immunobiology (1979), 2008-01, Vol.213 (3), p.193-203

2008

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Key Role of Splenic Myeloid DCs in the IFN-αβ Response to Adenoviruses In Vivo

PLoS pathogens, 2008-11, Vol.4 (11), p.e1000208

2008

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Water permeability is a measure of severity in acute appendicitis

Journal of enzyme inhibition and medicinal chemistry, 2017-01, Vol.32 (1), p.1036-1041

2017

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CD14 is required for MyD88-independent LPS signaling

Nature immunology, 2005-06, Vol.6 (6), p.565-570

2005

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Key role of splenic myeloid DCs in the IFN-alphabeta response to adenoviruses in vivo

PLoS pathogens, 2008-11, Vol.4 (11), p.e1000208

2008

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R‐form LPS, the master key to the activation ofTLR4/MD‐2‐positive cells

European Journal of Immunology, 2006-03, Vol.36 (3), p.701-711

2006

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Key Role of Splenic Myeloid DCs in the IFN- alpha beta Response to Adenoviruses In Vivo

PLoS pathogens, 2008-11, Vol.4 (11), p.e1000208-e1000208

2008

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Gain of function in the immune system caused by a ryanodine receptor 1 mutation

Journal of cell science, 2013-08, Vol.126 (Pt 15), p.3485-3492

2013

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A minimum number of autoimmune T cells to induce autoimmunity?

Cellular immunology, 2017-06, Vol.316, p.21-31

2017

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Lack of Mucosal Cholinergic Innervation Is Associated With Increased Risk of Enterocolitis in Hirschsprung’s Disease

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.12 (2), p.507-545

2021

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Absence of TRIF signaling in lipopolysaccharide-stimulated murine mast cells

The Journal of immunology (1950), 2011-05, Vol.186 (9), p.5478-5488

2011

Volltextzugriff (PDF)

Lack of Mucosal Cholinergic Innervation Is Associated With Increased Risk of Enterocolitis in Hirschsprung’s DiseaseSummary

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.12 (2), p.507-545

2021

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Increased regulatory T cells in pediatric acute appendicitis

Pediatric allergy and immunology, 2018-02, Vol.29 (1), p.104-108

2018

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Bruton's tyrosine kinase is dispensable for the Toll-like receptor-mediated activation of mast cells

Cellular signalling, 2009, Vol.21 (1), p.79-86

2009

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A Loss of Function Mutation of Presenilin-2 Interferes with Amyloid β-Peptide Production and Notch Signaling

The Journal of biological chemistry, 1999-10, Vol.274 (40), p.28669-28673

1999

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Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch

Nature cell biology, 2000-04, Vol.2 (4), p.205-211

2000

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Key Role of Splenic Myeloid DCs in the IFN-[alpha][beta] Response to Adenoviruses In Vivo: e1000208

PLoS pathogens, 2008-11, Vol.4 (11)

2008

Volltextzugriff (PDF)

Key Role of Splenic Myeloid DCs in the IFN-[alpha][beta] Response to Adenoviruses In Vivo

PLoS pathogens, 2008-11, Vol.4 (11)

2008

Volltextzugriff (PDF)

Presenilin-1 differentially facilitates endoproteolysis of the -amyloid precursor protein and Notch

Nature cell biology, 2000-04, Vol.2 (4), p.205-211

2000

Volltextzugriff (PDF)

Presenilin-1 differentially facilitates endoproteolysis of the b-amyloid precursor protein and Notch

Nature cell biology, 2000-04, Vol.2 (4), p.205-211

2000

Volltextzugriff (PDF)

A Loss of Function Mutation of Presenilin-2 Interferes with Amyloid Î²-Peptide Production and Notch Signaling

The Journal of biological chemistry, 1999-10, Vol.274 (40), p.28669

1999

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Crucial role for human Toll-like receptor 4 in the development of contact allergy to nickel

Antigen affinity and antigen dose exert distinct influences on CD4 T-cell differentiation

Activation of murine macrophages via TLR2 and TLR4 is negatively regulated by a Lyn/PI3K module and promoted by SHIP1

Cholinergic Signaling Attenuates Pro-Inflammatory Interleukin-8 Response in Colonic Epithelial Cells

Lipopolysaccharide sensing an important factor in the innate immune response to Gram-negative bacterial infections: Benefits and hazards of LPS hypersensitivity

Key Role of Splenic Myeloid DCs in the IFN-αβ Response to Adenoviruses In Vivo

Water permeability is a measure of severity in acute appendicitis

CD14 is required for MyD88-independent LPS signaling

Key role of splenic myeloid DCs in the IFN-alphabeta response to adenoviruses in vivo

R‐form LPS, the master key to the activation ofTLR4/MD‐2‐positive cells

Key Role of Splenic Myeloid DCs in the IFN- alpha beta Response to Adenoviruses In Vivo

Gain of function in the immune system caused by a ryanodine receptor 1 mutation

A minimum number of autoimmune T cells to induce autoimmunity?

Lack of Mucosal Cholinergic Innervation Is Associated With Increased Risk of Enterocolitis in Hirschsprung’s Disease

Absence of TRIF signaling in lipopolysaccharide-stimulated murine mast cells

Lack of Mucosal Cholinergic Innervation Is Associated With Increased Risk of Enterocolitis in Hirschsprung’s DiseaseSummary

Increased regulatory T cells in pediatric acute appendicitis

Bruton's tyrosine kinase is dispensable for the Toll-like receptor-mediated activation of mast cells

A Loss of Function Mutation of Presenilin-2 Interferes with Amyloid β-Peptide Production and Notch Signaling

Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch

Key Role of Splenic Myeloid DCs in the IFN-[alpha][beta] Response to Adenoviruses In Vivo: e1000208

Key Role of Splenic Myeloid DCs in the IFN-[alpha][beta] Response to Adenoviruses In Vivo

Presenilin-1 differentially facilitates endoproteolysis of the -amyloid precursor protein and Notch

Presenilin-1 differentially facilitates endoproteolysis of the b-amyloid precursor protein and Notch

A Loss of Function Mutation of Presenilin-2 Interferes with Amyloid Î²-Peptide Production and Notch Signaling

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