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TNFR1 and TNFR2 in the Control of the Life and Death Balance of Macrophages

Frontiers in cell and developmental biology, 2019-05, Vol.7, p.91-91

2019

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Cell death‐independent activities of the death receptors CD95, TRAILR1, and TRAILR2

The FEBS journal, 2017-04, Vol.284 (8), p.1131-1159

2017

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TNF Receptor Associated Factor 2 (TRAF2) Signaling in Cancer

Cancers, 2022-08, Vol.14 (16), p.4055

2022

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Activation of TNFR2 sensitizes macrophages for TNFR1-mediated necroptosis

Cell death & disease, 2016-09, Vol.7 (9), p.e2375-e2375

2016

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Fn14 and TNFR2 as regulators of cytotoxic TNFR1 signaling

Frontiers in cell and developmental biology, 2023-11, Vol.11, p.1267837-1267837

2023

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Membrane Tumor Necrosis Factor (TNF) Induces p100 Processing via TNF Receptor-2 (TNFR2)

The Journal of biological chemistry, 2010-03, Vol.285 (10), p.7394-7404

2010

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Fibroblast Growth Factor Inducible (Fn14)-specific Antibodies Concomitantly Display Signaling Pathway-specific Agonistic and Antagonistic Activity

The Journal of biological chemistry, 2013-05, Vol.288 (19), p.13455-13466

2013

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NF-κB Inducers Upregulate cFLIP, a Cycloheximide-Sensitive Inhibitor of Death Receptor Signaling

Molecular and Cellular Biology, 2001-06, Vol.21 (12), p.3964-3973

2001

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TRAF2 Controls Death Receptor-Induced Caspase-8 Processing and Facilitates Proinflammatory Signaling

Frontiers in immunology, 2019-08, Vol.10, p.2024-2024

2019

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TNF and TNF receptors as therapeutic targets for rheumatic diseases and beyond

Nature reviews. Rheumatology, 2023-09, Vol.19 (9), p.576-591

2023

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Membrane lymphotoxin-α2β is a novel tumor necrosis factor (TNF) receptor 2 (TNFR2) agonist

Cell death & disease, 2021-04, Vol.12 (4), p.360-360, Article 360

2021

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Cellular FLICE-inhibitory Protein (cFLIP) Isoforms Block CD95- and TRAIL Death Receptor-induced Gene Induction Irrespective of Processing of Caspase-8 or cFLIP in the Death-inducing Signaling Complex

The Journal of biological chemistry, 2011-05, Vol.286 (19), p.16631-16646

2011

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The IKK inhibitor Bay 11-7082 induces cell death independent from inhibition of activation of NFκB transcription factors

PloS one, 2013-03, Vol.8 (3), p.e59292

2013

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SMAC mimetic BV6 induces cell death in monocytes and maturation of monocyte-derived dendritic cells

PloS one, 2011-06, Vol.6 (6), p.e21556-e21556

2011

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TWEAK inhibits TRAF2-mediated CD40 signaling by destabilization of CD40 signaling complexes

The Journal of immunology (1950), 2013-09, Vol.191 (5), p.2308-2318

2013

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Implication of TNF-Related Apoptosis-Inducing Ligand in Inflammatory Intestinal Epithelial Lesions

Gastroenterology (New York, N.Y. 1943), 2006-06, Vol.130 (7), p.1962-1974

2006

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Death Receptor-Induced Signaling Pathways Are Differentially Regulated by Gamma Interferon Upstream of Caspase 8 Processing

Molecular and Cellular Biology, 2005-08, Vol.25 (15), p.6363-6379

2005

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TNFR2 unlocks a RIPK1 kinase activity-dependent mode of proinflammatory TNFR1 signaling

Cell death & disease, 2018-09, Vol.9 (9), p.921-11, Article 921

2018

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Redundant and receptor-specific activities of TRADD, RIPK1 and FADD in death receptor signaling

Cell death & disease, 2019-02, Vol.10 (2), p.122-122, Article 122

2019

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Cell death‐independent activities of the death receptors CD 95, TRAILR 1, and TRAILR 2

The FEBS journal, 2017-04, Vol.284 (8), p.1131-1159

2017

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Biallelic RIPK1 mutations in humans cause severe immunodeficiency, arthritis, and intestinal inflammation

Science (American Association for the Advancement of Science), 2018-08, Vol.361 (6404), p.810-813

2018

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Fas-associated Death Domain Protein (FADD) and Caspase-8 Mediate Up-regulation of c-Fos by Fas Ligand and Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL) via a FLICE Inhibitory Protein (FLIP)-regulated Pathway

The Journal of biological chemistry, 2001-08, Vol.276 (35), p.32585-32590

2001

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Genetically engineered IgG1 and nanobody oligomers acquire strong intrinsic CD40 agonism

Bioengineered, 2024-12, Vol.15 (1), p.2302246-2302246

2024

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MLN4924 sensitizes monocytes and maturing dendritic cells for TNF‐dependent and ‐independent necroptosis

British journal of pharmacology, 2015-03, Vol.172 (5), p.1222-1236

2015

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Hypertonicity-enforced BCL-2 addiction unleashes the cytotoxic potential of death receptors

Oncogene, 2018-07, Vol.37 (30), p.4122-4136

2018

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TNFR1 and TNFR2 in the Control of the Life and Death Balance of Macrophages

Cell death‐independent activities of the death receptors CD95, TRAILR1, and TRAILR2

TNF Receptor Associated Factor 2 (TRAF2) Signaling in Cancer

Activation of TNFR2 sensitizes macrophages for TNFR1-mediated necroptosis

Fn14 and TNFR2 as regulators of cytotoxic TNFR1 signaling

Membrane Tumor Necrosis Factor (TNF) Induces p100 Processing via TNF Receptor-2 (TNFR2)

Fibroblast Growth Factor Inducible (Fn14)-specific Antibodies Concomitantly Display Signaling Pathway-specific Agonistic and Antagonistic Activity

NF-κB Inducers Upregulate cFLIP, a Cycloheximide-Sensitive Inhibitor of Death Receptor Signaling

TRAF2 Controls Death Receptor-Induced Caspase-8 Processing and Facilitates Proinflammatory Signaling

TNF and TNF receptors as therapeutic targets for rheumatic diseases and beyond

Membrane lymphotoxin-α2β is a novel tumor necrosis factor (TNF) receptor 2 (TNFR2) agonist

Cellular FLICE-inhibitory Protein (cFLIP) Isoforms Block CD95- and TRAIL Death Receptor-induced Gene Induction Irrespective of Processing of Caspase-8 or cFLIP in the Death-inducing Signaling Complex

The IKK inhibitor Bay 11-7082 induces cell death independent from inhibition of activation of NFκB transcription factors

SMAC mimetic BV6 induces cell death in monocytes and maturation of monocyte-derived dendritic cells

TWEAK inhibits TRAF2-mediated CD40 signaling by destabilization of CD40 signaling complexes

Implication of TNF-Related Apoptosis-Inducing Ligand in Inflammatory Intestinal Epithelial Lesions

Death Receptor-Induced Signaling Pathways Are Differentially Regulated by Gamma Interferon Upstream of Caspase 8 Processing

TNFR2 unlocks a RIPK1 kinase activity-dependent mode of proinflammatory TNFR1 signaling

Redundant and receptor-specific activities of TRADD, RIPK1 and FADD in death receptor signaling

Cell death‐independent activities of the death receptors CD 95, TRAILR 1, and TRAILR 2

Biallelic RIPK1 mutations in humans cause severe immunodeficiency, arthritis, and intestinal inflammation

Fas-associated Death Domain Protein (FADD) and Caspase-8 Mediate Up-regulation of c-Fos by Fas Ligand and Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL) via a FLICE Inhibitory Protein (FLIP)-regulated Pathway

Genetically engineered IgG1 and nanobody oligomers acquire strong intrinsic CD40 agonism

MLN4924 sensitizes monocytes and maturing dendritic cells for TNF‐dependent and ‐independent necroptosis

Hypertonicity-enforced BCL-2 addiction unleashes the cytotoxic potential of death receptors

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