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Mitofusin 1 and 2 regulation of mitochondrial DNA content is a critical determinant of glucose homeostasis

Nature communications, 2022-04, Vol.13 (1), p.2340-2340, Article 2340

2022

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Clarifying the function of genes at the chromosome 16p13 locus in type 1 diabetes: CLEC16A and DEXI

Genes and immunity, 2020-02, Vol.21 (2), p.79-82

2020

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NSC23766, a Known Inhibitor of Tiam1-Rac1 Signaling Module, Prevents the Onset of Type 1 Diabetes in the NOD Mouse Model

Cellular physiology and biochemistry, 2016-01, Vol.39 (2), p.760-767

2016

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Mitophagy protects β cells from inflammatory damage in diabetes

JCI insight, 2020-12, Vol.5 (24)

2020

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VAV2, a guanine nucleotide exchange factor for Rac1, regulates glucose-stimulated insulin secretion in pancreatic beta cells

Diabetologia, 2015-11, Vol.58 (11), p.2573-2581

2015

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52-OR: Mitophagy Forms a Protective Response to Inflammatory Damage in Pancreatic ß-Cells

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Exposure to chronic hyperglycemic conditions results in Ras-related C3 botulinum toxin substrate 1 (Rac1)-mediated activation of p53 and ATM kinase in pancreatic β-cells

Apoptosis (London), 2017-05, Vol.22 (5), p.597-607

2017

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The Regulatory Roles of Mitogen-Activated Protein Kinase (MAPK) Pathways in Health and Diabetes: Lessons Learned from the Pancreatic β-Cell

Recent patents on endocrine, metabolic & immune drug discovery, 2017-01, Vol.10 (2), p.76

2017

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Exposure to chronic hyperglycemic conditions results in Ras-related C3 botulinum toxin substrate 1 -mediated activation of p53 and ATM kinase in pancreatic [beta]-cells

Apoptosis (London), 2017-05, Vol.22 (5), p.597

2017

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37-OR: Mitofusins 1 and 2 Collaborate to Fuel Pancreatic Beta-Cell Insulin Release via Regulation of Both Mitochondrial Structure and DNA Content

Diabetes (New York, N.Y.), 2021-06, Vol.70 (Supplement_1)

2021

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Mitochondrial metabolism and dynamics in pancreatic beta cell glucose sensing

Biochemical journal, 2023-06, Vol.480 (11), p.773-789

2023

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Complementary Approaches to Interrogate Mitophagy Flux in Pancreatic β-Cells

Journal of visualized experiments, 2023-09 (199)

2023

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Phagocyte-like NADPH oxidase (Nox2) promotes activation of p38MAPK in pancreatic β-cells under glucotoxic conditions: Evidence for a requisite role of Ras-related C3 botulinum toxin substrate 1 (Rac1)

Biochemical pharmacology, 2015-06, Vol.95 (4), p.301-310

2015

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EHT 1864, a small molecule inhibitor of Ras-related C3 botulinum toxin substrate 1 (Rac1), attenuates glucose-stimulated insulin secretion in pancreatic β-cells

Cellular signalling, 2015-06, Vol.27 (6), p.1159-1167

2015

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Glucotoxic and diabetic conditions induce caspase 6-mediated degradation of nuclear lamin A in human islets, rodent islets and INS-1 832/13 cells

Apoptosis (London), 2014-12, Vol.19 (12), p.1691-1701

2014

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Cell Cre Expression and Reduced Ins1 Gene Dosage Protect Mice From Type 1 Diabetes

Endocrinology (Philadelphia), 2022-11, Vol.163 (11), p.1

2022

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Proinsulin folding and trafficking defects trigger a common pathological disturbance of endoplasmic reticulum homeostasis

Protein science, 2024-04, Vol.33 (4), p.e4949-n/a

2024

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Effects of Pro-Inflammatory Cytokines on the Pancreatic Islet Transcriptome: A View at Both Temporal and Single-Cell Resolution

Function (Oxford, England), 2022, Vol.3 (3), p.zqac026-zqac026

2022

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Reciprocal regulatory balance within the CLEC16A–RNF41 mitophagy complex depends on an intrinsically disordered protein region

The Journal of biological chemistry, 2023-04, Vol.299 (4), p.103057-103057, Article 103057

2023

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β-Cell Cre Expression and Reduced Ins1 Gene Dosage Protect Mice From Type 1 Diabetes

Endocrinology (Philadelphia), 2022-10, Vol.163 (11)

2022

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An intrinsically disordered protein region encoded by the human disease gene CLEC16A regulates mitophagy

Autophagy, 2023-02, Vol.19 (2), p.525-543

2023

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Phagocyte-Like NADPH Oxidase [Nox2] Promotes Activation of P38MAPK in Pancreatic Beta-Cells under Glucotoxic Conditions: Evidence for a Novel Role of Rac1

Free radical biology & medicine, 2014-11, Vol.76, p.S113

2014

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262 - Phagocyte-Like NADPH Oxidase [Nox2] Promotes Activation of P38MAPK in Pancreatic Beta-Cells under Glucotoxic Conditions: Evidence for a Novel Role of Rac1

Free radical biology & medicine, 2014-11, Vol.76, p.S113-S113

2014

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Mechanisms of pancreatic beta cell dysfunction in diabetes

Sidarala, Vaibhav

2016

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Mitofusin 1 and 2 regulation of mitochondrial DNA content is a critical determinant of glucose homeostasis

Clarifying the function of genes at the chromosome 16p13 locus in type 1 diabetes: CLEC16A and DEXI

NSC23766, a Known Inhibitor of Tiam1-Rac1 Signaling Module, Prevents the Onset of Type 1 Diabetes in the NOD Mouse Model

Mitophagy protects β cells from inflammatory damage in diabetes

VAV2, a guanine nucleotide exchange factor for Rac1, regulates glucose-stimulated insulin secretion in pancreatic beta cells

52-OR: Mitophagy Forms a Protective Response to Inflammatory Damage in Pancreatic ß-Cells

Exposure to chronic hyperglycemic conditions results in Ras-related C3 botulinum toxin substrate 1 (Rac1)-mediated activation of p53 and ATM kinase in pancreatic β-cells

The Regulatory Roles of Mitogen-Activated Protein Kinase (MAPK) Pathways in Health and Diabetes: Lessons Learned from the Pancreatic β-Cell

Exposure to chronic hyperglycemic conditions results in Ras-related C3 botulinum toxin substrate 1 -mediated activation of p53 and ATM kinase in pancreatic [beta]-cells

37-OR: Mitofusins 1 and 2 Collaborate to Fuel Pancreatic Beta-Cell Insulin Release via Regulation of Both Mitochondrial Structure and DNA Content

Mitochondrial metabolism and dynamics in pancreatic beta cell glucose sensing

Complementary Approaches to Interrogate Mitophagy Flux in Pancreatic β-Cells

Phagocyte-like NADPH oxidase (Nox2) promotes activation of p38MAPK in pancreatic β-cells under glucotoxic conditions: Evidence for a requisite role of Ras-related C3 botulinum toxin substrate 1 (Rac1)

EHT 1864, a small molecule inhibitor of Ras-related C3 botulinum toxin substrate 1 (Rac1), attenuates glucose-stimulated insulin secretion in pancreatic β-cells

Glucotoxic and diabetic conditions induce caspase 6-mediated degradation of nuclear lamin A in human islets, rodent islets and INS-1 832/13 cells

Cell Cre Expression and Reduced Ins1 Gene Dosage Protect Mice From Type 1 Diabetes

Proinsulin folding and trafficking defects trigger a common pathological disturbance of endoplasmic reticulum homeostasis

Effects of Pro-Inflammatory Cytokines on the Pancreatic Islet Transcriptome: A View at Both Temporal and Single-Cell Resolution

Reciprocal regulatory balance within the CLEC16A–RNF41 mitophagy complex depends on an intrinsically disordered protein region

β-Cell Cre Expression and Reduced Ins1 Gene Dosage Protect Mice From Type 1 Diabetes

An intrinsically disordered protein region encoded by the human disease gene CLEC16A regulates mitophagy

Phagocyte-Like NADPH Oxidase [Nox2] Promotes Activation of P38MAPK in Pancreatic Beta-Cells under Glucotoxic Conditions: Evidence for a Novel Role of Rac1

262 - Phagocyte-Like NADPH Oxidase [Nox2] Promotes Activation of P38MAPK in Pancreatic Beta-Cells under Glucotoxic Conditions: Evidence for a Novel Role of Rac1

Mechanisms of pancreatic beta cell dysfunction in diabetes

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