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Down's syndrome suppression of tumour growth and the role of the calcineurin inhibitor DSCR1

Nature (London), 2009-06, Vol.459 (7250), p.1126-1130

2009

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Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome

Neuropharmacology, 2005-07, Vol.49 (1), p.122-128

2005

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Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Journal of neurochemistry, 2006-08, Vol.98 (4), p.1266-1277

2006

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Increased synaptic depression in the Ts65Dn mouse, a model for mental retardation in Down syndrome

Neuropharmacology, 1999-12, Vol.38 (12), p.1917-1920

1999

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Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: A model of Down syndrome

Journal of comparative neurology (1911), 2006-02, Vol.494 (5), p.815-833

2006

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Speeding of miniature excitatory post-synaptic currents in Ts65Dn cultured hippocampal neurons

Neuroscience letters, 2008-06, Vol.438 (3), p.356-361

2008

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Elevated expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in cerebellar unipolar brush cells of a Down syndrome mouse model

Cellular and molecular neurobiology, 2006-07, Vol.26 (4-6), p.719-732

2006

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Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABA B -Induced Potassium Current

Journal of neurophysiology, 2007-01, Vol.97 (1), p.892-900

2007

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Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABAB-Induced Potassium Current

Journal of neurophysiology, 2007-01, Vol.97 (1), p.892-900

2007

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Trisomy for the Down syndrome ‘critical region’ is necessary but not sufficient for brain phenotypes of trisomic mice

Human molecular genetics, 2007-04, Vol.16 (7), p.774-782

2007

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On the cause of mental retardation in Down syndrome: extrapolation from full and segmental trisomy 16 mouse models

Brain Research Reviews, 2001-04, Vol.35 (2), p.115-145

2001

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Increased expression of NR2A subunit does not alter NMDA‐evoked responses in cultured fetal trisomy 16 mouse hippocampal neurons

Journal of neurochemistry, 2001-03, Vol.76 (6), p.1663-1669

2001

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Ts65Dn mouse, a Down syndrome model, exhibits elevated myo-Inositol in selected brain regions and peripheral tissues

Neurochemical research, 2000-04, Vol.25 (4), p.431-435

2000

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GABAB–GIRK2-Mediated Signaling in Down Syndrome

Advances in Pharmacology, 2010, Vol.58, p.397-426

2010

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Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABA sub(B)-Induced Potassium Current

Journal of neurophysiology, 2007-01, Vol.97 (1), p.892-900

2007

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ERRATUM: Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Journal of neurochemistry, 2006-11, Vol.99 (4), p.1320

2006

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Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Journal of neurochemistry, 2006-11, Vol.99 (4), p.1320-1320

2006

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Down syndrome suppression of tumor growth and the role of the calcineurin inhibitor DSCR1

Nature (London), 2009-05, Vol.459 (7250), p.1126-1130

2009

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Abnormal Expression of the GIRK2 Potassium Channel in Hippocampus, Frontal Cortex and Substantia Nigra of Ts65Dn Mouse: A Model of Down Syndrome

Journal of comparative neurology (1911), 2006-02, Vol.494 (5), p.815-833

2006

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The role of 5-HT2A receptor antagonism in amphetamine-induced inhibition of A10 dopamine neurons in vitro

European journal of pharmacology, 2005-09, Vol.520 (1-3), p.77-85

2005

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The role of 5-HT(2A) receptor antagonism in amphetamine-induced inhibition of A10 dopamine neurons in vitro

European journal of pharmacology, 2005-09, Vol.520 (1-3), p.77-85

2005

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Serotoninergic modulation of excitability in area CA1 of the in vitro rat hippocampus

Neuroscience letters, 1995-10, Vol.199 (3), p.211-214

1995

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Potentiation of synaptic reflexes by D-serine in the rat spinal cord in vitro

European journal of pharmacology, 1991-03, Vol.195 (2), p.241-244

1991

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Down Syndrome
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Down's syndrome suppression of tumour growth and the role of the calcineurin inhibitor DSCR1

Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome

Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Increased synaptic depression in the Ts65Dn mouse, a model for mental retardation in Down syndrome

Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: A model of Down syndrome

Speeding of miniature excitatory post-synaptic currents in Ts65Dn cultured hippocampal neurons

Elevated expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in cerebellar unipolar brush cells of a Down syndrome mouse model

Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABA B -Induced Potassium Current

Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABAB-Induced Potassium Current

Trisomy for the Down syndrome ‘critical region’ is necessary but not sufficient for brain phenotypes of trisomic mice

On the cause of mental retardation in Down syndrome: extrapolation from full and segmental trisomy 16 mouse models

Increased expression of NR2A subunit does not alter NMDA‐evoked responses in cultured fetal trisomy 16 mouse hippocampal neurons

Ts65Dn mouse, a Down syndrome model, exhibits elevated myo-Inositol in selected brain regions and peripheral tissues

GABAB–GIRK2-Mediated Signaling in Down Syndrome

Ts65Dn, a Mouse Model of Down Syndrome, Exhibits Increased GABA sub(B)-Induced Potassium Current

ERRATUM: Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Down syndrome suppression of tumor growth and the role of the calcineurin inhibitor DSCR1

Abnormal Expression of the GIRK2 Potassium Channel in Hippocampus, Frontal Cortex and Substantia Nigra of Ts65Dn Mouse: A Model of Down Syndrome

The role of 5-HT2A receptor antagonism in amphetamine-induced inhibition of A10 dopamine neurons in vitro

The role of 5-HT(2A) receptor antagonism in amphetamine-induced inhibition of A10 dopamine neurons in vitro

Serotoninergic modulation of excitability in area CA1 of the in vitro rat hippocampus

Potentiation of synaptic reflexes by D-serine in the rat spinal cord in vitro

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