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Drug Discovery Strategies for SARS-CoV-2

The Journal of pharmacology and experimental therapeutics, 2020-10, Vol.375 (1), p.127-138

2020

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Drug combination therapy for emerging viral diseases

Drug discovery today, 2021-10, Vol.26 (10), p.2367-2376

2021

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SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes through mitigation of oxidative and ER stress

PloS one, 2022-02, Vol.17 (2), p.e0258054

2022

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Therapeutic development approaches to treat haploinsufficiency diseases: restoring protein levels

Drug discovery today, 2024-10, p.104201, Article 104201

2024

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318-OR: Intermittent Fasting Improves ß-cell Function in Nonobese KATP-Induced Neonatal Diabetes through Reduction of Cellular Stress and Enhanced Autophagy

Diabetes (New York, N.Y.), 2022-06, Vol.71 (Supplement_1)

2022

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Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes

Diabetes (New York, N.Y.), 2022-06, Vol.71 (6), p.1233-1245

2022

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The role of membrane excitability in pancreatic β-cell glucotoxicity

Scientific reports, 2019-05, Vol.9 (1), p.6952-6952, Article 6952

2019

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1789-P: Inactivation of Acyl-Protein Thioesterase 1, a Major Mediator of Palmitoylation Cycling, Promotes Insulin Secretion and ß-Cell Failure

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Alterations in pancreatic islet cell function in response to small bowel resection

American journal of physiology: Gastrointestinal and liver physiology, 2020-07, Vol.319 (1), p.G36-G42

2020

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High-fat-diet-induced remission of diabetes in a subset of K ATP -GOF insulin-secretory-deficient mice

Diabetes, obesity & metabolism, 2018-11, Vol.20 (11), p.2574-2584

2018

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High‐fat‐diet‐induced remission of diabetes in a subset of KATP‐GOF insulin‐secretory‐deficient mice

Diabetes, obesity & metabolism, 2018-11, Vol.20 (11), p.2574-2584

2018

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Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

Physiological reports, 2019-06, Vol.7 (11), p.e14101-n/a

2019

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Viral Proteases as Targets for Coronavirus Disease 2019 Drug Development

The Journal of pharmacology and experimental therapeutics, 2021-08, Vol.378 (2), p.166-172

2021

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SGLT2 inhibitors therapy protects glucotoxicity-induced [beta]-cell failure in a mouse model of human K.sub.ATP-induced diabetes through mitigation of oxidative and ER stress

PloS one, 2022-02, Vol.17 (2), p.e0258054

2022

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334-LB: Dapagliflozin, a SGLT2 Inhibitor, Therapy Improves ß-Cell Function in Neonatal Diabetes by Reducing Oxidative and ER Stress, and Loss of ß-Cell Identity

Diabetes (New York, N.Y.), 2019-06, Vol.68 (Supplement_1)

2019

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Mechanisms Underlying the Progressive Loss of Beta-Cell Function and Mass in Neonatal Diabetes Are Reverted by Dapagliflozin Therapy

Diabetes (New York, N.Y.), 2018-07, Vol.67 (Supplement_1)

2018

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Palmitoylation couples insulin hypersecretion with β cell failure in diabetes

Cell metabolism, 2023-02, Vol.35 (2), p.332-344.e7

2023

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Beta-Cell Glucotoxicity in KATP-Induced Diabetes Is Alleviated by Genetic Reduction of Glucose Metabolism

Diabetes (New York, N.Y.), 2018-07, Vol.67 (Supplement_1)

2018

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SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes through mitigation of oxidative and ER stress

PloS one, 2022-01, Vol.17 (2), p.e0258054

2022

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Drug Discovery Strategies for SARS-CoV-2

Drug combination therapy for emerging viral diseases

SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes through mitigation of oxidative and ER stress

Therapeutic development approaches to treat haploinsufficiency diseases: restoring protein levels

318-OR: Intermittent Fasting Improves ß-cell Function in Nonobese KATP-Induced Neonatal Diabetes through Reduction of Cellular Stress and Enhanced Autophagy

Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes

The role of membrane excitability in pancreatic β-cell glucotoxicity

1789-P: Inactivation of Acyl-Protein Thioesterase 1, a Major Mediator of Palmitoylation Cycling, Promotes Insulin Secretion and ß-Cell Failure

Alterations in pancreatic islet cell function in response to small bowel resection

High-fat-diet-induced remission of diabetes in a subset of K ATP -GOF insulin-secretory-deficient mice

High‐fat‐diet‐induced remission of diabetes in a subset of KATP‐GOF insulin‐secretory‐deficient mice

Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

Viral Proteases as Targets for Coronavirus Disease 2019 Drug Development

SGLT2 inhibitors therapy protects glucotoxicity-induced [beta]-cell failure in a mouse model of human K.sub.ATP-induced diabetes through mitigation of oxidative and ER stress

334-LB: Dapagliflozin, a SGLT2 Inhibitor, Therapy Improves ß-Cell Function in Neonatal Diabetes by Reducing Oxidative and ER Stress, and Loss of ß-Cell Identity

Mechanisms Underlying the Progressive Loss of Beta-Cell Function and Mass in Neonatal Diabetes Are Reverted by Dapagliflozin Therapy

Palmitoylation couples insulin hypersecretion with β cell failure in diabetes

Beta-Cell Glucotoxicity in KATP-Induced Diabetes Is Alleviated by Genetic Reduction of Glucose Metabolism

SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes through mitigation of oxidative and ER stress

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