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Activity of the Bcl-2 Family Inhibitor ABT-263 in a Panel of Small Cell Lung Cancer Xenograft Models

Clinical cancer research, 2008-06, Vol.14 (11), p.3268-3277

2008

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ABT-263 and rapamycin act cooperatively to kill lymphoma cells in vitro and in vivo

Molecular cancer therapeutics, 2008-10, Vol.7 (10), p.3265-3274

2008

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Hypoxia-Inducible Factor-1 Inhibition in Combination with Temozolomide Treatment Exhibits Robust Antitumor Efficacy In vivo

Clinical cancer research, 2006-08, Vol.12 (15), p.4747-4754

2006

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The HPK1 Inhibitor A‑745 Verifies the Potential of Modulating T Cell Kinase Signaling for Immunotherapy

ACS chemical biology, 2022-03, Vol.17 (3), p.556-566

2022

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Tumor Regionality in the Mouse Intestine Reflects the Mechanism of Loss of Apc Function

Proceedings of the National Academy of Sciences - PNAS, 2004-06, Vol.101 (26), p.9769-9773

2004

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A small-molecule inhibitor of Bcl-XL potentiates the activity of cytotoxic drugs in vitro and in vivo

Cancer research (Chicago, Ill.), 2006-09, Vol.66 (17), p.8731-8739

2006

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PARP inhibitors attenuate chemotherapy-induced painful neuropathy

Journal of the peripheral nervous system, 2012-09, Vol.17 (3), p.324-330

2012

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Studies of neoplasia in the Min mouse

Biochimica et biophysica acta, 1997-04, Vol.1332 (2), p.F25-F48

1997

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N-ethyl-N-nitrosourea treatment of multiple intestinal neoplasia (Min) mice : age-related effects on the formation of intestinal adenomas : cystic crypts, and epidermoid cysts

Cancer research (Chicago, Ill.), 1995-10, Vol.55 (19), p.4479-4485

1995

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Somatic mutational mechanisms involved in intestinal tumor formation in Min mice

Cancer research (Chicago, Ill.), 1997-05, Vol.57 (10), p.1999-2006

1997

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A Resistant Genetic Background Leading to Incomplete Penetrance of Intestinal Neoplasia and Reduced Loss of Heterozygosity in ApcMin/+ Mice

Proceedings of the National Academy of Sciences - PNAS, 1998-09, Vol.95 (18), p.10826-10831

1998

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Manipulation of the mouse germline in the study ofMin-induced neoplasia

Seminars in cancer biology, 1996-10, Vol.7 (5), p.249-260

1996

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Mlh1 deficiency enhances several phenotypes of Apc(Min)/+ mice

Oncogene, 2000-05, Vol.19 (23), p.2774-2779

2000

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Homozygosity for the Minallele of Apc results in disruption of mouse development prior to gastrulation

Developmental dynamics, 1995-08, Vol.203 (4), p.422-433

1995

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Homozygosity for the Min allele of Apc results in disruption of mouse development prior to gastrulation

Developmental dynamics, 1995-08, Vol.203 (4), p.422-433

1995

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The Bcl-2 Family Inhibitor ABT-263 Shows Significant Anti-Tumor Efficacy in Models of B Cell Non-Hodgkin's Lymphoma

Blood, 2006-11, Vol.108 (11), p.825-825

2006

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Manipulation of the mouse germline in the study of Min-induced neoplasia

Seminars in cancer biology, 1996-10, Vol.7 (5), p.249-260

1996

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Mlh1 deficiency enhances several phenotypes of ApcMin/+ mice

Oncogene, 2000-05, Vol.19 (23), p.2774-2779

2000

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Retinoic acid nuclear receptors and tumor promotion: decreased expression of retinoic acid nuclear receptors by the tumor promoter 12-O-tetradecanoylphorbol-13-acetate

Carcinogenesis (New York), 1994-04, Vol.15 (4), p.701-705

1994

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A resistant genetic background leading to incomplete penetrance of intestinal neoplasia and reduced loss of heterozygosity in Apc Min /+ mice

Proceedings of the National Academy of Sciences - PNAS, 1998-09, Vol.95 (18), p.10826-10831

1998

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Studies of neoplasia in the Min mouse1C57BL/6J Min/+ mice are available worldwide from The Jackson Laboratory, Bar Harbor, ME 04609, USA.1

Biochimica et biophysica acta. Reviews on cancer, 1997-04, Vol.1332 (2), p.F25-F48

1997

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Intestinal tumorigenesis in Apc(Min) mice: Developmental and genetic factors that regulate tumor formation

Shoemaker, Alex R

1997

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ProQuest

Prevalence and risk factors of Rift Valley fever in humans and animals from Kabale district in Southwestern Uganda, 2016

PLoS neglected tropical diseases, 2018-05, Vol.12 (5), p.e0006412-e0006412

2018

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Impact of enhanced viral haemorrhagic fever surveillance on outbreak detection and response in Uganda

The Lancet infectious diseases, 2018-04, Vol.18 (4), p.373-375

2018

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Activity of the Bcl-2 Family Inhibitor ABT-263 in a Panel of Small Cell Lung Cancer Xenograft Models

ABT-263 and rapamycin act cooperatively to kill lymphoma cells in vitro and in vivo

Hypoxia-Inducible Factor-1 Inhibition in Combination with Temozolomide Treatment Exhibits Robust Antitumor Efficacy In vivo

The SUV4-20 inhibitor A-196 verifies a role for epigenetics in genomic integrity

The HPK1 Inhibitor A‑745 Verifies the Potential of Modulating T Cell Kinase Signaling for Immunotherapy

Tumor Regionality in the Mouse Intestine Reflects the Mechanism of Loss of Apc Function

A small-molecule inhibitor of Bcl-XL potentiates the activity of cytotoxic drugs in vitro and in vivo

PARP inhibitors attenuate chemotherapy-induced painful neuropathy

Studies of neoplasia in the Min mouse

N-ethyl-N-nitrosourea treatment of multiple intestinal neoplasia (Min) mice : age-related effects on the formation of intestinal adenomas : cystic crypts, and epidermoid cysts

Somatic mutational mechanisms involved in intestinal tumor formation in Min mice

A Resistant Genetic Background Leading to Incomplete Penetrance of Intestinal Neoplasia and Reduced Loss of Heterozygosity in ApcMin/+ Mice

Manipulation of the mouse germline in the study ofMin-induced neoplasia

Mlh1 deficiency enhances several phenotypes of Apc(Min)/+ mice

Homozygosity for the Minallele of Apc results in disruption of mouse development prior to gastrulation

Homozygosity for the Min allele of Apc results in disruption of mouse development prior to gastrulation

The Bcl-2 Family Inhibitor ABT-263 Shows Significant Anti-Tumor Efficacy in Models of B Cell Non-Hodgkin's Lymphoma

Manipulation of the mouse germline in the study of Min-induced neoplasia

Mlh1 deficiency enhances several phenotypes of ApcMin/+ mice

Retinoic acid nuclear receptors and tumor promotion: decreased expression of retinoic acid nuclear receptors by the tumor promoter 12-O-tetradecanoylphorbol-13-acetate

A resistant genetic background leading to incomplete penetrance of intestinal neoplasia and reduced loss of heterozygosity in Apc Min /+ mice

Studies of neoplasia in the Min mouse1C57BL/6J Min/+ mice are available worldwide from The Jackson Laboratory, Bar Harbor, ME 04609, USA.1

Intestinal tumorigenesis in Apc(Min) mice: Developmental and genetic factors that regulate tumor formation

Prevalence and risk factors of Rift Valley fever in humans and animals from Kabale district in Southwestern Uganda, 2016

Impact of enhanced viral haemorrhagic fever surveillance on outbreak detection and response in Uganda

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