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Fibroblasts in myocardial infarction: A role in inflammation and repair

Journal of molecular and cellular cardiology, 2014-05, Vol.70, p.74-82

2014

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Smad7 effects on TGF-β and ErbB2 restrain myofibroblast activation and protect from postinfarction heart failure

The Journal of clinical investigation, 2022-02, Vol.132 (3), p.1-19

2022

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Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart

Journal of molecular and cellular cardiology, 2019-07, Vol.132, p.84-97

2019

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Tissue transglutaminase in the pathogenesis of heart failure

Cell death and differentiation, 2018-03, Vol.25 (3), p.453-456

2018

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The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling

Biochimica et biophysica acta. Molecular basis of disease, 2017-01, Vol.1863 (1), p.298-309

2017

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The α4β1 integrin and the EDA domain of fibronectin regulate a profibrotic phenotype in dermal fibroblasts

Matrix biology, 2015-01, Vol.41, p.26-35

2015

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Validation of diagnostic criteria and histopathological characterization of cardiac rupture in the mouse model of nonreperfused myocardial infarction

American journal of physiology. Heart and circulatory physiology, 2020-11, Vol.319 (5), p.H948-H964

2020

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Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

Circulation (New York, N.Y.), 2018-02, Vol.137 (7), p.707-724

2018

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Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

Circulation research, 2019-04, Vol.124 (8), p.1214-1227

2019

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Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

The American journal of pathology, 2016-05, Vol.186 (5), p.1114-1127

2016

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Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium

Journal of molecular and cellular cardiology, 2022-10, Vol.171, p.1-15

2022

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Collagen denaturation in the infarcted myocardium involves temporally distinct effects of MT1-MMP-dependent proteolysis and mechanical tension

Matrix biology, 2021-05, Vol.99, p.18-42

2021

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Left atrial remodeling, hypertrophy and fibrosis in mouse models of heart failure

Cardiovascular pathology, 2017-09, Vol.30, p.27-37

2017

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Fibroblast Smad7 Induction Protects the Remodeling Pressure-Overloaded Heart

Circulation research, 2024-07, Vol.135 (3), p.453-469

2024

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Store-Independent Orai1/3 Channels Activated by Intracrine LeukotrieneC4: Role in Neointimal Hyperplasia

Circulation research, 2013-03, Vol.112 (7), p.1013-1025

2013

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Pharmacologic inhibition of the enzymatic effects of tissue transglutaminase reduces cardiac fibrosis and attenuates cardiomyocyte hypertrophy following pressure overload

Journal of molecular and cellular cardiology, 2018-04, Vol.117, p.36-48

2018

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Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling

Cardiovascular research, 2017-07, Vol.113 (8), p.892-905

2017

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Mechanisms of Fibroblast Activation in the Remodeling Myocardium

Current pathobiology reports, 2017-06, Vol.5 (2), p.145-152

2017

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The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction

Journal of molecular and cellular cardiology, 2015-12, Vol.89 (Pt B), p.223-231

2015

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STIM1 controls endothelial barrier function independently of Orai1 and Ca2+ entry

Science signaling, 2013-03, Vol.6 (267), p.ra18

2013

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Mediators Secreted by Myeloid Cells May Protect and Repair the Infarcted Myocardium

Circulation research, 2015-06, Vol.117 (1), p.10-12

2015

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The group A streptococcal collagen‐like protein‐1, Scl1, mediates biofilm formation by targeting the extra domain A‐containing variant of cellular fibronectin expressed in wounded tissue

Molecular microbiology, 2013-02, Vol.87 (3), p.672-689

2013

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Identification of the Peptide Sequences within the EIIIA (EDA) Segment of Fibronectin That Mediate Integrin α9β1-dependent Cellular Activities

The Journal of biological chemistry, 2008-02, Vol.283 (5), p.2858-2870

2008

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Identification of the Peptide Sequences within the EIIIA (EDA) Segment of Fibronectin That Mediate Integrin alpha 9 beta 1-dependent Cellular Activities

The Journal of biological chemistry, 2008-02, Vol.283 (5), p.2858-2870

2008

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Abstract 395: The Role of Transglutaminase 2 (tg2) in Regulation of Macrophage Phenotype

Circulation research, 2020-07, Vol.127 (Suppl_1)

2020

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Fibroblasts in myocardial infarction: A role in inflammation and repair

Smad7 effects on TGF-β and ErbB2 restrain myofibroblast activation and protect from postinfarction heart failure

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart

Tissue transglutaminase in the pathogenesis of heart failure

The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling

The α4β1 integrin and the EDA domain of fibronectin regulate a profibrotic phenotype in dermal fibroblasts

Validation of diagnostic criteria and histopathological characterization of cardiac rupture in the mouse model of nonreperfused myocardial infarction

Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium

Collagen denaturation in the infarcted myocardium involves temporally distinct effects of MT1-MMP-dependent proteolysis and mechanical tension

Left atrial remodeling, hypertrophy and fibrosis in mouse models of heart failure

Fibroblast Smad7 Induction Protects the Remodeling Pressure-Overloaded Heart

Store-Independent Orai1/3 Channels Activated by Intracrine LeukotrieneC4: Role in Neointimal Hyperplasia

Pharmacologic inhibition of the enzymatic effects of tissue transglutaminase reduces cardiac fibrosis and attenuates cardiomyocyte hypertrophy following pressure overload

Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling

Mechanisms of Fibroblast Activation in the Remodeling Myocardium

The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction

STIM1 controls endothelial barrier function independently of Orai1 and Ca2+ entry

Mediators Secreted by Myeloid Cells May Protect and Repair the Infarcted Myocardium

The group A streptococcal collagen‐like protein‐1, Scl1, mediates biofilm formation by targeting the extra domain A‐containing variant of cellular fibronectin expressed in wounded tissue

Identification of the Peptide Sequences within the EIIIA (EDA) Segment of Fibronectin That Mediate Integrin α9β1-dependent Cellular Activities

Identification of the Peptide Sequences within the EIIIA (EDA) Segment of Fibronectin That Mediate Integrin alpha 9 beta 1-dependent Cellular Activities

Abstract 395: The Role of Transglutaminase 2 (tg2) in Regulation of Macrophage Phenotype

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