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Exploration of New Therapies for Heart Failure Targeting Age-Related Mechanisms

Circulation Journal, 2024/04/25, Vol.88(5), pp.626-630

Shimizu, Ippei

2024

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Physiological and pathological cardiac hypertrophy

Journal of molecular and cellular cardiology, 2016-08, Vol.97, p.245-262

2016

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“Reproducibility and reporting of negative results in cardiovascular research” from the perspective of mid-career investigator

Journal of Molecular and Cellular Cardiology Plus (Online), 2022-12, Vol.2, p.100017, Article 100017

Shimizu, Ippei

2022

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Cellular senescence in cardiac diseases

Journal of cardiology, 2019-10, Vol.74 (4), p.313-319

2019

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Capillaries as a Therapeutic Target for Heart Failure

Journal of Atherosclerosis and Thrombosis, 2022/07/01, Vol.29(7), pp.971-988

2022

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Vascular Senescence in Cardiovascular and Metabolic Diseases

Frontiers in cardiovascular medicine, 2018-03, Vol.5, p.18-18

2018

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Metabolomic Analysis in Heart Failure

Circulation Journal, 2017/12/25, Vol.82(1), pp.10-16

2018

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The Whitening of Brown Fat and Its Implications for Weight Management in Obesity

Current obesity reports, 2015-06, Vol.4 (2), p.224-229

2015

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Glycoprotein nonmetastatic melanoma protein B regulates lysosomal integrity and lifespan of senescent cells

Scientific reports, 2022-04, Vol.12 (1), p.6522-6522, Article 6522

2022

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Semaphorin3E-Induced Inflammation Contributes to Insulin Resistance in Dietary Obesity

Cell metabolism, 2013-10, Vol.18 (4), p.491-504

2013

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Empagliflozin maintains capillarization and improves cardiac function in a murine model of left ventricular pressure overload

Scientific reports, 2021-09, Vol.11 (1), p.18384-18384, Article 18384

2021

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A crucial role for adipose tissue p53 in the regulation of insulin resistance

Nature medicine, 2009-09, Vol.15 (9), p.1082-1087

2009

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A role for circadian clock in metabolic disease

Hypertension research, 2016-07, Vol.39 (7), p.483-491

2016

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Secreted Frizzled-related Protein 5 Diminishes Cardiac Inflammation and Protects the Heart from Ischemia/Reperfusion Injury

The Journal of biological chemistry, 2016-02, Vol.291 (6), p.2566-2575

2016

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Boysenberry polyphenol inhibits endothelial dysfunction and improves vascular health

PloS one, 2018-08, Vol.13 (8), p.e0202051-e0202051

2018

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Cardiac mitofusin-1 is reduced in non-responding patients with idiopathic dilated cardiomyopathy

Scientific reports, 2021-03, Vol.11 (1), p.6722-6722, Article 6722

2021

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Arachidonate 12/15-lipoxygenase-induced inflammation and oxidative stress are involved in the development of diabetic cardiomyopathy

Diabetes (New York, N.Y.), 2015-02, Vol.64 (2), p.618-630

2015

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An antiangiogenic isoform of VEGF-A contributes to impaired vascularization in peripheral artery disease

Nature medicine, 2014-12, Vol.20 (12), p.1464-1471

2014

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p53-Induced inflammation exacerbates cardiac dysfunction during pressure overload

Journal of molecular and cellular cardiology, 2015-08, Vol.85, p.183-198

2015

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Exploration of New Therapies for Heart Failure Targeting Age-Related Mechanisms

Circulation Journal, 2023/07/13, pp.CJ-23-0419

Shimizu, Ippei

2023

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p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

Nature, 2007-03, Vol.446 (7134), p.444-448

2007

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Maintenance of Subcutaneous Fat Homeostasis Improves Systemic Metabolic Dysfunction in Obesity

Diabetes (New York, N.Y.), 2015-12, Vol.64 (12), p.3984-3986

2015

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p53-Induced Adipose Tissue Inflammation Is Critically Involved in the Development of Insulin Resistance in Heart Failure

Cell metabolism, 2012-01, Vol.15 (1), p.51-64

2012

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Role of circulating molecules in age-related cardiovascular and metabolic disorders

Inflammation and regeneration, 2022-01, Vol.42 (1), p.2-2

2022

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Brain-derived neurotrophic factor protects against cardiac dysfunction after myocardial infarction via a central nervous system-mediated pathway

Arteriosclerosis, thrombosis, and vascular biology, 2012-08, Vol.32 (8), p.1902

2012

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Exploration of New Therapies for Heart Failure Targeting Age-Related Mechanisms

Physiological and pathological cardiac hypertrophy

“Reproducibility and reporting of negative results in cardiovascular research” from the perspective of mid-career investigator

Cellular senescence in cardiac diseases

Capillaries as a Therapeutic Target for Heart Failure

Vascular Senescence in Cardiovascular and Metabolic Diseases

Metabolomic Analysis in Heart Failure

The Whitening of Brown Fat and Its Implications for Weight Management in Obesity

Glycoprotein nonmetastatic melanoma protein B regulates lysosomal integrity and lifespan of senescent cells

Semaphorin3E-Induced Inflammation Contributes to Insulin Resistance in Dietary Obesity

Empagliflozin maintains capillarization and improves cardiac function in a murine model of left ventricular pressure overload

A crucial role for adipose tissue p53 in the regulation of insulin resistance

A role for circadian clock in metabolic disease

Secreted Frizzled-related Protein 5 Diminishes Cardiac Inflammation and Protects the Heart from Ischemia/Reperfusion Injury

Boysenberry polyphenol inhibits endothelial dysfunction and improves vascular health

Cardiac mitofusin-1 is reduced in non-responding patients with idiopathic dilated cardiomyopathy

Arachidonate 12/15-lipoxygenase-induced inflammation and oxidative stress are involved in the development of diabetic cardiomyopathy

An antiangiogenic isoform of VEGF-A contributes to impaired vascularization in peripheral artery disease

p53-Induced inflammation exacerbates cardiac dysfunction during pressure overload

Exploration of New Therapies for Heart Failure Targeting Age-Related Mechanisms

p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

Maintenance of Subcutaneous Fat Homeostasis Improves Systemic Metabolic Dysfunction in Obesity

p53-Induced Adipose Tissue Inflammation Is Critically Involved in the Development of Insulin Resistance in Heart Failure

Role of circulating molecules in age-related cardiovascular and metabolic disorders

Brain-derived neurotrophic factor protects against cardiac dysfunction after myocardial infarction via a central nervous system-mediated pathway

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