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Involvement of the Na+/Ca2+ Exchanger in the Automaticity of Guinea-Pig Pulmonary Vein Myocardium as Revealed by SEA0400

Journal of Pharmacological Sciences, 2009, Vol.110(1), pp.111-116

2009

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Effect of SEA0400, a novel inhibitor of sodium‐calcium exchanger, on myocardial ionic currents

British journal of pharmacology, 2002-03, Vol.135 (5), p.1096-1100

2002

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Pathophysiological Significance of T-type Ca2+ Channels: T-type Ca2+ Channels and Drug Development

Journal of Pharmacological Sciences, 2005, Vol.99(3), pp.214-220

2005

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Role of Nitric Oxide in Ca2+ Sensitivity of the Slowly Activating Delayed Rectifier K+ Current in Cardiac Myocytes

Circulation research, 2005-01, Vol.96 (1), p.64-72

2005

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New Aspects for the Treatment of Cardiac Diseases Based on the Diversity of Functional Controls on Cardiac Muscles: Diversity in the Excitation–Contraction Mechanisms of the Heart

Journal of Pharmacological Sciences, 2009, Vol.109(3), pp.327-333

2009

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A facile access to spiro furanone skeleton based on Pd(II)-mediated cyclization–carbonylation of propargylic esters

Tetrahedron, 2006-03, Vol.62 (11), p.2545-2554

2006

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Reduction by SEA0400 of myocardial ischemia-induced cytoplasmic and mitochondrial Ca2+ overload

European journal of pharmacology, 2006-08, Vol.543 (1-3), p.108-115

2006

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Blockade by NIP-142, an Antiarrhythmic Agent, of Carbachol-Induced Atrial Action Potential Shortening and GIRK1/4 Channel

Journal of Pharmacological Sciences, 2006, Vol.101(4), pp.303-310

2006

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Developmental Changes in Action Potential Prolongation by K+-Channel Blockers in Chick Myocardium

Journal of Pharmacological Sciences, 2011, Vol.115(2), pp.235-238

2011

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Involvement of the Na+/Ca2+ Exchanger in Ouabain-Induced Inotropy and Arrhythmogenesis in Guinea-Pig Myocardium as Revealed by SEA0400

Journal of Pharmacological Sciences, 2007, Vol.103(2), pp.241-246

2007

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Simultaneous Real-Time Detection of Initiator- and Effector-Caspase Activation by Double Fluorescence Resonance Energy Transfer Analysis

Journal of Pharmacological Sciences, 2005, Vol.97(3), pp.361-368

2005

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Kv channels contribute to nitric oxide- and atrial natriuretic peptide-induced relaxation of a rat conduit artery

The Journal of pharmacology and experimental therapeutics, 2006-04, Vol.317 (1), p.341

2006

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Species Difference in the Contribution of T-Type Calcium Current to Cardiac Pacemaking as Revealed by R(−)-Efonidipine

Journal of Pharmacological Sciences, 2008, Vol.107(1), pp.99-102

2008

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β1-Subunit of MaxiK Channel in Smooth Muscle: a Key Molecule Which Tunes Muscle Mechanical Activity

Journal of Pharmacological Sciences, 2004, Vol.94(4), pp.339-347

2004

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Atria Selective Prolongation by NIP-142, an Antiarrhythmic Agent, of Refractory Period and Action Potential Duration in Guinea Pig Myocardium

Journal of Pharmacological Sciences, 2005, Vol.98(1), pp.33-40

2005

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Quantitative Fluorescence Measurement of Cardiac Na+/Ca2+ Exchanger Inhibition by Kinetic Analysis in Stably Transfected HEK293 Cells

Journal of Pharmacological Sciences, 2006, Vol.101(4), pp.356-360

2006

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The R(−)-Enantiomer of Efonidipine Blocks T-type but Not L-type Calcium Current in Guinea Pig Ventricular Myocardium

Journal of Pharmacological Sciences, 2004, Vol.96(4), pp.499-501

2004

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Chronic left atrial volume overload abbreviates the action potential duration of the canine pulmonary vein myocardium via activation of IK channel

European journal of pharmacology, 2008-11, Vol.597 (1), p.81-85

2008

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Muscarinic Receptor Subtypes Mediating Positive and Negative Inotropy in the Developing Chick Ventricle

Journal of Pharmacological Sciences, 2007, Vol.103(1), pp.75-82

2007

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Analysis of Arrhythmogenic Profile in a Canine Model of Chronic Atrioventricular Block by Comparing In Vitro Effects of the Class III Antiarrhythmic Drug Nifekalant on the Ventricular Action Potential Indices Between Normal Heart and Atrioventricular Block Heart

Journal of Pharmacological Sciences, 2007, Vol.103(2), pp.181-188

2007

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Cardioprotection without cardiosuppression by SEA0400, a novel inhibitor of Na+-Ca2+ exchanger, during ischemia and reperfusion in guinea-pig myocardium

Life sciences (1973), 2005-06, Vol.77 (3), p.312-324

2005

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Effect of Cilnidipine on L- and T-type Calcium Currents in Guinea Pig Ventricle and Action Potential in Rabbit Sinoatrial Node

Journal of Pharmacological Sciences, 2004, Vol.95(3), pp.398-401

2004

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Involvement of Intracellular Ca2+ in the Regulatory Volume Decrease After Hyposmotic Swelling in MDCK Cells

Journal of Pharmacological Sciences, 2007, Vol.104(4), pp.397-401

2007

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Intracellular mechanisms and receptor types for endothelin-1-induced positive and negative inotropy in mouse ventricular myocardium

Naunyn-Schmiedeberg's archives of pharmacology, 2008-02, Vol.376 (6), p.385-395

2008

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Unique excitation-contraction characteristics of mouse myocardium as revealed by SEA0400, a specific inhibitor of Na+-Ca2+ exchanger

Naunyn-Schmiedeberg's archives of pharmacology, 2005-06, Vol.371 (6), p.526-534

2005

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Involvement of the Na+/Ca2+ Exchanger in the Automaticity of Guinea-Pig Pulmonary Vein Myocardium as Revealed by SEA0400

Effect of SEA0400, a novel inhibitor of sodium‐calcium exchanger, on myocardial ionic currents

Pathophysiological Significance of T-type Ca2+ Channels: T-type Ca2+ Channels and Drug Development

Role of Nitric Oxide in Ca2+ Sensitivity of the Slowly Activating Delayed Rectifier K+ Current in Cardiac Myocytes

New Aspects for the Treatment of Cardiac Diseases Based on the Diversity of Functional Controls on Cardiac Muscles: Diversity in the Excitation–Contraction Mechanisms of the Heart

A facile access to spiro furanone skeleton based on Pd(II)-mediated cyclization–carbonylation of propargylic esters

Reduction by SEA0400 of myocardial ischemia-induced cytoplasmic and mitochondrial Ca2+ overload

Blockade by NIP-142, an Antiarrhythmic Agent, of Carbachol-Induced Atrial Action Potential Shortening and GIRK1/4 Channel

Developmental Changes in Action Potential Prolongation by K+-Channel Blockers in Chick Myocardium

Involvement of the Na+/Ca2+ Exchanger in Ouabain-Induced Inotropy and Arrhythmogenesis in Guinea-Pig Myocardium as Revealed by SEA0400

Simultaneous Real-Time Detection of Initiator- and Effector-Caspase Activation by Double Fluorescence Resonance Energy Transfer Analysis

Kv channels contribute to nitric oxide- and atrial natriuretic peptide-induced relaxation of a rat conduit artery

Species Difference in the Contribution of T-Type Calcium Current to Cardiac Pacemaking as Revealed by R(−)-Efonidipine

β1-Subunit of MaxiK Channel in Smooth Muscle: a Key Molecule Which Tunes Muscle Mechanical Activity

Atria Selective Prolongation by NIP-142, an Antiarrhythmic Agent, of Refractory Period and Action Potential Duration in Guinea Pig Myocardium

Quantitative Fluorescence Measurement of Cardiac Na+/Ca2+ Exchanger Inhibition by Kinetic Analysis in Stably Transfected HEK293 Cells

The R(−)-Enantiomer of Efonidipine Blocks T-type but Not L-type Calcium Current in Guinea Pig Ventricular Myocardium

Chronic left atrial volume overload abbreviates the action potential duration of the canine pulmonary vein myocardium via activation of IK channel

Muscarinic Receptor Subtypes Mediating Positive and Negative Inotropy in the Developing Chick Ventricle

Analysis of Arrhythmogenic Profile in a Canine Model of Chronic Atrioventricular Block by Comparing In Vitro Effects of the Class III Antiarrhythmic Drug Nifekalant on the Ventricular Action Potential Indices Between Normal Heart and Atrioventricular Block Heart

Cardioprotection without cardiosuppression by SEA0400, a novel inhibitor of Na+-Ca2+ exchanger, during ischemia and reperfusion in guinea-pig myocardium

Effect of Cilnidipine on L- and T-type Calcium Currents in Guinea Pig Ventricle and Action Potential in Rabbit Sinoatrial Node

Involvement of Intracellular Ca2+ in the Regulatory Volume Decrease After Hyposmotic Swelling in MDCK Cells

Intracellular mechanisms and receptor types for endothelin-1-induced positive and negative inotropy in mouse ventricular myocardium

Unique excitation-contraction characteristics of mouse myocardium as revealed by SEA0400, a specific inhibitor of Na+-Ca2+ exchanger

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