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Syntaxin 17 regulates the localization and function of PGAM5 in mitochondrial division and mitophagy

The EMBO journal, 2018-11, Vol.37 (21), p.n/a

2018

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α‐Synuclein V15A Variant in Familial Parkinson's Disease Exhibits a Weaker Lipid‐Binding Property

Movement disorders, 2022-10, Vol.37 (10), p.2075-2085

2022

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PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

The Journal of cell biology, 2010-04, Vol.189 (2), p.211-221

2010

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PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy

Scientific reports, 2012-12, Vol.2 (1), p.1002-1002, Article 1002

2012

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Twin CHCH Proteins, CHCHD2, and CHCHD10: Key Molecules of Parkinson's Disease, Amyotrophic Lateral Sclerosis, and Frontotemporal Dementia

International journal of molecular sciences, 2019-02, Vol.20 (4), p.908

2019

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Loss of Parkinson's disease-associated protein CHCHD2 affects mitochondrial crista structure and destabilizes cytochrome c

Nature communications, 2017-06, Vol.8 (1), p.15500-15500, Article 15500

2017

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Parkinson disease-associated Leucine-rich repeat kinase regulates UNC-104-dependent axonal transport of Arl8-positive vesicles in Drosophila

iScience, 2022-12, Vol.25 (12), p.105476-105476, Article 105476

2022

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PINK1 autophosphorylation upon membrane potential dissipation is essential for Parkin recruitment to damaged mitochondria

Nature communications, 2012-08, Vol.3 (1), p.1016-1016, Article 1016

2012

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PINK1-mediated phosphorylation of Parkin boosts Parkin activity in Drosophila

PLoS genetics, 2014-06, Vol.10 (6), p.e1004391-e1004391

2014

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Loss of nuclear REST/NRSF in aged-dopaminergic neurons in Parkinson’s disease patients

Neuroscience letters, 2019-04, Vol.699, p.59-63

2019

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Phosphorylation of mitochondrial polyubiquitin by PINK1 promotes Parkin mitochondrial tethering

PLoS genetics, 2014-12, Vol.10 (12), p.e1004861-e1004861

2014

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Light-driven activation of mitochondrial proton-motive force improves motor behaviors in a Drosophila model of Parkinson's disease

Communications biology, 2019-11, Vol.2 (1), p.424-424, Article 424

2019

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Tricornered/NDR kinase signaling mediates PINK1-directed mitochondrial quality control and tissue maintenance

Genes & development, 2013-01, Vol.27 (2), p.157-162

2013

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Ubiquitination at the lysine 27 residue of the Parkin ubiquitin-like domain is suggestive of a new mechanism of Parkin activation

Human molecular genetics, 2022-08, Vol.31 (15), p.2623-2638

2022

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Identifying Therapeutic Agents for Amelioration of Mitochondrial Clearance Disorder in Neurons of Familial Parkinson Disease

Stem cell reports, 2020-06, Vol.14 (6), p.1060-1075

2020

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Clinical characteristics and pathophysiological properties of newly discovered LRRK2 variants associated with Parkinson's disease

Neurobiology of disease, 2024-09, Vol.199, p.106571, Article 106571

2024

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Monitoring Mitochondrial Changes by Alteration of the PINK1-Parkin Signaling in Drosophila

Methods in molecular biology (Clifton, N.J.), 2018, Vol.1759, p.47

2018

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A Cell-Based High-Throughput Screening Identified Two Compounds that Enhance PINK1-Parkin Signaling

iScience, 2020-05, Vol.23 (5), p.101048-101048, Article 101048

2020

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Pathophysiological evaluation of the LRRK2 G2385R risk variant for Parkinson’s disease

NPJ Parkinson's Disease, 2022-08, Vol.8 (1), p.97-97, Article 97

2022

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Reduced TDP-43 Expression Improves Neuronal Activities in a Drosophila Model of Perry Syndrome

EBioMedicine, 2017-07, Vol.21 (C), p.218-227

2017

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Reduced astrocytic reactivity in human brains and midbrain organoids with PRKN mutations

NPJ Parkinson's Disease, 2020-11, Vol.6 (1), p.33-33, Article 33

2020

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Parkin stabilizes PINK1 through direct interaction

Biochemical and biophysical research communications, 2009-06, Vol.383 (3), p.331-335

2009

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Parkinson’s disease-associated iPLA2-VIA/PLA2G6 regulates neuronal functions and α-synuclein stability through membrane remodeling

Proceedings of the National Academy of Sciences - PNAS, 2019-10, Vol.116 (41), p.20689-20699

2019

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Lysine 63-linked Polyubiquitination Is Dispensable for Parkin-mediated Mitophagy

The Journal of biological chemistry, 2014-11, Vol.289 (48), p.33131-33136

2014

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Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson's disease

Human molecular genetics, 2017-08, Vol.26 (16), p.3172-3185

2017

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Syntaxin 17 regulates the localization and function of PGAM5 in mitochondrial division and mitophagy

α‐Synuclein V15A Variant in Familial Parkinson's Disease Exhibits a Weaker Lipid‐Binding Property

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy

Twin CHCH Proteins, CHCHD2, and CHCHD10: Key Molecules of Parkinson's Disease, Amyotrophic Lateral Sclerosis, and Frontotemporal Dementia

Loss of Parkinson's disease-associated protein CHCHD2 affects mitochondrial crista structure and destabilizes cytochrome c

Parkinson disease-associated Leucine-rich repeat kinase regulates UNC-104-dependent axonal transport of Arl8-positive vesicles in Drosophila

PINK1 autophosphorylation upon membrane potential dissipation is essential for Parkin recruitment to damaged mitochondria

PINK1-mediated phosphorylation of Parkin boosts Parkin activity in Drosophila

Loss of nuclear REST/NRSF in aged-dopaminergic neurons in Parkinson’s disease patients

Phosphorylation of mitochondrial polyubiquitin by PINK1 promotes Parkin mitochondrial tethering

Light-driven activation of mitochondrial proton-motive force improves motor behaviors in a Drosophila model of Parkinson's disease

Tricornered/NDR kinase signaling mediates PINK1-directed mitochondrial quality control and tissue maintenance

Ubiquitination at the lysine 27 residue of the Parkin ubiquitin-like domain is suggestive of a new mechanism of Parkin activation

Identifying Therapeutic Agents for Amelioration of Mitochondrial Clearance Disorder in Neurons of Familial Parkinson Disease

Clinical characteristics and pathophysiological properties of newly discovered LRRK2 variants associated with Parkinson's disease

Monitoring Mitochondrial Changes by Alteration of the PINK1-Parkin Signaling in Drosophila

A Cell-Based High-Throughput Screening Identified Two Compounds that Enhance PINK1-Parkin Signaling

Pathophysiological evaluation of the LRRK2 G2385R risk variant for Parkinson’s disease

Reduced TDP-43 Expression Improves Neuronal Activities in a Drosophila Model of Perry Syndrome

Reduced astrocytic reactivity in human brains and midbrain organoids with PRKN mutations

Parkin stabilizes PINK1 through direct interaction

Parkinson’s disease-associated iPLA2-VIA/PLA2G6 regulates neuronal functions and α-synuclein stability through membrane remodeling

Lysine 63-linked Polyubiquitination Is Dispensable for Parkin-mediated Mitophagy

Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson's disease

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