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Stress granules regulate stress-induced paraspeckle assembly

The Journal of cell biology, 2019-12, Vol.218 (12), p.4127-4140

2019

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Connecting the “dots”: RNP granule network in health and disease

Biochimica et biophysica acta. Molecular cell research, 2021-07, Vol.1868 (8), p.119058-119058, Article 119058

2021

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Compromised paraspeckle formation as a pathogenic factor in FUSopathies

Human molecular genetics, 2014-05, Vol.23 (9), p.2298-2312

2014

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Multistep process of FUS aggregation in the cell cytoplasm involves RNA-dependent and RNA-independent mechanisms

Human molecular genetics, 2014-10, Vol.23 (19), p.5211-5226

2014

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ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity

Neurobiology of disease, 2022-01, Vol.162, p.105585-105585, Article 105585

2022

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Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis

Molecular neurodegeneration, 2018-06, Vol.13 (1), p.30-30, Article 30

2018

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Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies

PLoS genetics, 2019-08, Vol.15 (8), p.e1008308

2019

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NEAT1 and paraspeckles in neurodegenerative diseases: A missing lnc found?

Non-coding RNA research, 2018-12, Vol.3 (4), p.243-252

2018

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ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles

Acta neuropathologica communications, 2019-01, Vol.7 (1), p.7-7, Article 7

2019

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Modelling FUSopathies: focus on protein aggregation

Biochemical Society transactions, 2013-12, Vol.41 (6), p.1613-1617

Shelkovnikova, Tatyana A

2013

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Long non-coding RNA NEAT1_1 ameliorates TDP-43 toxicity in in vivo models of TDP-43 proteinopathy

RNA biology, 2021-11, Vol.18 (11), p.1546-1554

2021

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The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function

Nature communications, 2023-09, Vol.14 (1), p.5496-5496, Article 5496

2023

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Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis

Cell reports (Cambridge), 2019-12, Vol.29 (13), p.4496-4508.e4

2019

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Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletion

Cell reports (Cambridge), 2024-07, Vol.43 (7), p.114421, Article 114421

2024

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Fused in Sarcoma (FUS) Protein Lacking Nuclear Localization Signal (NLS) and Major RNA Binding Motifs Triggers Proteinopathy and Severe Motor Phenotype in Transgenic Mice

The Journal of biological chemistry, 2013-08, Vol.288 (35), p.25266-25274

2013

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Chronically stressed or stress-preconditioned neurons fail to maintain stress granule assembly

Cell death & disease, 2017-05, Vol.8 (5), p.e2788-e2788

2017

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Stress granules regulate paraspeckles: RNP granule continuum at work

Cell Stress, 2019-11, Vol.3 (12), p.385-387

2019

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Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS

Molecular brain, 2020-05, Vol.13 (1), p.77-77, Article 77

2020

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TDP-43 in nuclear condensates: where, how, and why

Biochemical Society transactions, 2024-08, Vol.52 (4), p.1809-1825

2024

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Selective pattern of motor system damage in gamma-synuclein transgenic mice mirrors the respective pathology in amyotrophic lateral sclerosis

Neurobiology of disease, 2012-10, Vol.48 (1), p.124-131

2012

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Modulation of p-eIF2α cellular levels and stress granule assembly/disassembly by trehalose

Scientific reports, 2017-03, Vol.7 (1), p.44088-44088, Article 44088

2017

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A toolkit for the identification of NEAT1_2/paraspeckle modulators

Nucleic acids research, 2022-11, Vol.50 (20), p.e119-e119

2022

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Assessment of radiological techniques application possibility for non-invasive diagnostics of latent inflammatory processes in myocardium in patients with atrial fibrillation

Annals of nuclear medicine, 2016-12, Vol.30 (10), p.738-748

2016

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RNP granules in ALS and neurodegeneration: From multifunctional membraneless organelles to therapeutic opportunities

International review of neurobiology, 2024, Vol.176, p.455

2024

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Recruitment into stress granules prevents irreversible aggregation of FUS protein mislocalized to the cytoplasm

Cell cycle (Georgetown, Tex.), 2013-10, Vol.12 (19), p.3383-3391

2013

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Stress granules regulate stress-induced paraspeckle assembly

Connecting the “dots”: RNP granule network in health and disease

Compromised paraspeckle formation as a pathogenic factor in FUSopathies

Multistep process of FUS aggregation in the cell cytoplasm involves RNA-dependent and RNA-independent mechanisms

ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity

Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis

Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies

NEAT1 and paraspeckles in neurodegenerative diseases: A missing lnc found?

ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles

Modelling FUSopathies: focus on protein aggregation

Long non-coding RNA NEAT1_1 ameliorates TDP-43 toxicity in in vivo models of TDP-43 proteinopathy

The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function

Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis

Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletion

Fused in Sarcoma (FUS) Protein Lacking Nuclear Localization Signal (NLS) and Major RNA Binding Motifs Triggers Proteinopathy and Severe Motor Phenotype in Transgenic Mice

Chronically stressed or stress-preconditioned neurons fail to maintain stress granule assembly

Stress granules regulate paraspeckles: RNP granule continuum at work

Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS

TDP-43 in nuclear condensates: where, how, and why

Selective pattern of motor system damage in gamma-synuclein transgenic mice mirrors the respective pathology in amyotrophic lateral sclerosis

Modulation of p-eIF2α cellular levels and stress granule assembly/disassembly by trehalose

A toolkit for the identification of NEAT1_2/paraspeckle modulators

Assessment of radiological techniques application possibility for non-invasive diagnostics of latent inflammatory processes in myocardium in patients with atrial fibrillation

RNP granules in ALS and neurodegeneration: From multifunctional membraneless organelles to therapeutic opportunities

Recruitment into stress granules prevents irreversible aggregation of FUS protein mislocalized to the cytoplasm

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