UB Paderborn / Katalog / Suche / in CDI

Tubulin binding blocks mitochondrial voltage-dependent anion channel and regulates respiration

Proceedings of the National Academy of Sciences - PNAS, 2008-12, Vol.105 (48), p.18746-18751

2008

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Phosphorylation of voltage-dependent anion channel by serine/threonine kinases governs its interaction with tubulin

PloS one, 2011-10, Vol.6 (10), p.e25539

2011

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S1597 Modeling Gastric Luminal Rifabutin Concentrations: RHB-105 (Rifabutin 50 mg Q8H) Provides More Favorable Exposure for Helicobacter pylori Eradication Than Generic Rifabutin 150 mg BID or 300 mg QD

The American journal of gastroenterology, 2022-10, Vol.117 (10S), p.e1140-e1141

2022

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Physiologically‐based pharmacokinetic modelling to predict intragastric rifabutin concentrations in the treatment of Helicobacter pylori infection

Alimentary pharmacology & therapeutics, 2023-07, Vol.58 (2), p.159-167

2023

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S1451 Pitfalls of Physician-Directed Treatment of H. pylori Infection: Results From Two Phase 3 Clinical Trials and Real-World Prescribing Data

The American journal of gastroenterology, 2021-10, Vol.116 (1), p.S665-S665

2021

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Voltage-dependent anion channels modulate mitochondrial metabolism in cancer cells: regulation by free tubulin and erastin

The Journal of biological chemistry, 2013-04, Vol.288 (17), p.11920-11929

2013

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Pitfalls of Physician-Directed Treatment of Helicobacter pylori: Results from Two Phase 3 Clinical Trials and Real-World Prescribing Data

Digestive diseases and sciences, 2022-09, Vol.67 (9), p.4382-4386

2022

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Tubulin Tail Sequences and Post-translational Modifications Regulate Closure of Mitochondrial Voltage-dependent Anion Channel (VDAC)

The Journal of biological chemistry, 2015-10, Vol.290 (44), p.26784-26789

2015

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S1417 RHB-105 Q8H Rifabutin Dosing Provides Favorable Exposure for Helicobacter pylori Eradication

The American journal of gastroenterology, 2021-10, Vol.116 (1), p.S650-S650

2021

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Role of Subplasmalemmal Mitochondria in Angiotensin II–Mediated Contraction

Circulation research, 2015-12, Vol.117 (12), p.984-986

2015

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Sa1381 PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODELING CONFIRMS RIFABUTIN, AMOXICILLIN, AND OMEPRAZOLE BIOEQUIVALENCE BETWEEN Q8H AND TID DOSING FOR LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) FOR THE ERADICATION OF HELICOBACTER PYLORI IN ADULT PATIENTS

Gastroenterology (New York, N.Y. 1943), 2023-05, Vol.164 (6), p.S-380-S-381

2023

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Tu1078: LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) MAINTAINS HIGH HELICOBACTER PYLORI ERADICATION RATES AND SHOWS FAVORABLE SAFETY AND EFFICACY IN SUBJECTS WITH DIABETES MELLITUS

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-873-S-874

2022

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Tu1077: PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODEL TO ESTIMATE INTRAGASTRIC RIFABUTIN CONCENTRATIONS FOLLOWING RIFABUTIN 50 MG Q8H (RHB-105) OR 150 MG QD: MORE FAVORABLE EXPOSURE FOR HELICOBACTER PYLORI ERADICATON WITH Q8H DOSING

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-873-S-873

2022

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S1342 Efficacy of H. pylori Eradication by Low-Dose Rifabutin Triple Therapy (RHB-105) Is Unaffected by BMI: Post-hoc Analysis From Two Phase 3 Trials

The American journal of gastroenterology, 2020-10, Vol.115 (1), p.S677-S677

2020

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Abstract 3044: The ability of tubulin to close mitochondrial VDAC pores depends on beta tubulin isotype

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.3044-3044

2015

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Helicobacter pylori eradication by low‐dose rifabutin triple therapy (RHB‐105) is unaffected by high body mass index

GastroHep, 2021-12, Vol.3 (7), p.426-434

2021

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Novel Mechanism of Mitochondrial Respiration Control through Competition between Hexokinase-2 and Tubulin for VDAC Binding

Biophysical journal, 2013-01, Vol.104 (2), p.655a-655a

2013

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Antagonists of the Inhibitory Effect of Free Tubulin on VDAC Induce Oxidative Stress and Mitochondrial Dysfunction

Biophysical journal, 2014-01, Vol.106 (2), p.591a-591a

2014

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Antagonists of tubulin–VDAC interaction induce oxidative stress and mitochondrial dysfunction

Biochimica et biophysica acta. Bioenergetics, 2014-07, Vol.1837, p.e111-e111

2014

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Current Insights into the Molecular Mechanisms of VDAC-Tubulin Interaction

Biophysical journal, 2013-01, Vol.104 (2), p.215a-215a

2013

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VDAC Regulation by Tubulin and its Physiological Implications

Biophysical journal, 2012-01, Vol.102 (3), p.161a-161a

2012

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VDAC Phosphorylation in Control of Mitochondrial Respiration

Biophysical journal, 2012-01, Vol.102 (3), p.436a-437a

2012

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VDAC Phosphorylation Regulates Interaction with Tubulin

Biophysical journal, 2010-01, Vol.98 (3), p.735a-735a

2010

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Voltage-dependent Anion Channels Modulate Mitochondrial Metabolism in Cancer Cells

The Journal of biological chemistry, 2013-04, Vol.288 (17), p.11920-11929

2013

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Membrane Lipid Composition Regulates Tubulin-VDAC Interaction

Biophysical journal, 2011-02, Vol.100 (3), p.42a-42a

2011

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Tubulin binding blocks mitochondrial voltage-dependent anion channel and regulates respiration

Phosphorylation of voltage-dependent anion channel by serine/threonine kinases governs its interaction with tubulin

S1597 Modeling Gastric Luminal Rifabutin Concentrations: RHB-105 (Rifabutin 50 mg Q8H) Provides More Favorable Exposure for Helicobacter pylori Eradication Than Generic Rifabutin 150 mg BID or 300 mg QD

Physiologically‐based pharmacokinetic modelling to predict intragastric rifabutin concentrations in the treatment of Helicobacter pylori infection

S1451 Pitfalls of Physician-Directed Treatment of H. pylori Infection: Results From Two Phase 3 Clinical Trials and Real-World Prescribing Data

Voltage-dependent anion channels modulate mitochondrial metabolism in cancer cells: regulation by free tubulin and erastin

Pitfalls of Physician-Directed Treatment of Helicobacter pylori: Results from Two Phase 3 Clinical Trials and Real-World Prescribing Data

Tubulin Tail Sequences and Post-translational Modifications Regulate Closure of Mitochondrial Voltage-dependent Anion Channel (VDAC)

S1417 RHB-105 Q8H Rifabutin Dosing Provides Favorable Exposure for Helicobacter pylori Eradication

Role of Subplasmalemmal Mitochondria in Angiotensin II–Mediated Contraction

Sa1381 PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODELING CONFIRMS RIFABUTIN, AMOXICILLIN, AND OMEPRAZOLE BIOEQUIVALENCE BETWEEN Q8H AND TID DOSING FOR LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) FOR THE ERADICATION OF HELICOBACTER PYLORI IN ADULT PATIENTS

Tu1078: LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) MAINTAINS HIGH HELICOBACTER PYLORI ERADICATION RATES AND SHOWS FAVORABLE SAFETY AND EFFICACY IN SUBJECTS WITH DIABETES MELLITUS

Tu1077: PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODEL TO ESTIMATE INTRAGASTRIC RIFABUTIN CONCENTRATIONS FOLLOWING RIFABUTIN 50 MG Q8H (RHB-105) OR 150 MG QD: MORE FAVORABLE EXPOSURE FOR HELICOBACTER PYLORI ERADICATON WITH Q8H DOSING

S1342 Efficacy of H. pylori Eradication by Low-Dose Rifabutin Triple Therapy (RHB-105) Is Unaffected by BMI: Post-hoc Analysis From Two Phase 3 Trials

Abstract 3044: The ability of tubulin to close mitochondrial VDAC pores depends on beta tubulin isotype

Helicobacter pylori eradication by low‐dose rifabutin triple therapy (RHB‐105) is unaffected by high body mass index

Novel Mechanism of Mitochondrial Respiration Control through Competition between Hexokinase-2 and Tubulin for VDAC Binding

Antagonists of the Inhibitory Effect of Free Tubulin on VDAC Induce Oxidative Stress and Mitochondrial Dysfunction

Antagonists of tubulin–VDAC interaction induce oxidative stress and mitochondrial dysfunction

Current Insights into the Molecular Mechanisms of VDAC-Tubulin Interaction

VDAC Regulation by Tubulin and its Physiological Implications

VDAC Phosphorylation in Control of Mitochondrial Respiration

VDAC Phosphorylation Regulates Interaction with Tubulin

Voltage-dependent Anion Channels Modulate Mitochondrial Metabolism in Cancer Cells

Membrane Lipid Composition Regulates Tubulin-VDAC Interaction

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